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Intervensi media massa untuk pemberhentian merokok dalam kalangan dewasa

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Referencias

References to studies included in this review

California TCP 2003 {published data only}

Al‐Delaimy WK, White MM, Mills AL, Pierce JP, Emory K, Boman M, et al. Two Decades of the California Tobacco Control Program: California Tobacco Survey, 1990‐2008. libraries.ucsd.edu/ssds/pub/CTS/cpc00009/Final_CTS_2008.pdf (accessed prior to 7 November 2017). CENTRAL
Al‐Delaimy WK, Zablocki RW, Myers MG, Strong DR, Edland SD, Hofstetter CR, et al. The 2011 California Smokers Cohort: A Longitudinal Perspective. archive.cdph.ca.gov/programs/tobacco/Pages/Reports.aspx (accessed 20 June 2017). CENTRAL
Bal DG. Designing effective statewide tobacco control programme ‐ California. Cancer 1998;83(Suppl 12):2717‐21. CENTRAL
Breslow L, Johnson M. California's Proposition 99 on tobacco, and its impact. Annual Review of Public Health 1993;14:585‐604. CENTRAL
California Department of Health Services, Tobacco Control Section. California tobacco control update. California Department of Health Services, Tobacco Control Section, Sacramento, CA2006. CENTRAL
California Department of Public Health, California Tobacco Control Program. California Tobacco Control Update 2009: 20 Years of Tobacco Control in California:. California Department of Public Health, California Tobacco Control Program, Sacramento, CA2009:1‐30. CENTRAL
Elder JP, Edwards CC, Conway TL, Kenney E, Johnson CA, Bennett ED. Independent evaluation of the California Tobacco Education Program. Public Health Reports 1996;111(4):353‐8. CENTRAL
Gilpin EA, Emery SL, Farkas AJ, Distefan JM, White MM, Pierce JP. The California Tobacco Control Program: A decade of progress: Results from the California Tobacco Surveys, 1990‐1998. libraries.ucsd.edu/ssds/pub/CTS/cpc00006/1999_Final_Report.pdf (accessed prior to 7 November 2017). CENTRAL
Gilpin EA, Messer K, White MM, Pierce JP. What contributed to the major decline in per capita cigarette consumption during California's comprehensive tobacco control programme?. Tobacco Control 2006;15(4):308‐16. CENTRAL
Gilpin EA, White MM, White VM, Distefan JM, Trinidad DR, James L, et al. Tobacco control successes in California: a focus on young people: results from the California Tobacco Surveys, 1990‐2002. libraries.ucsd.edu/ssds/pub/CTS/cpc00007/2002FINAL_RPT.pdf (accessed prior to 7 November 2017). CENTRAL
Glantz SA. Changes in cigarette consumption, prices, and tobacco industry revenues associated with California's Propostion 99. Tobacco Control 1993;2(4):311‐4. CENTRAL
Goldman LK, Glantz SA. Evaluation of antismoking advertising campaigns. JAMA 1998;279(10):772‐7. CENTRAL
Hu T, Keeler TE, Sung H, Barnett PG. The impact of California anti‐smoking legislation on cigarette sales, consumption, and prices. Tobacco Control 1995;4(suppl 1):S34‐8. CENTRAL
Hu T, Sung H‐Y, Keeler TE. Reducing cigarette consumption in California: tobacco taxes vs an anti‐smoking media campaign. American Journal of Public Health 1995;85(9):1218‐22. CENTRAL
Independent Evaluation Consortium (The Gallup Organization). Final Report. Independent Evaluation of the California Tobacco Control Prevention and Education Program: Waves 1, 2, and 3 (1996‐2000) Tobacco Control Prevention and Education Program: Waves 1, 2, and 3 (1996‐2000). The Gallup Organisation (accessed 8 December 2006). CENTRAL
Independent Evaluation Consortium (The Gallup Organization). Interim report. Independent evaluation of the California Tobacco Control Prevention and Education Program: Wave 2 data, 1998; Wave 1 and Wave 2 data comparisons, 1996‐1998. The Gallup Organisation (accessed 12 November 2007). CENTRAL
Independent Evaluation Consortium [The Gallup Organization]. Final Report of the Independent Evaluation of the California Tobacco Control Prevention and Education Program: Wave I Data, 1996‐1997. The Gallup Organisation (accessed 12 November 2007). CENTRAL
Norman GJ, Ribisl KM, Howard‐Pitney B, Howard KA, Unger JB. The relationship between home smoking bans and exposure to state tobacco control efforts and smoking behaviors. American Journal of Health Promotion 2000;15(2):81‐8. CENTRAL
Novotny TE, Siegel MB. California's tobacco control saga. Health Affairs 1996;15(1):58‐72. CENTRAL
Pierce JP, Burns DM, Berry C, Rosbrook B, Goodman J, Gilpin E, et al. Reducing tobacco consumption in California: Proposition 99 seems to work [letter]. JAMA 1991;265(10):1257‐8. CENTRAL
Pierce JP, Emery S, Gilpin E. The California Tobacco Control Program: a long term health communication project. In: Hornik RC editor(s). Public Health Communication. Evidence for Behavior Change. Lawrence Erlbaum Associates, 2002. CENTRAL
Pierce JP, Evans N, Farkas AJ, Cavin SW, Berry C, Kramer M, et al. Tobacco use in California. An evaluation of the Tobacco Control Program, 1989‐1993. http://libraries.ucsd.edu/ssds/pub/CTS/cpc00003/finalrpt1993.pdf (accessed prior to 7 November 2017). CENTRAL
Pierce JP, Farkas AJ, Evans N, Berry C, Choi W, Rosbrook B, et al. Tobacco Use in California 1992. A focus on preventing uptake in adolescents. library.ucsd.edu/dc/object/bb38582123/_3_1.pdf (accessed prior to 7 November 2017). CENTRAL
Pierce JP, Gilpin EA, Emery SL, Farkas AJ, Zhu SH, Choi WS, et al. Tobacco Control in California: Who's Winning the War? An Evaluation of The Tobacco Control Program, 1989‐1996. libraries.ucsd.edu/ssds/pub/CTS/cpc00004/1996FullReport.pdf (accessed prior to 7 November 2017). CENTRAL
Pierce JP, Gilpin EA, Emery SL, White MM, Rosbrook B, Berry CC. Has the California Tobacco Control Program reduced smoking?. JAMA 1998;280(10):893‐9. CENTRAL
Popham WJ, Potter LD, Bal DG, Johnson MD, Duerr JM, Quinn V. Do anti‐smoking media campaigns help smokers quit?. Public Health Reports 1993;108(4):510‐3. CENTRAL
Popham WJ, Potter LD, Hetrick MA, Muthen LK, Duerr JM, Johnson MD. Effectiveness of the California 1990‐1991 tobacco education media campaign. American Journal of Preventive Medicine 1994;10(6):319‐26. CENTRAL
Rohrbach LA, Howard‐Pitney B, Unger JB, Dent CW, Howard KA, Cruz TB, et al. Independent evaluation of the California Tobacco Control Program: relationships between program exposure and outcomes, 1996‐1998. American Journal of Public Health 2002;92(6):975‐83. CENTRAL
Russell CM. Evaluation: methods and strategy for evaluation ‐ California. Cancer 1998;83(12 Suppl):2755‐9. CENTRAL
Schleicher NC, Johnson T, Ahmad I, Henriksen L. Tobacco Marketing in California’s Retail Environment (2011‐2014). Final report for the California Tobacco Advertising Survey (2014) Submitted to the California Tobacco Control Program, California Department of Public Health. Stanford Prevention Research Center. Stanford University School of Medicine 2015; available fromarchive.cdph.ca.gov/programs/tobacco/Pages/Reports.aspx (accessed June 20th, 2017). CENTRAL
Siegel M, Pechacek TP, Strauss WJ, Schooley MW, Merritt RK, Novotny TE, et al. Trends in adult cigarette smoking in California compared with the rest of the United States, 1978‐1994. American Journal of Public Health 2000;90(3):372‐9. CENTRAL
Stevens C. Designing an effective counteradvertising campaign ‐ California. Cancer 1998;83(12 Suppl):2736‐41. CENTRAL
The California Department of Public Health, California Tobacco Control Program. 2016 Healthy Stores for a Healthy Community Technical Report. healthystoreshealthycommunity.com/wp‐content/uploads/2017/03/2016‐Technical‐Report‐Final.pdf (accessed 20 June 2017). CENTRAL

CORIS 1997 {published data only}

Rossouw JE, Jooste PL, Chalton DO, Jordaan ER, Langenhoven ML, Jordaan PC, et al. Community‐based intervention: the Coronary Risk Factor Study (CORIS). International Journal of Epidemiology 1993;22(3):428‐38. CENTRAL
Steenkamp HJ, Jooste PL, Jordaan PCJ, Swanepoel AS, Roussouw JE. Changes in smoking during a community‐based cardiovascular disease intervention programme. South African Medical Journal 1991;79(5):250‐3. CENTRAL
Steyn K, Steyn M, Swanepoel ASP, Jordaan PCT, Jooste PL, Fourie JM, et al. Twelve‐year results of the Coronary Risk Factor Study (CORIS). International Journal of Epidemiology 1997;26(5):964‐71. CENTRAL

Jenkins 1997 {published data only}

Jenkins CN, McPhee SJ, Le A, Pham GQ, Ha NT, Stewart S. The effectiveness of a media‐led intervention to reduce smoking among Vietnamese‐American men. American Journal of Public Health 1997;87(6):1031‐4. CENTRAL

Massachusetts 2003 {published data only}

Biener L. Adult and youth response to the Massachusetts anti‐tobacco television campaign. Journal of Public Health Management and Practice 2000;6(3):40‐4. CENTRAL
Biener L, Harris JE, Hamilton W. Impact of the Massachusetts tobacco control programme: population based trend analysis. BMJ 2000;321(7257):351‐4. CENTRAL
Biener L, McCallum‐Keeler G, Nyman AL. Adults' response to Massachusetts anti‐tobacco television advertisements: impact of viewer and advertisement characteristics. Tobacco Control 2000;9(4):401‐7. CENTRAL
Celebucki C, Biener L, Koh HK. Evaluation: methods and strategy for evaluation ‐ Massachusetts. Cancer 1998;83(12 Suppl):2760‐5. CENTRAL
Centers for Disease Control and Prevention. Cigarette smoking before and after an excise tax increase and an antismoking campaign ‐ Massachusetts, 1990‐1996. Morbidity and Mortality Weekly Report. Surveillance Summaries : MMWR 1996;45:966‐70. CENTRAL
Connolly G, Robbins H. Designing an effective statewide tobacco control program ‐ Massachusetts. Cancer 1998;83(Suppl 12A):2722‐7. CENTRAL
DeJong W, Hoffman KD. A content analysis of television advertising for the Massachusetts Tobacco Control Program media campaign 1993‐1996. Journal of Public Health Management and Practice 2000;6(3):27‐39. CENTRAL
Goldman LK, Glantz SA. Evaluation of antismoking advertising campaigns.. JAMA 1998;279(10):772‐7. CENTRAL
Hamilton W, diStefano Norton G, Weintraub J. Independent evaluation of the Massachusetts Tobacco Control Program, 7th annual report ‐ January 1994 to June. masslib‐dspace.longsight.com/bitstream/handle/2452/113191/ocm33663460‐2000.pdf?sequence=1&isAllowed=y (accessed prior to 7 November 2017). CENTRAL
Hamilton WL, Rodger CN, Chen X, Njobe TK, Kling R, diStefano Norton G. Independent Evaluation of the Massachusetts Tobacco Control Program. Eighth Annual Report: January 1994‐June 2001. masslib‐dspace.longsight.com/bitstream/handle/2452/113192/ocm33663460‐2001.pdf?sequence=1&isAllowed=y (accessed prior to 7 November 2017). CENTRAL
Massachusetts Tobacco Cessation and Prevention Program. Annual Report Massachusetts Tobacco Cessation and Prevention Program, Massachusetts Department of Public Health Fiscal Year 2009.. www.mass.gov/eohhs/docs/dph/tobacco‐control/annual‐report‐2009.pdf (accessed 20 June 2017). CENTRAL
Massachusetts Tobacco Control Program. Annual Report Massachusetts Tobacco Control Program, Massachusetts Department of Public Health Fiscal Year 2008. www.mass.gov/eohhs/docs/dph/tobacco‐control/annual‐report‐2008.pdf (accessed 20 June 2017).. CENTRAL
Massachusetts Tobacco Control Program. Annual Report of the Massachusetts Tobacco Control Program Fiscal Year 2007. www.mass.gov/eohhs/docs/dph/tobacco‐control/annual‐report‐2007.pdf (accessed prior to 7 November 2017). CENTRAL
Miller A. Designing an effective counteradvertising campaign ‐ Massachusetts. Cancer 1998;83(12 Suppl):2742‐5. CENTRAL
Robbins H, Krakow M, Warner D. Adult smoking intervention programmes in Massachusetts: a comprehensive approach with promising results. Tobacco Control 2002;11(Suppl II):ii4‐ii7. CENTRAL
Tobacco Control Program: Bureau for Famliy Health (Massachusetts Department of Public Health). Accomplishments of the Massachusetts Tobacco Control Program. www.mass.gov/eohhs/docs/dph/tobacco‐control/accomplishments.pdf (accessed 20 June 2017). CENTRAL
Weintraub JM, Hamilton WL. Trends in prevalence of current smoking, Massachusetts and states without tobacco control programmes, 1990 to 1999. Tobacco Control 2002;11(Supplement II):ii8‐ii13. CENTRAL

McAlister 2004 {published data only}

McAlister A, Morrison TC, Hu SH, Meshack AF, Ramirez A, Gallion K, et al. Media and community campaign effects on adult tobacco use in Texas. Journal of Health Communication 2004;9(2):95‐109. CENTRAL

McPhee 1995 {published data only}

McPhee SJ, Jenkins CNH, Wong C, Fordham D, Lai KQ, Bird JA, et al. Smoking cessation intervention among Vietnamese Americans: a controlled trial. Tobacco Control 1995;4(Suppl 1):S16‐24. CENTRAL

McVey 2000 {published data only}

McVey D, Stapleton J. Can anti‐smoking television advertising affect smoking behaviour? Controlled trial of the Health Education Authority for England's anti‐smoking TV campaign. Tobacco Control 2000;9(3):273‐82. CENTRAL

Mogielnicki 1986 {published data only}

Mogielnicki RP, Neslin S, Dulac J, Balestra D, Gillie E, Corson J. Tailored media can enhance the success of smoking cessation clinics. Journal of Behavioral Medicine 1986;9(2):141‐61. CENTRAL

North Coast QFL 1983 {published data only}

Egger G, Fitzgerald W, Frape G, Monaem A, Rubinstein P, Tyler C, et al. Results of a large scale antismoking campaign in Australia: North Coast 'Quit for Life' programme. British Medical Journal (Clinical Research Ed.) 1983;287(6399):1125‐8. CENTRAL
Egger G, Frape G, Mackay B. Applied problems of media use in health promotion ‐ the North Coast experience. New Doctor 1981;January:25‐9. CENTRAL

Stanford 3 City 1977 {published data only}

Farquhar JW, Maccoby N, Wood PD, Alexander JK, Breitrose H, Brown BW, et al. Community education for cardiovascular health. Lancet 1977;4(8023):1192‐5. CENTRAL
Kasl SV. Cardiovascular risk reduction in a community setting: some comments. Journal of Consulting and Clinical Psychology 1980;48(2):143‐9. CENTRAL
Leventhal H, Safer MA, Cleary PD, Gutmann M. Cardiovascular risk modification by community‐based programs for life‐style change: comments on the Stanford study. Journal of Consulting and Clinical Psychology 1980;48(2):150‐8. CENTRAL
Maccoby N, Farquhar JW, Wood PD, Alexander J. Reducing the risk of cardiovascular disease: effects of a community‐based campaign on knowledge and behavior. Journal of Community Health 1977;3(2):100‐14. CENTRAL
Meyer AJ. Skills training in a cardiovascular health education campaign. Journal of Consulting and Clinical Psychology 1980;48(2):129‐42. CENTRAL
Meyer AJ, Maccoby N, Farquhar JW. Reply to Kasl and Leventhal et al. Journal of Consulting and Clinical Psychology 1980;48(2):159‐63. CENTRAL

Sydney QFL 1986 {published data only}

Dwyer T, Pierce JP, Hannam CD, Burke N. Evaluation of the Sydney "Quit for Life" anti‐smoking campaign. Part 2. Changes in smoking prevalence. Medical Journal of Australia 1986;144(7):344‐7. CENTRAL
Pierce JP, Dwyer T, Frape G, Chapman S, Chamberlain A, Burke N. Evaluation of the Sydney "Quit For Life" anti‐smoking campaign. Part 1. Achievement of intermediate goals. Medical Journal of Australia 1986;144(7):341‐4. CENTRAL
Pierce JP, Macaskill P, Hill D. Long‐term effectiveness of mass media led antismoking campaigns in Australia. American Journal of Public Health 1990;80(5):565. CENTRAL

References to studies excluded from this review

Arizona 1998 {published data only}

Arizona Department of Health Services Tobacco Education & Prevention Program. 2004 Biennial Evaluation Report FY 2002‐2004. azmemory.azlibrary.gov/cdm/ref/collection/statepubs/id/1131 (accessed prior to 7 November 2017). CENTRAL
Lightwood J, Glantz S. Effect of the Arizona tobacco control program on cigarette consumption and healthcare expenditures. Social Science & Medicine (1982) 2011;72(2):166‐172. CENTRAL
Loeb J. Evaluation: methods and strategy for evaluation ‐ Arizona. Cancer 1998;83(Suppl 12A):2766‐9. CENTRAL
Meister JS. Designing an effective statewide tobacco control program ‐ Arizona. Cancer 1998;83(Suppl 12A):2728‐32. CENTRAL

ASSIST 2003 {published data only}

Manley M, Lynn W, Payne Epps R, Grande D, Glynn T, Shopland D. The American Stop Smoking Intervention Study for cancer prevention: an overview. Tobacco Control 1997;6(Suppl 2):S5‐S11. CENTRAL
Manley MW, Pierce JP, Gilpin EA, Rosbrook B, Berry C, Wun LM. Impact of the American Stop Smoking Intervention Study on cigarette consumption. Tobacco Control 1997;6(Suppl 2):S12‐S16. CENTRAL
Stillman F, Hartman A, Graubard B, Gilpin E, Chavis D, Garcia J, et al. The American Stop Smoking Intervention Study. Conceptual framework and evaluation design. Evaluation Review 1999;23(3):259‐80. CENTRAL
Stillman FA, Cronin KA, Evans WD, Ulasevich A. Can media advocacy influence newspaper coverage of tobacco: measuring the effectiveness of the American Stop Smoking Intervention Study's (ASSIST) media advocacy strategies. Tobacco Control 2001;10(2):137‐44. CENTRAL
Stillman FA, Hartman AM, Graubard BI, Gilpin EA, Murray DM, Gibson JT. Evaluation of the American Stop Smoking Intervention Study (ASSIST): a report of outcomes. Journal of the National Cancer Institute 2003;95(22):1681‐91. CENTRAL

A Su Salud 1990 {published data only}

Amezcua C, McAlister A, Ramirez A, Espinoza R. A Su salud. Health promotion in a Mexican‐American border community. In: Bracht N editor(s). Health promotion at the community level. Newbury Park, CA: Sage Publications, 1990. CENTRAL
McAlister AL, Ramirez AG, Amezcua C, Pulley LV, Stern MP, Mercado S. Smoking cessation in Texas‐Mexico border communities: a quasi‐experimental panel study. American Journal of Health Promotion 1992;6(4):274‐9. CENTRAL
Ramirez AG, McAlister AL. Mass media campaign‐‐A Su Salud. Preventive Medicine 1988;17(5):608‐21. CENTRAL

Barber 1990 {published data only}

Barber JJ, Grichting WL. Australia's media campaign against drug abuse. International Journal of the Addictions 1990;25(6):693‐708. CENTRAL

Boyd 1998 {published data only}

Boyd NR, Sutton C, Orleans CT, McClatchey MW, Bingler R, Fleisher L, et al. Quit Today! A targeted communications campaign to increase use of the cancer information service by African American smokers. Preventive Medicine 1998;27(5 Pt 2):S50‐60. CENTRAL

Brownson 1996 {published data only}

Brownson RC, Smith CA, Pratt M, Mack N, Jackson‐Thompson J, Dean CG, et al. Preventing cardiovascular disease through community‐based risk reduction: The Bootheel Heart Health Project. American Journal of Public Health 1996;86(2):206‐13. CENTRAL

Chicago I 1989 {published data only}

Flay BR, Gruder CL, Warnecke RB, Jason LA, Peterson P. One year follow‐up of the Chicago televised smoking cessation program. American Journal of Public Health 1989;79(10):1377‐80. CENTRAL
Gruder CL, Warnecke RB, Jason LA, Flay BR, Peterson P. A televised, self‐help, cigarette smoking cessation intervention. Addictive Behaviors 1990;15(6):505‐16. CENTRAL
Warnecke RB, Langenberg P, Gruder CL, Flay BR, Jason LA. Factors in smoking cessation among participants in a televised intervention. Preventive Medicine 1989;18:833‐46. CENTRAL

Chicago II 1992 {published data only}

Flay BR, McFall S, Burton D, Cook TD, Warnecke RB. Health behavior changes through television: the roles of de facto and motivated selection processes. Journal of Health and Social Behavior 1993;34(4):322‐35. CENTRAL
Warnecke RB, Flay BR, Kviz FJ, Gruder CL, Langenberg P, Crittenden KS, et al. Characteristics of participants in a televised smoking cessation intervention. Preventive Medicine 1991;20(3):389‐403. CENTRAL
Warnecke RB, Langenberg P, Wong SC, Flay BR, Cook TD. The second Chicago televised smoking cessation program: a 24‐month follow‐up. American Journal of Public Health 1992;82(6):835‐40. CENTRAL

Chow 2009 {published data only}

Chow CK, Joshi R, Gottumukkala AK, Raju K, Raju R, Reddy S, et al. Rationale and design of the Rural Andhra Pradesh Cardiovascular Prevention Study (RAPCAPS): A factorial, cluster‐randomized trial of 2 practical cardiovascular disease prevention strategies developed for rural Andhra Pradesh, India. American Heart Journal 2009;158(3):349‐55. CENTRAL
Joshi R, Chow CK, Raju PK, Raju KR, Gottumukkala AK, Reddy KS, et al. The Rural Andhra Pradesh Cardiovascular Prevention Study (RAPCAPS): a cluster randomized trial. Journal of the American College of Cardiology 2012;59(13):1188‐96. CENTRAL

Coeur en santé 1999 {published data only}

O'Loughlin J, Paradis G, Kishchuk N, Gray‐Donald K, Renaud L, Finès P, et al. Coeur en santé St‐Henri ‐ a heart health promotion program in Montreal, Canada: design and methods for evaluation. Journal of Epidemiology and Community Health 1995;49(5):495‐502. CENTRAL
O'Loughlin J, Paradis G, Meshefedijan G. Evaluation of two strategies for heart health promotion by direct mail in a low‐income urban community. Preventive Medicine 1997;26(5 Pt 1):745‐53. CENTRAL
O'Loughlin JL, Paradis G, Gray‐Donald K, Renaud L. The impact of a community‐based heart disease prevention program in a low‐income, inner‐city neighborhood. American Journal of Public Health 1999;89(12):1819‐26. CENTRAL
Paradis G, O'Loughlin J, Elliot M. Coeur en santé St‐Henri ‐ a heart health promotion program in a low income, low education neighborhood in Montreal, Canada: theoretical model and early field experience. Journal of Epidemiology and Community Health 1995;49(5):503‐12. CENTRAL

COMMIT 1995 {published data only}

Hyland A, Wakefield M, Higbee C, Szczypka G, Cummings KM. Anti‐tobacco television advertising and indicators of smoking cessation in adults: a cohort study. Health Education Research 2006;21(2):296‐302. CENTRAL
Sciandra RC, Wallack L, Johnson CL, Sadlik J, Thompson J. Activities to involve the smoking public in tobacco control in COMMIT. cancercontrol.cancer.gov/brp/tcrb/monographs/6/m6_6.pdf (accessed prior to 7 November 2017). CENTRAL
The COMMIT Research Group. Community Intervention Trial for Smoking Cessation (COMMIT): I. Cohort Results from a Four‐Year Community Intervention. American Journal of Public Health 1995;85(2):183‐92. CENTRAL
The COMMIT Research Group. Community Intervention Trial for Smoking Cessation (COMMIT): II. Changes in adult cigarette smoking prevalence. American Journal of Public Health 1995;85(2):193‐200. CENTRAL
Wallack L, Sciandra R. Media advocacy and public education in the Community Intervention Trial to Reduce Heavy Smoking (COMMIT). International Quarterly of Community Health Education 1990;11(3):205‐22. CENTRAL

Cummings 1987 {published data only}

Cummings KM, Sciandra R, Markello S. Impact of a newspaper mediated quit smoking program. American Journal of Public Health 1987;77(11):1452‐3. CENTRAL

Cummings 1993 {published data only}

Cummings KM, Sciandra R, Davis S, Rimer BK. Response to anti‐smoking campaign aimed at mothers with young children. Health Education and Research 1989;4(4):429‐37. CENTRAL
Cummings KM, Sciandra R, Davis S, Rimer BK. Results of an antismoking media campaign utilizing the Cancer Information Service. Journal of the National Cancer Institute Monograph 1993;14:113‐8. CENTRAL

Danaher 1984 {published data only}

Danaher BG, Berkanovic E, Gerber B. Mass media based health behavior change: televised smoking cessation program. Addictive Behaviors 1984;9(3):245‐53. CENTRAL

Davidson 1990 {published data only}

Davidson M. Wake up. Quit! The use of mass media and community participation to reduce smoking prevalence among young adults. Drug Education Journal of Australia 1990;4(2):151‐7. CENTRAL

Donovan 1984 {published data only}

Donovan RJ, Fisher DA, Armstrong BK. "Give it away for a day": an evaluation of Western Australia's first smoke free day. Community Health Studies 1984;8(3):301‐6. CENTRAL

Doxiadis 1985 {published data only}

Doxiadis SA, Trihopoulos DV, Phylactou HD. Impact of a nationwide anti‐smoking campaign. Lancet 1985;2(8457):712‐3. CENTRAL

Dubren 1977 {published data only}

Dubren R. Evaluation of a televised stop‐smoking clinic. Public Health Reports 1977;92(1):81‐4. CENTRAL

Dyer 1983 {published data only}

Dyer N. Evaluation of the BBC TV series "So you want to stop smoking". Conference on Smoking and Health, Fifth World Congress, Winnipeg Canada. Ottawa, Ontario, Canada: Canadian Council on Smoking and Health, 1983:46. CENTRAL
Dyer N. Smokers' Luck: Can a "shocking" programme change attitudes to smoking?. Addictive Behaviors 1983;8(1):43‐6. CENTRAL

Eiser 1978 {published data only}

Eiser JR, Rutton SR, Wober M. Can television influence smoking? Further evidence. British Journal of Addiction to Alcohol and Other Drugs 1978;73(3):291‐8. CENTRAL
Eiser JR, Sutton SR, Wober M. Can television influence smoking?. British Journal of Addiction to Alcohol and Other Drugs 1978;73(2):215‐9. CENTRAL

Etter 2005 {published data only}

Etter JF, Laszlo E. Evaluation of a poster campaign against passive smoking for World No‐Tobacco Day. Patient Education and Counseling 2005;57(2):190–8. CENTRAL

Etter 2007 {published data only}

Etter JF. Informing smokers on additives in cigarettes: A randomized trial. Patient Education and Counseling 2007;66(2):188–91. CENTRAL

EX campaign 2010 {published data only}

Duke J, Vallone D, Mowery P, McCausland K, Xiao H, Asche E, et al. LEGACY’S EX: Implementation and evaluation of a smoking cessation campaign. Annals of Behavioral Medicine 2008;35(1 Suppl):Paper Session 20. CENTRAL
Vallone DM, Duke JC, Cullen J, McCausland KL, Allen JA. Evaluation of EX: a national mass media smoking cessation campaign. American Journal of Public Health 2011;101(2):302–9. CENTRAL
Vallone DM, Duke JC, Mowery PD, McCausland KL, Xiao H, Costantino JC, et al. The impact of EX® Results from a pilot smoking‐cessation media campaign. American Journal of Preventive Medicine 2010;38(3 Suppl):S312–8. CENTRAL
Vallone DM, Niederdeppe J, Kalaydjian Richardson A, Patwardhan P, Niaura R, et al. A national mass media smoking cessation campaign: Effects by race/ethnicity and education. American Journal of Health Promotion 2011;25(5 Suppl):S38–50. CENTRAL

Frith 1997 {published data only}

Frith C, Roberts C, Kingdon A, Tudor‐Smith C. An evaluation of the 1996 No Smoking Day in Wales. Health Education Journal 1997;56(3):287‐95. CENTRAL

GASO 2002 {published data only}

Centers for Disease Control and Prevention. Impact of promotion of the Great American Smokeout and availability of over‐the‐counter nicotine medications, 1996. Morbidity and Mortality Weekly Report. Surveillance Summaries : MMWR 1997;46(37):867‐71. CENTRAL
Freels SA, Warnecke RB, Johnson TP, Flay BR. Evaluation of the effects of a smoking cessation intervention using the multilevel thresholds of change model. Evaluation Review 2002;26(1):40‐58. CENTRAL
Gritz ER, Carr CR, Marcus AC. Unaided smoking cessation: Great American Smokeout and New Year's Day quitters. Special Issue: Clinical research issues in psychosocial oncology. Journal of Psychosocial Oncology 1989;6(3‐4):217‐34. CENTRAL
Hantula DA, Stillman FA, Waranch HR. Can a mass‐media campaign modify tobacco smoking in a large organization? Evaluation of the Great American Smokeout in an urban hospital. Journal of Organizational Behavior Management 1993;13(1):33‐47. CENTRAL

Gredler 1981 {published data only}

Gredler B, Kunze M. Impact of a national campaign on smoking attitudes and patterns in Austria. International Journal of Health Education 1981;24(4):271‐9. CENTRAL
Gredler B, Kunze M. You can do without smoking ‐ a mass media campaign in Austria. Conference: Smoking and Health, Fifth World Congress, Winnipeg, Manitoba, Canada, 10‐15 Jul. 1983. [World Meeting Number 833 0083]CENTRAL

Heartbeat Wales 1998 {published data only}

Nutbeam D, Catford J. The Welsh Heart Programme evaluation strategy: progress, plans and possibilities. Health Promotion 1987;2(1):5‐18. CENTRAL
Nutbeam D, Smith C, Murphy S, Catford J. Maintaining evaluation designs in long term community based health promotion programmes: Heartbeat Wales case study. Journal of Epidemiology and Community Health 1993;47(2):127‐33. CENTRAL
Tudor Smith C, Nutbeam D, Moore L, Catford J. Effects of the Heartbeat Wales programme over five years on behavioural risks for cardiovascular disease: quasi‐experimental comparison of results from Wales and a matched reference area. BMJ 1998;316(7134):818‐22. CENTRAL

HEBS 1997 {published data only}

Donnan PT, Watson J, Platt S, Tannahill A, Raymond M. Predictors of successful quitting: findings from a six‐month evaluation of Smokeline campaign. Journal of Smoking Related Disorders 1994;5(suppl 1):271‐6. CENTRAL
Platt S, Tannahill A, Watson J, Fraser E. Effectiveness of antismoking telephone helpline: follow up survey. BMJ 1997;314(7091):1371‐5. CENTRAL
Ratcliffe J, Cairns J, Platt S. Cost effectiveness of a mass media‐led anti‐smoking campaign in Scotland. Tobacco Control 1997;6(2):104‐10. CENTRAL

Hill 2003 {published data only}

Borland R, Balmford J. Understanding how mass media campaigns impact on smokers. Tobacco Control 2003;12(Suppl II):ii45‐ii52. CENTRAL
Carroll T, Rock B. Generating Quitline calls during Australia's National Tobacco Campaign: effect of television advertisement execution and programme placement. Tobacco Control 2003;12(Suppl II):ii40‐ii44. CENTRAL
Donovan RJ, Boulter J, Borland R, Jalleh G, Carter O. Continuous tracking of the Australian National Tobacco Campaign: advertising effects on recall, recognition, cognitions, and behaviour. Tobacco Control 2003;12(Suppl II):ii30‐ii39. CENTRAL
Hill D, Carroll T. Australia's National Tobacco Campaign. Tobacco Control 2003;12(Suppl II):ii9‐ii14. CENTRAL
Hurley SF, Matthews JP. Cost‐effectiveness of the Australian National Tobacco Campaign. Tobacco Control 2008;17(6):379–84. CENTRAL
Miller CL, Wakefield M, Roberts L. Uptake and effectiveness of the Australian telephone Quitline service in the context of a mass media campaign. Tobacco Control 2003;12(Suppl II):ii53‐ii58. CENTRAL
Wakefield M, Freeman J, Donovan R. Recall and response of smokers and recent quitters to the Australian National Tobacco Campaign. Tobacco Control 2003;12(Suppl II):ii15‐ii22. CENTRAL
Wakefield MA, Durkin S, Spittal MJ, Siahpush M, Scollo M, Simpson JA, et al. Impact of tobacco control policies and mass media campaigns on monthly adult smoking prevalence. American Journal of Public Health 2008;98(8):1443–50. CENTRAL

Hunkeler 1990 {published data only}

Hunkeler EF, Davis EM, McNeil B, Powell JW, et al. Richmond quits smoking: a minority community fights for health. In: Bracht N editor(s). Health promotion at the community level. Newbury Park (CA): Sage Publications, 1990. CENTRAL

IHHP 2003 {published data only}

Mohammadifard N, Kelishadi R, Safavi M, Sarrafzadegan N, Sajadi F, Sadri GH, et al. Effect of a community‐based intervention on nutritional behaviour in a developing country setting: the Isfahan Healthy Heart Programme.. Public Health Nutrition 2009;12(9):1422‐30. CENTRAL
Sarraf‐Zadegan N, Sadri G, Malek Afzali H, Baghaei M, Mohammadi Fard N, Shahrokhi S, et al. Isfahan Healthy Heart Programme: A comprehensive integrated community‐based programme for cardiovascular disease prevention and control. Design, methods and initial experience. Acta Cardiologica 2003;58(4):309‐20. CENTRAL
Sarrafzadegan N, Kelishadi R, Esmaillzadeh A, Mohammadifard N, Rabiei K, Roohafza H, et al. Do lifestyle interventions work in developing countries? Findings from the Isfahan Healthy Heart Program in the Islamic Republic of Iran.. Bulletin of the World Health Organization 2009;87(1):39‐50. CENTRAL

Jason 1988 {published data only}

Jason LA, Tait E, Goodman D, Buckenberger L, Gruder CL. Effects of a televised smoking cessation intervention among low‐income and minority smokers. American Journal of Community Psychology 1988;16(6):863‐76. CENTRAL

Laugesen 2000 {published data only}

Laugesen M, Swinburn B. New Zealand's tobacco control programme 1985‐1998. Tobacco Control 2000;9(2):155‐62. CENTRAL

Ledwith 1984 {published data only}

Ledwith F. Immediate and delayed effects of postal advice on stopping smoking. Health Bulletin 1984;42(6):332‐44. CENTRAL
Ledwith, F. Effectiveness of the leaflet and individualized postal advice in aiding smoking cessation with large numbers of smokers. Conference: Smoking and Health, Fifth World Congress, Winnipeg, Manitoba, Canada, 10‐15 Jul. 1983. [World Meeting Number 833 0083]CENTRAL

Le Net 1977 {published data only}

Le Net M. Evaluation of a national campaign against smoking (France) [Evaluation d'une campagne nationale contre l'abus du tabac (France)]. Sozial und Praventivmedizin 1977;22(5):263‐75. CENTRAL

Leroux 1983 {published data only}

Leroux RS, Miller ME. Electronic media‐based smoking cessation clinic, in the USA. Hygie 1983;2(1):23‐37. CENTRAL

Lichtenstein 2008 {published data only}

Lichtenstein E, Boles SM, Lee ME, Hampson SE, Glasgow RE, Fellows J. Using radon risk to motivate smoking reduction II: randomized evaluation of brief telephone counseling and a targeted video. Health Education Research 2008;23(2):191‐201. CENTRAL

McAlister 2006 {published data only}

McAlister AL, Huang P, Ramirez AG. Settlement‐funded tobacco control in Texas: 2000‐2004 pilot project effects on cigarette smoking. Public Health Reports 2006;121(3):235‐8. CENTRAL

MHHP 1995 {published data only}

Dean AG, Shultz JM, Gust SW, Harty KC, Moen ME. Minnesota plan for nonsmoking and health: multidisciplinary approach to risk factor control. Public Health Reports 1986;101(3):270‐7. CENTRAL
Glasgow RE, Klesges RC, Mizes JS, Pechacek TF. Quitting smoking: strategies used and variables associated with success in a stop‐smoking contest. Journal of Consulting and Clinical Psychology 1985;53(6):905‐12. CENTRAL
Harty KC. Animals and butts: Minnesota's media campaign against tobacco. Tobacco Control 1993;2(4):271‐4. CENTRAL
Jacobs DR, Luepker RV, Mittelmark MB, Folsom AR, Pirie PL, Mascioli SR, et al. Community‐wide prevention strategies: Evaluation design of the Minnesota Heart Health Program. Journal of Chronic Diseases 1986;39(10):775‐88. CENTRAL
Lando HA, Pechacek TF, Fruetel J. The Minnesota Heart Health Program Community Quit and Win Contests. American Journal of Health Promotion 1994;9(2):85‐7, 124. CENTRAL
Lando HA, Pechacek TF, Pirie PL, Murray DM, Mittelmark MB, Lichtenstein E, et al. Changes in adult cigarette smoking in Minnesota Heart Health Program. American Journal of Public Health 1995;85(2):201‐8. CENTRAL
Luepker RV. An update and review of the Minnesota Heart Health Program. Annals of Epidemiology 1993;3(Suppl 5):S8‐S12. CENTRAL
Luepker RV, Murray DM, Jacobs DR, Mittelmark MB, Bracht N, Carlaw R, et al. Community education for cardiovascular disease prevention: risk factor changes in the Minnesota Heart Health Program. American Journal of Public Health 1994;84(9):1383‐93. CENTRAL
Mittelmark MB, Luepker RV, Jacobs DR, Bracht NF, Carlaw RW, Crow RS, et al. Community‐wide prevention of cardiovascular disease: education strategies of the Minnesota Heart Health Program. Preventive Medicine 1986;15(1):1‐17. CENTRAL
Pavlik J, Finnegan JR, Strickland D, Salmon CT, Viswanath K, Wackman DB. Increasing public understanding of heart disease: an analysis of data from the MHHP. Health Communication 1993;5:1‐20. CENTRAL

Millar 1987 {published data only}

Millar WJ, Naegele BE. Time to quit: community involvement in smoking cessation. Canadian Journal of Public Health 1987;78(2):109‐14. CENTRAL

Minnesota 2011 {published data only}

Centers for Disease Control and Prevention (CDC). Decrease in smoking prevalence‐‐Minnesota, 1999 ‐ 2010. Morbidity and Mortality Weekly Report. Surveillance Summaries : MMWR 2011;60(5):138‐41. CENTRAL

Mudde 1995 {published data only}

Mudde AN, de Vries H, Dolders MG. Evaluation of a Dutch community‐based smoking cessation intervention. Preventive Medicine 1995;24(1):61‐70. CENTRAL

Mudde 1999 {published data only}

Mudde AN, De Vries H. The reach and effectiveness of a national mass media‐led smoking cessation campaign in the Netherlands. American Journal of Public Health 1999;89(3):346‐50. CENTRAL

Multicity 1997 {published data only}

Cernada GP, Darity WA, Chen TTL, Winder AE, Benn S, Jackson R, et al. Mass media usage among black smokers: a first look. International Quarterly of Community Health Education 1989;10(4):347‐64. CENTRAL
Darity WA, Tuthill RW, Winder AE, Cernada GP, Chen TTL, Buchanan DR, et al. A multi‐city community based smoking research intervention project in the African‐American population. International Quarterly of Community Health Education 1997;17(2):117‐30. CENTRAL

New York {published data only}

Center for Disease Control and Prevention. Decline in Smoking Prevalence ‐ New York City, 2002 ‐ 2006. Morbidity and Mortality Weekly Report. Surveillance Summaries : MMWR 2007;56(24):604‐8. CENTRAL
Coady MH, Jasek J, Davis K, Kerker B, Kilgore EA, Perl SB. Changes in smoking prevalence and number of cigarettes smoked per day following the implementation of a comprehensive tobacco control plan in New York City. Journal of Urban Health 2012;89(5):802‐8. [DOI: 10.1007/s11524‐012‐9683‐9]CENTRAL
Davis KC, Farrelly MC, Duke J, Kelly L, Willett J. Antismoking media campaign and smoking cessation outcomes, New York State, 2003 ‐ 2009. Preventing Chronic Disease 2012;9:E40. CENTRAL
Farrelly MC, Davis KC, Nonnemaker JM, Kamyab K, Jackson C. Promoting calls to a quitline: quantifying the influence of message theme, strong negative emotions and graphic images in television advertisements. Tobacco Control 2011;20(4):279‐84. CENTRAL
Farrelly MC, Duke JC, Davis KC, Nonnemaker JM, Kamyab K, Willett JG, et al. Promotion of smoking cessation with emotional and/or graphic antismoking advertising. American Journal of Preventive Medicine 2012;43(5):475‐82. CENTRAL

North Karelia 1998 {published data only}

Korhonen T, Uutela A, Korhonen HJ, Puska P. Impact of mass media and interpersonal health communication on smoking cessation attempts: a study in North Karelia, 1989‐1996. Journal of Health Communication 1998;3(2):105‐18. CENTRAL
Puska P, Nissinen A, Tuomilehto J, Salonen JT, Koskela K, McAlister A, et al. The community‐based strategy to prevent coronary heart disease: conclusions from the ten years of the North Karelia project. Annual Review of Public Health 1985;6:147‐93. CENTRAL
Vartiainen E, Puska P, Jousilahti P, Korhonen HJ, Tuomilehto J, Nissinen A. Twenty‐year trends in coronary risk factors in north Karelia and in other areas of Finland. Interantional Journal of Epidemiology 1994;23(3):495‐504. CENTRAL

Oregon 1999 {published data only}

Bjornson W, Moore JM. Designing an effective counteradvertising campaign ‐ Oregon. Cancer 1998;83(Suppl 12A):2752‐4. CENTRAL
Centers for Disease Control and Prevention. Decline in cigarette consumption following implementation of a comprehensive tobacco prevention and education program‐Oregon, 1996‐1998. Morbidity and Mortality Weekly Report. Surveillance Summaries : MMWR 1999;48(7):140‐3. CENTRAL
Moore JM. Designing an effective statewide tobacco control program ‐ Oregon. Cancer 1998;83(Suppl 12A):2733‐5. CENTRAL
Moore JM, Bjornson W. Evaluation: methods and strategy for evaluation ‐ Oregon. Cancer 1998;83(Suppl 12A):2770‐2. CENTRAL

Osler 1993 {published data only}

Osler M, Jespersen NB. The effect of a community‐based cardiovascular disease prevention project in a Danish municipality. Danish Medical Bulletin 1993;40(4):485‐9. CENTRAL

Pallonen 1994 {published data only}

Pallonen UE, Leskinen L, Prochaska JO, Willey CJ, Kaariainen R, Salonen JT. A 2‐year self‐help smoking cessation manual intervention among middle‐aged Finnish men: an application of the transtheoretical model. Preventive Medicine 1994;23(4):507‐14. CENTRAL

Perkins 1986 {published data only}

Perkins KA, Scott RR. A low‐cost environmental intervention for reducing smoking among cardiac patients. International Journal of the Addictions 1986;21(11):1173‐82. CENTRAL

Programa Latino 1994 {published data only}

Marin BV, Perez‐Stable EJ, Marin G, Hauck WW. Effects of a community intervention to change smoking behavior among Hispanics. American Journal of Preventive Medicine 1994;10(6):340‐7. CENTRAL
Marín G, Marín BV, Pérez‐Stable EJ, Sabogal F, Otero‐Sabogal R. Changes in information as a function of a culturally appropriate smoking cessation community intervention for Hispanics. American Journal of Community Psychology 1990;18(6):847‐64. CENTRAL
Pérez‐Stable EJ, Marin BV, Marin G. A comprehensive smoking cessation program for the San Francisco Bay area Latino community: Programa Latino para Dejar de Fumar. American Journal of Health Promotion 1993;7(6):430‐42. CENTRAL

Sansores 2002 {published data only}

Sansores RH, Giraldo‐Buitrago F, Valdelamar‐Vazquez F, Ramirez‐Venegas A, Sandoval RA. The impact of mass media on an anti‐tobacco campaign. Salud Publica Mexico 2002;44(Suppl 1):S101‐S108. CENTRAL

Secker‐Walker 2000 {published data only}

Secker‐Walker RH, Flynn BS, Solomon LJ, Skelly JM, Dorwaldt AL, Ashikaga T. Helping Women Quit Smoking: Results of a Community Intervention Program. American Journal of Public Health 2000;90(6):940‐6. CENTRAL

Sogaard 1992 {published data only}

Sogaard AJ, Fonnebo V. Self‐reported change in health behaviour after a mass media‐based health education campaign. Scandinavian Journal of Psychology 1992;33(2):125‐34. CENTRAL

Stanford 5 City 2000 {published data only}

Farquhar JW, Fortmann SP, Flora JA, Taylor CB, Haskells WI, Williams PT, et al. Effects of communitywide education on cardiovascular disease risk factors. The Stanford Five‐City Project. JAMA 1990;264(3):359‐65. CENTRAL
Farquhar JW, Fortmann SP, Maccoby N, Haskells WL, Williams PT, Flora JA, et al. The Stanford Five‐City Project: design and methods. American Journal of Epidemiology 1985;122(2):323‐34. CENTRAL
Fortmann SP, Taylor CB, Flora JA, Jatulis DE. Changes in adult cigarette smoking prevalence after 5 years of community health education: the Stanford Five‐City Project. American Journal of Epidemiology 1993;137(1):82‐96. CENTRAL
Fortmann SP, Varady AN. Effects of a community‐wide health education program on cardiovascular disease morbidity and mortality: The Stanford Five‐City Project. American Journal of Epidemiology 2000;152(4):316‐23. CENTRAL
Jackson C, WInkleby MA, Flora JA. Use of educational resources for cardiovascular risk reduction in the Stanford Five Cit Project. American Journal of Preventive Medicine 1991;7(2):82‐8. CENTRAL
Rimal RN, Flora JA, Schooler C. Achieving improvements in overall health orientation: Effects of campaign exposure, information seeking and health media use. Communication Research 1999;26(3):322‐48. CENTRAL
Sallis JF, Flora JA, Fortmann SP, Taylor CB, Maccoby N. Mediated smoking cessation programs in the Stanford Five‐City Project. Addictive Behaviors 1985;10(4):441‐3. CENTRAL
Schooler C, Flora JA, Farquar JW. Moving Project toward synergy: media supplementation in the Stanford Five City. Communication Research 1993;20(4):587‐610. CENTRAL
Winkleby MA, Flora JA, Kraemer HC. A community based heart disease intervention: predictors of change. American Journal of Public Health 1994;84(5):767‐72. CENTRAL
Winkleby MA, Taylor CB, Jatulis D. The long‐term effects of a cardiovascular disease prevention trial: the Stanford Five City Project. American Journal of Public Health 1996;86(12):1773‐9. CENTRAL

Stevens 2002 {published data only}

Stevens W, Thorogood M, Kayikki S. Cost‐effectiveness of a community anti‐smoking campaign targeted at a high risk group in London. Health Promotion International 2002;17(1):43‐50. CENTRAL

Sussman 1994 {published data only}

Sussman S, Dent CW, Wang E, Cruz NT, Sanford D, Johnson CA. Participants and nonparticipants of a mass media self‐help smoking cessation program. Addictive Behaviors 1994;19(6):643‐54. CENTRAL

Sutton 1987 {published data only}

Hallett R, Sutton SR. Factors influencing the decision to attempt to stop smoking in a media‐based smoking intervention programme. Health Education Research 1986;1(3):163‐73. CENTRAL
Sutton SR, Hallett R. Experimental evaluation of the BBC TV series "So You Want To Stop Smoking?". Addictive Behaviors 1987;12(4):363‐6. CENTRAL

Terry‐McElrath 2013 {published data only}

Terry‐McElrath YM, Emery S, Wakefield MA, O'Malley PM, Szczypka G, Johnston LD. Effects of tobacco‐related media campaigns on smoking among 20‐30‐year‐old adults: longitudinal data from the USA. Tobacco Control 2013;22(1):38‐45. CENTRAL

TIPS {published data only}

Ayers JW, Althouse BM. "TIPS" "Tips From Former Smokers" Can Benefit From Considering All Available Data: Reply to McAfee et al. American Journal of Preventive Medicine 2015;49(6):e133‐4. CENTRAL
Centers for Disease Control and Prevention (CDC). "TIPS"Increases in quitline calls and smoking cessation website visitors during a national tobacco education campaign‐‐March 19‐June 10, 2012. Morbidity and Mortality Weekly Report. Surveillance Summaries : MMWR 2012;61(34):667‐70. CENTRAL
Centers for Disease Control and Prevention (CDC). Impact of a national tobacco education campaign on weekly numbers of quitline calls and website visitors‐‐United States, March 4‐June 23, 2013. Morbidity and Mortality Weekly Report. Surveillance Summaries : MMWR 2013;62(37):763‐7. CENTRAL
Davis KC, Alexander RL, Shafer P, Mann N, Malarcher A, Zhang L. The Dose‐Response Relationship Between Tobacco Education Advertising and Calls to Quitlines in the United States, March‐June, 2012. Preventing Chronic Disease 2015;12:E191. CENTRAL
Davis KC, Duke J, Shafer P, Patel D, Rodes R, Beistle D. Perceived Effectiveness of Antismoking Ads and Association with Quit Attempts Among Smokers: Evidence from the Tips From Former Smokers Campaign. Health Communication 2017;32:931‐8. CENTRAL
Duke JC, Davis KC, Alexander RL, MacMonegle AJ, Fraze JL, Rodes RM, et al. Impact of a U.S. antismoking national media campaign on beliefs, cognitions and quit intentions. Health Education Research 2015;30(3):466‐83. CENTRAL
Huang LL, Thrasher JF, Abad EN, Cummings KM, Bansal‐Travers M, Brown A, et al. The U.S. National Tips From Former Smokers Antismoking Campaign: Promoting Awareness of Smoking‐Related Risks, Cessation Resources, and Cessation Behaviors. Health Education & Behavior 2015;42(4):480‐6. CENTRAL
Kim A, Hansen H, Duke J, Davis K, Alexander R, Rowland A, et al. Does digital ad exposure influence information‐seeking behavior online? Evidence from the 2012 Tips From Former Smokers national tobacco prevention campaign. Journal of Medical Internet Research 2016;18(3):e64. CENTRAL
McAfee T. Encouraging smokers to talk with their physicians about quitting. JAMA 2013;309(22):2329‐30. CENTRAL
McAfee T, Davis KC, Alexander RL, Pechacek TF, Bunnell R. Effect of the first federally funded US antismoking national media campaign. Lancet 2013;382(9909):2003‐11. CENTRAL
McAfee T, Davis KC, Cox SN, Beistle DM. Google Searches or Quit Attempts as a Success Measure for an Antismoking Campaign. American Journal of Preventive Medicine 2015;49(6):e131‐2. CENTRAL
McAfee T, Davis KC, Shafer P, Patel D, Alexander R, Bunnell R. Increasing the dose of television advertising in a national antismoking media campaign: results from a randomised field trial. Tobacco Control 2017;26(1):19‐28. CENTRAL
Neff LJ, Patel D, Davis K, Ridgeway W, Shafer P, Cox S. Evaluation of the National Tips From Former Smokers Campaign: the 2014 Longitudinal Cohort. Preventing Chronic Disease 2016;13:E42. CENTRAL
Vickerman KA, Zhang L, Malarcher A, Mowery P, Nash C. Cessation Outcomes Among Quitline Callers in Three States During a National Tobacco Education Campaign. Preventing Chronic Disease 2015;12:E110. CENTRAL
Xu X, Alexander RL, Simpson SA, Goates S, Nonnemaker JM, Davis KC, et al. A cost‐effectiveness analysis of the first federally funded antismoking campaign. American Journal of Preventive Medicine 2015;48(3):318‐25. CENTRAL
Zhang L, Malarcher A, Babb S, Mann N, Davis K, Campbell K, et al. The Impact of a National Tobacco Education Campaign on State‐Specific Quitline Calls. American Journal of Health Promotion 2016;30(5):374‐81. CENTRAL
Zhang L, Vickerman K, Malarcher A, Carpenter K. Changes in Quitline Caller Characteristics During a National Tobacco Education Campaign. Nicotine & Tobacco Research 2015;17(9):1161‐6. CENTRAL
Zhang L, Vickerman K, Malarcher A, Mowery P. Intermediate cessation outcomes among quitline callers during a national tobacco education campaign. Nicotine & Tobacco Research 2014;16(11):1478‐86. CENTRAL

TV Finland 1992 {published data only}

Korhonen HJ, Niemensivu H, Piha T, Koskela K, Wiio J, Johnson CA, et al. National TV smoking cessation program and contest in Finland. Preventive Medicine 1992;21(1):74‐87. CENTRAL
Korhonen HJ, Puska P, Lipand A, Kasmel A. Combining mass media and contest in smoking cessation. An experience from a series of national activities in Finland. Hygie 1993;12(1):15. CENTRAL
Koskela K, Puska P, Smolander A. Use of TV in national smoking reduction in Finland. Conference: Smoking and Health, Fifth World Congress, Winnipeg, Manitoba, Canada, 10‐15 Jul. 1983. [World Meeting Number 833 0083]CENTRAL
McAlister A, Puska P, Koskela K, Pallonen U, Maccoby N. Mass communication and community organization for public health education. American Psychologist 1980;35(4):375‐9. CENTRAL
Puska P, Koskela K, McAlister A, Pallonen U, Vartiainen H, Homan K. A comprehensive television smoking cessation program in Finland. International Journal of Health Education 1979;22(4 Supp):1‐28. CENTRAL
Puska P, Wiio J, McAlister A, Koskela K, Smolander A, Pekkola J, et al. Planned use of mass media in national health promotion: the "Keys to Health" TV program in 1982 in Finland. Canadian Journal of Public Health 1985;76(5):336‐42. CENTRAL

Valois 1996 {published data only}

Valois RF, Adams KG, Kammermann SK. One‐year evaluation results from CableQuit: a community cable television smoking cessation pilot program. Journal of Behavioral Medicine 1996;19(5):479‐99. CENTRAL

Van Assema 1994 {published data only}

Van Assema P, Steenbakkers M, Kok G, Eriksen M, De Vries H. Results of the Dutch community project "Healthy Bergeyk". Preventive Medicine 1994;23(3):394‐401. CENTRAL

Webb 2009 {published data only}

Webb MS. Culturally specific interventions for African American smokers: An efficacy experiment. Journal of the National Medical Association 2009;101(9):927‐35. CENTRAL

Wewers 1991 {published data only}

Wewers ME, Ahijevych K, Page JA. Evaluation of a mass media community smoking cessation campaign. Addictive Behaviors 1991;16(5):289‐94. CENTRAL

Wheeler 1988 {published data only}

Wheeler RJ. Effects of a community‐wide smoking cessation program. Social Science & Medicine (1982) 1988;27(12):1387‐92. CENTRAL

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Characteristics of studies

Characteristics of included studies [ordered by study ID]

California TCP 2003

Methods

Country: USA.
Objective: to reduce tobacco use in California ‒ reduce exposure to ETS, counter pro‐tobacco influences, promote tobacco use cessation, reduce youth access to tobacco products and to promote social norm of not accepting tobacco.
Study sites: state of California (intervention group), rest of USA (control group).
Programme name: California Tobacco Control Program (CTCP).
Design: interrupted time series study design using data from national and California population surveys.
Analysis: regression models, 2‐tailed statistical tests; smoking behaviour was measured in several population‐based cross‐sectional surveys conducted nationally and in California for several years before the start of the programme, and during and after the programme.
No power estimates.

Participants

Population of study sites: CA 1999 ‒ 23,788,205; rest of US ‒ not provided.
Target population: adult smokers, adolescents, general audience, minority populations (Hispanic, Asian, African Americans).
Age: 18+ and 15+ (depending on survey).
Ethnicity: White, Hispanic, Asian, African Americans, Chinese, Vietnamese, Korean.

Interventions

Theoretical basis: social diffusion model. Yr started: 1989.

Duration of media campaign: April 1990 to June 1991; then funding reduced, campaign restarted October 1992 to May 1993; funding decreased till mid‐1996; campaign restored January 1997 to June 1998, then funding reduced again.
Focus groups were used in development of messages. Ongoing independent evaluation.
Components:

(i) statewide media campaign disseminating anti‐tobacco messages;

(ii) local tobacco control initiatives, policy development and public education programmes;

(iii) school‐based tobacco prevention programmes, activities and policies.

Media campaign includes paid commercials and PSAs for TV, radio, outdoor advertisements, newspaper ads and public relations activities, in English, Spanish, Cantonese, Mandarin, Vietnamese, Korean, Laotian, Cambodian, Japanese, Hmong. Messages were designed to de‐glamorise smoking for young people, show the dishonesty of the tobacco industry, encourage smokers to quit, publicize the health risks of exposure to environmental tobacco smoke.
Community programme includes a variety of activities implemented by county health departments and community‐based organization, focused on changing community norms regarding tobacco use, getting support for decreasing tobacco advertising and sponsorship, reducing environmental tobacco smoke (in the workplaces, vehicles and at home), announce the statewide telephone quitline.
Year started: 1990.
Duration: ongoing

Outcomes

Smoking prevalence, quit ratio (% of ever‐smokers now ex‐smokers), per capita cigarette consumption (based on aggregated sales data).
Definitions: smoker ‒ currently smoking and 100+ lifetime cigs; in more recent surveys (NHIS since 1993, CPS since 1992, BRFS/CATS since 1994) respondents were asked if they currently smoked 'every day', 'some days' or 'not at all'; in CTS, prevalence was based on 'smoke now' question, while in the other surveys smokers must have reported smoking at least 100 cigs in their lifetime; former smoker ‒ smoked at least 1+ cigs in the past 30 days and does not currently smoke; quit ratio ‒ for a given year % of ever‐smokers (current smokers + former smokers) who were now former smokers.
Questionnaire: by telephone and in person.
Biochemical confirmation of abstinence: none reported.
Measured at baseline (1978), then yearly to 2002; campaign ongoing.

Notes

Intermediate measures:

health beliefs, health‐enhancing attitude score, percentage of smokers thinking about quitting, attempts to quit, support for further increase in tax on tobacco with funds devoted to tobacco control, support for ban on tobacco advertising and tobacco company sponsorship, support for smoking restrictions in public place, smokefree worksites, home smoking bans, nonsmokers exposed to ETS at work.
Process measures:

media weight, campaign awareness/reach, expenditures and cost effectiveness.
Intermediate measures and process measures were assessed in California only, without other state comparisons.

The evaluation of the campaign continues with California Smokers' Cohort (CSC), but without comparison to control group.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Not RCT: the results of the programme were compared with the smoking prevalence of the rest of the USA in interrupted time series study design using data from national and California population survey.

Allocation concealment (selection bias)

High risk

Not RCT: the results of the programme were compared with the smoking prevalence of the rest of the USA in interrupted time series study design using data from national and California population survey.

Blinding of participants and personnel (performance bias)
All outcomes

High risk

Not done due to the nature of the intervention, making people aware of the programme.

Blinding of outcome assessment (detection bias)
All outcomes

High risk

Self‐reported cigarette consumption from population‐based surveys.

Incomplete outcome data (attrition bias)
All outcomes

High risk

Cross‐sectional surveys, no cohort follow‐up; response rates 71% to 99%.

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in Methods section.

Other bias

Unclear risk

Insufficient information:

ITS assessment (Pierce 1998) —

clearly defined point in time when the intervention occurred DONE

At least three data points before and three after the intervention DONE

The intervention is independent of other changes NOT CLEAR

There are sufficient data points to enable reliable statistical inference NOT CLEAR

Formal test for trend NOT CLEAR

Intervention unlikely to affect data collection DONE

Blinded assessment of primary outcome NOT DONE

Completeness of data set NOT CLEAR

Reliable primary outcome NOT CLEAR

ITS assessment (Siegel 2000) —

Clearly defined point in time when the intervention occurred DONE

At least three data points before and three after the intervention DONE

The intervention is independent of other changes NOT CLEAR

There are sufficient data points to enable reliable statistical inference NOT CLEAR

Formal test for trend NOT CLEAR

Intervention unlikely to affect data collection DONE

Blinded assessment of primary outcome NOT DONE

Completeness of data set NOT CLEAR

Reliable primary outcome NOT CLEAR

CORIS 1997

Methods

Country: South Africa.
Objective: to reduce levels of coronary heart disease factors (e.g. high blood pressure, high blood cholesterol, stress, sedentary life style and smoking).
Study sites: Robertson, Swellenden and Riversdale in South‐Western Cape Province.
Programme name: The Coronary Risk Factor Study (CORIS).
Design: quasi‐experimental study (i) Robertson (mass media intervention + community‐based intervention) (ii) Swellenden (similar mass media intervention alone), (iii) Riversdale (control). Only Swellenden and Riversdale comparison included in our review.
Analysis: t‐tests paired and unpaired with and without covariance adjustment, two‐tailed P values < 0.01, Chi², with individual as the unit of analysis.
No power estimates.

Participants

Population of study sites: 1980 census estimate Swellenden 6176 (low‐intensity mass media intervention), Riversdale 6049 (control).
Target population: all Afrikaner adult inhabitants.
Age: 15 to 64 at baseline and at 12 yr follow‐up, 15 to 68 at 4 yr follow up.
Sex: M & F.
Ethnicity: > 95% White (Afrikaner).

Interventions

Theoretical basis: not specified. Year started: baseline survey 1979, mass media intervention 1980.

Duration: 4 yrs.
Components: structured mass media health education intervention addressing each of the risk factors. Baseline survey 1 yr before the campaign and results reported to the participants as the first intervention. Interviewers and observers centrally trained, standardized instruments.
Initial 4 m general awareness campaign followed by risk factor programmes, repeated during subsequent 2 yrs, singly and in combinations, with new materials and varied intensity and duration. The intervention included: blood pressure screening stations + educational materials, billboards, posters, mailings, frequent news items, health messages on electricity accounts and special supplement in the local newspaper. During 2nd and 3rd yrs frequency of the billboards, posters and mailings was halved, but news items and supplement remained the same.
Robertson (High‐intensity arm) received 5‐day smoking cessation seminars not offered to the low‐intensity or control communities.

Outcomes

Smoking prevalence, cigarette consumption, quit rate; smoking behaviour was measured in cross‐sectional surveys at baseline and at 4 yrs, in cohort identified at baseline. Additional follow‐up survey at 12 yrs from baseline.
Definitions: Smoker ‒ smoking on average at least 1 cig (= 1 g tobacco) daily. Ex‐smoker ‒ abstinent for at least 3 m before study start.
Questionnaire: in‐person interview.
Participation rates: 60% to 74% at baseline, 56% to 70% at 4 yr survey.
Biochemical confirmation of abstinence: none reported.
Measured at baseline, 4 yrs, 12 yrs.

Notes

Intermediate measures: 43‐item questionnaire on knowledge of risk factors, diet and attitudes at baseline, but only knowledge score was reported.
Process measures: media weight, intervention costs.
We have not included the Robertson community intervention (high intensity) in this review, but it is covered fully in Secker‐Walker 2002.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Quasi‐experimental with two intervention towns, Robertson receiving a mass media intervention and a community‐based intervention, Swellenden receiving a similar mass media intervention alone, and Riversdale acting as the comparison town. ONLY COMPARISON SWELLENDEN AND RIVERSDALE INCLUDED IN THE ANALYSIS

Allocation concealment (selection bias)

High risk

Quasi‐experimental, no randomisation attempt

Blinding of participants and personnel (performance bias)
All outcomes

High risk

Not done due to the nature of the intervention.

Blinding of outcome assessment (detection bias)
All outcomes

High risk

Self‐reported cigarette consumption.

Incomplete outcome data (attrition bias)
All outcomes

High risk

Cross‐sectional surveys in the whole population, 56% of participants re‐surveyed after 4 yrs (cohort follow‐up); response rates for cross‐sectional surveys 62% to 74%.

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in methods section.

Other bias

Low risk

No other bias identified.

Jenkins 1997

Methods

Country: USA.
Objective: to lower smoking prevalence among Vietnamese‒American men.
Study sites: San Francisco and Alameda counties, California (intervention), and Houston, Texas (control).
Programme name: Smoking Cessation among Vietnamese‐American Men ‒ II.
Design: quasi‐experimental.
Analysis: Chi² , t‐tests, multiple logistic regression, with individual as the unit of analysis. Smoking behaviour assessed in cross‐sectional surveys at baseline and 2 yrs.
No power estimates.

Participants

Age: 18+, Vietnamese‒American male current smokers (smoked a cigarette during prior week).

Interventions

Theoretical basis: not specified. Yr started: 1990; Duration: 2yrs.
Components:

15 m uncontrolled pilot campaign. Then

(i) Newspaper and magazine articles in Vietnamese language, a videotape broadcast ×2 on Vietnamese‐language TV, calendar, bumper stickers, lapel buttons, 3 posters, 2 brochures and self‐help 'quit kit'.
(ii) Anti‐tobacco counter‐advertising campaign included billboards (3 different types), newspaper ads and paid TV ads.
(iii) Short anti‐tobacco presentations at community events, 'Saturday' schools in Vietnamese language for students, courses of smoking cessation counselling for Vietnamese physicians, Vietnamese 'no smoking' signs and smoking control ordinances to local businesses and restaurants.

Outcomes

Smoking prevalence, cigarette consumption, quit rates.
Definitions: current smoker ‒ answered yes to 2 questions: (a) ever smoked a cig (b) smoked a cig during the previous week; former smoker ‒ answered yes to the 1st question and no to 2nd; never‐smoker ‒ answered no to both questions; recent quitter ‒ quit smoking during 2 yrs before either pretest or post‐test interview.
Questionnaire: by telephone.
Biochemical confirmation of abstinence: none.
Measured at baseline and 2 yrs.

Notes

Intermediate measures: motivation to quit, self‐efficacy, quit attempts assessed at baseline and follow‐up.
Process measures: media weight, awareness/reach.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Quasi‐experimental, with SF and Alameda County chosen as the intervention area and Houston, Texas as the comparison area.

Allocation concealment (selection bias)

High risk

Quasi‐experimental, with SF and Alameda County chosen as the intervention area and Houston, Texas as the comparison area.

Blinding of participants and personnel (performance bias)
All outcomes

High risk

Not done due to the nature of the intervention.

Blinding of outcome assessment (detection bias)
All outcomes

Unclear risk

Self‐reported smoking ‒ telephone interview, not clear if interviewers were aware of the intervention received.

Incomplete outcome data (attrition bias)
All outcomes

High risk

cross‐sectional surveys, no cohort follow up, response rates 82% to 94%.

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in Methods section.

Other bias

Low risk

No other bias identified.

Massachusetts 2003

Methods

Country: USA.
Objectives: to reduce tobacco use in Massachusetts residents.
Study sites: state of Massachusetts (intervention) and the rest of the USA (all states except Alaska, Arkansas, California, Kansas, Nevada, New Jersey, Rhode Island, Wyoming).
Programme name: Massachusetts Tobacco Control Program (MTCP).
Design: interrupted time series study design using data from national and Massachusetts population surveys.
Analysis: regression models, test of model coefficients, test for trends, Chi². Smoking behaviour was measured in several population‐based cross‐sectional surveys conducted nationally and in Massachusetts for several years before the start of the programme, and during and after the programme.
No power estimates.

Participants

Population of study sites: not given.
Target population: adult smokers, adolescent, general audience.
Age: 18+
Sex: M & F.

Interventions

Theoretical basis: social diffusion theory.

Year started: baseline measures 1989, intervention 1993;

Duration: still ongoing but with very low funding.
Formative research in the message development. Systematic monitoring of the campaign and independent evaluations of the programme.
Components:

(i) high‐profile statewide media campaign, targeting adult smokers, youth and general audience;

(ii) programme to establish community‐based tobacco control efforts;

(iii) statewide initiatives.

Mass media campaign started in October 1993, still ongoing, with100+ ads for TV, radio, billboards, newspapers and public relations events. Two aims: Public Education Media Campaign, focused on general population, raising awareness and explaining tobacco control issues; and Strategic and Targeted Marketing (tailored messages for selected populations). The intention of messages was to de‐glamorise smoking among young people, show the dishonesty of the tobacco industry, encourage smokers to quit, inform about the health risks of exposure to ETS. Media ads were translated into Spanish, Portuguese, Chinese and Vietnamese.
Community‐based tobacco control run from existing institutions, e.g. local health departments, plus new initiatives such as cessation counselling and/or public information and education services, promoting local policies, regulations, and ordinances limiting smoking in public places or restricting youth access to tobacco. Statewide initiatives included telephone hotline, technical assistance in establishing worksite smoking policies, efforts to build tobacco control infrastructure.

Outcomes

Smoking prevalence, per capita cigarette consumption (based on aggregated sales data).
Definitions: current smoker ‒ (BRFSS surveys): answered 'yes' to the questions 'Have you ever smoked at least 100 cigs in your entire life?' and 'Do you smoke cigs now?', after 1996 the latter question was changed to 'Do you now smoke cigs everyday, some days or not at all?' and current smokers post‐1996 answered 'every day' or 'some days; in MTS ‒ adult who reported to have smoked 100 cigs in their lifetime and currently smoked 'every day or some days'; quit success ‒ smoking at baseline and reporting smoking 'not at all' at the time of the interview.
Questionnaire: by telephone.
Biochemical confirmation of abstinence: none reported.
Measured at baseline, then yearly from 1993 to 2000; campaign ongoing.

Notes

Intermediate measures: attitudes and health beliefs about smoking, support for further increase in tax on tobacco with funds devoted to tobacco control, support for ban on vending machines, support for ban on sponsorship of sports and cultural events by tobacco companies, smokefree worksites, homes with smoking ban, ETS at work, support for restricting smoking in public buildings, and for some form of restriction on smoking in restaurants, social pressure to quit.
Process measures: media weight, campaign awareness/reach, expenditures and cost‐effectiveness.
Intermediate and process measures were assessed in survey carried out in Massachusetts, without other state comparisons

Tobacco control efforts ongoing, information about smoking and quitting in Massachusetts is available from BRFSS surveys, no control group, no evaluation.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Not RCT: the results of the programme were compared with the smoking prevalence of the rest of the USA in interrupted time series study design using data from national and California population survey.

Allocation concealment (selection bias)

High risk

Not RCT: the results of the programme were compared with the smoking prevalence of the rest of the USA in interrupted time series study design using data from national and California population survey

Blinding of participants and personnel (performance bias)
All outcomes

High risk

Not done due to the nature of the intervention, making people aware of the programme.

Blinding of outcome assessment (detection bias)
All outcomes

Unclear risk

Self‐reported cigarette consumption from population based surveys.

Incomplete outcome data (attrition bias)
All outcomes

Unclear risk

Cross‐sectional surveys, no cohort follow up; response rates 43% to 84%.

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in Methods section.

Other bias

Unclear risk

Insufficient information:

ITS assessment:

Clearly defined point in time when the intervention occurred DONE

At least three data points before and three after the intervention DONE

The intervention is independent of other changes NOT CLEAR

There are sufficient data points to enable reliable statistical inference NOT CLEAR

Formal test for trend NOT CLEAR

Intervention unlikely to affect data collection DONE

Blinded assessment of primary outcome NOT DONE

Completeness of data set NOT CLEAR

Reliable primary outcome NOT CLEAR

ITS assessment:

Clearly defined point in time when the intervention occurred DONE

At least three data points before and three after the intervention DONE

The intervention is independent of other changes NOT CLEAR

There are sufficient data points to enable reliable statistical inference NOT CLEAR

Formal test for trend NOT CLEAR

Intervention unlikely to affect data collection DONE

Blinded assessment of primary outcome NOT DONE

Completeness of data set NOT CLEAR

Reliable primary outcome NOT CLEAR

McAlister 2004

Methods

Country: USA.
Objective: to promote smoking cessation among adults.
Study site: eastern Texas.
Programme name: Texas Tobacco Prevention Pilot Initiative.
Design: quasi‐experimental study, 13 intervention regions and 1 control region; 3 levels of media exposure (none, low‐level, high‐level) and 5 community programme options (no programme, law enforcement programmes only, cessation programmes only, school‐community prevention programmes only, or all programmes combined); for this review only areas with media programmes and without community programmes were included ‒ 4 areas: 2 low‐level media (Liberty‐Chambers, Northeast Harris County), one intensive media (Tyler County) and one control (Bell County).
Analysis: logistic regression, Chi², Pearson's Correlation, one‐way ANOVA, with individual as unit of analysis; smoking behaviour was assessed in a cohort of smokers identified at baseline and at follow‐up 7 m later; smoking prevalence was assessed in 2 independent cross‐sectional samples at same time points (no results presented).
No power estimates.

Participants

Population of study sites: exact number not given, all treatment areas had populations of over 100,000.
Target population: residents of Texas, primary target audience ‐ smokers aged 25 to 49. Prevalence samples were 9407 at baseline and 8974 at follow‐up. Cessation cohort of 622 daily smokers, 62.9% F.
Age: 18+.
Sex: M & F.
Ethnicity: White 82.9%, Black 8.3%, Hispanic 6.0%, Asian 0.2%, other 2.6%. High‐level media/cessation areas had disproportionately more African‒Americans (20.6%) than the other four conditions (4.4% to 6.9%)

Interventions

Theoretical basis: social learning theory, the transtheoretical model, modelling, social reinforcement for behaviour change, emotional arousal.

Year conducted: 2000.

Duration: 7 m.
Community forums, focus groups and pre‐testing were used to develop messages.
Components and content:

TV, radio, newspapers, billboards, posters.

Ads were created in English, Spanish and Vietnamese. TV ads and radio spots included 2 developed by the CDC, one of which was also printed in local newspapers. All ads promoted the American Cancer Society Smokers' Quitline; radio and print ads encouraged smokers to seek doctor's or pharmacist's help in quitting. 10‐second PSAs promoting the Quitline were broadcast during morning drive times.

Outcomes

Point prevalence of daily smoking.
Definition: smoker ‐ answered 'yes' to the question 'Do you now smoke cigs everyday?'
Questionnaire: by random digit dialled telephone.
Biochemical confirmation of abstinence: none reported.

Notes

Process measures: awareness/reach was measured by 3 questions about frequency of being exposed to media messages through TV, radio and newspaper ads over past 30 days. Answer choices were never, 1 to 3 times, 1 to 3 times per week, daily or almost daily, more than once a day.
Processes of change variables adapted from TTM.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

A quasi‐experimental cross‐sectional study.

Allocation concealment (selection bias)

High risk

A quasi‐experimental cross‐sectional study.

Blinding of participants and personnel (performance bias)
All outcomes

High risk

Not done due to the nature of the intervention.

Blinding of outcome assessment (detection bias)
All outcomes

Low risk

Data collection was done by independent contractors, the interviewers were blinded to the intervention received by the respondent.

Incomplete outcome data (attrition bias)
All outcomes

High risk

Cross‐sectional surveys, no cohort follow‐up, a panel of smokers followed (58% to 65% followed up), reasons for attrition described.

Selective reporting (reporting bias)

High risk

Outcomes presented combined, not as specified in Methods section.

Other bias

Low risk

No other bias identified.

McPhee 1995

Methods

Country: USA.
Objective: to reduce smoking prevalence among Vietnamese‐American men.
Study sites: Santa Clara County, California (intervention), and Houston Texas (control).
Programme name: Smoking Cessation among Vietnamese‐American Men ‐ I.
Design: quasi‐experimental.
Analysis: Chi² tests, t‐tests, multiple logistic regression, with individual as the unit of analysis. Smoking behaviour was assessed in cross‐sectional surveys at baseline and 2 yr follow‐up.
Power estimate of 0.8 power to detect a 5%+ reduction in prevalence. Required a sample of at least 1200 in each community (achieved).

Participants

Population of study sites: Santa Clara County ‒ 54,212 Vietnamese (18,770 men 18+); Houston ‒ 33035 Vietnamese (11878 men 18+).
Age: 18+ Vietnamese‒American men.

Interventions

Theoretical basis: not specified. In 1989 ran a 15 m pilot media programme in San Francisco.

This trial started: Nov 1990.

Duration: 2 yrs.
Components:

(i) 35 print articles in Vietnamese‐language newspapers and magazines; videotape broadcast ×2 on Vietnamese‐language TV, interviews with smokers, physicians and quitters, health education materials, e.g. calendar, bumper stickers, lapel buttons, 3 posters, 2 brochures (one for male smokers ‒ effects of smoking and quitting; 1 for female smokers ‒ effects of ETS) and self‐help 'quit kit'.
(ii) An anti‐tobacco counter‐advertising campaign included billboards (3 different types), newspaper and magazine ads and paid TV ads.
(iii) Short anti‐tobacco presentations at community events (adaptation of American Cancer Society Great Smokeout Programme for Vietnamese population) and a CME course on smoking cessation counselling methods for Vietnamese physicians, Vietnamese 'no smoking' signs and smoking control ordinances to local businesses and restaurants.

Outcomes

Smoking prevalence, cigarette consumption, quit rates.
Definitions: current smoker ‒ answered yes to 2 questions: (a) ever smoked a cig (b) smoked a cig during the previous week; former smoker ‒ answered yes to 1st and no to 2nd question; never smoker ‒ answered no to both questions; recent quitter ‒ quit smoking during 2 yrs before either pretest or post‐test interview.
Questionnaire: by telephone.
Biochemical confirmation of abstinence: none.

Notes

Intermediate measures: motivation to quit, self‐efficacy, quit attempts assessed at baseline and follow up.
Process measures: media weight, awareness/reach.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Quasi‐experimental, with Santa Clara County chosen as the intervention area and Houston, Texas as the comparison area.

Allocation concealment (selection bias)

High risk

Quasi‐experimental, with Santa Clara County chosen as the intervention area and Houston, Texas as the comparison area.

Blinding of participants and personnel (performance bias)
All outcomes

High risk

Not done due to the nature of the intervention.

Blinding of outcome assessment (detection bias)
All outcomes

Unclear risk

Self‐reported smoking ‒ telephone interview, not clear if interviewers were aware of the intervention received.

Incomplete outcome data (attrition bias)
All outcomes

High risk

Cross‐sectional surveys, no cohort follow up, response rates 81% to 88%

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in Methods section.

Other bias

Low risk

No other bias identified.

McVey 2000

Methods

Country: England.
Objective: to motivate smokers to give up and ex‐smokers to stay stopped.
Study site: 4 central and northern English independent TV regions (Central, Granada, Tyne Tees, and Yorkshire).
Programme name: Health Education Authority for England's anti‐smoking TV campaign.
Power calculation: assuming a control quit rate of 7%, looking for a 50% increase by TV campaign alone, and 100% increase in TV + LTCN, and alpha set to 0.05, 4000 smokers required to detect at 0.9 power (TV alone) and 0.8 power (TV + LTCN). Assuming a 30% drop‐out rate, 5800 needed at baseline
Design: quasi‐experimental, 3 intervention regions: Granada, Tyne Tees and a region of Yorkshire (TV advertising), West Yorkshire (TV campaign and a local tobacco control network); 1 control region: Central TV. Only TV media and control regions analysed in this review.
Analysis: multiple logistic regression, Chi², ORs of smoking/not smoking with 95% CIs, calculated for smokers and ex‐smokers. The ORs adjusted for all pre‐intervention predictors of change in smoking status for smokers and ex‐smokers were pooled to estimate a common intervention effect using fixed‐effect meta‐analysis method, with individual as unit of analysis; smoking behaviour assessed in a cohort of smokers and ex‐smokers at baseline, 6 m and 18 m.
Power estimate 0.9 (TV alone) to detect a 3.5% increase in cessation, and 0.8 (TV + LTCN) to detect a 7% increase in cessation.

Participants

Population of study sites: inhabitants of Central, Granada, Tyne Tees and Yorkshire TV regions, overall numbers not given; chosen for higher prevalence of smoking.
Age: adults, aged 16+, 5468 at baseline, 3610 at 6 m and 2381 smokers or ex‐smokers at 18 m; interviews conducted in a 2‐stage cluster sampling design, including pseudo‐random Kish‐grid method. Interviewers and participants at baseline were not informed about forthcoming TV campaign, or that they were part of a trial; follow‐up interviews conducted by different team, blinded to intervention or pre‐campaign status.
Sex: M & F.
Ethnicity: not recorded.

Interventions

Theoretical basis: not specified.

Year started: 1992.

Duration: 18 m.
Formative process: a series of qualitative pilot studies using focus groups and in‐depth interviews with smokers and ex‐smokers.
Components:

(i) paid TV anti‐smoking ads aimed at current smokers and those who had already given up. In the ads morbid or 'black' humour, macabre or bizarre scenarios were used, featuring John Cleese (well‐known English comic actor). Each ad ended with a 'Quitline' number for further information and advice. Ads were screened in 2 phases over 18 m, at varying intensity during 1st phase (10 ads, each 30 to 40 secs, Dec 1992 to March 1993). Granada received single weight advertising, and Tyne Tees and Yorkshire double weight. In 2nd phase (9 ads (4 new) December 1993 to March 1994) all 3 regions received double weight advertising.
LTCN intervention (not considered in this review) in West Yorkshire only: organised network 'West Yorkshire Smoking and Health' (WYSH) to fund and co‐ordinate multiple anti‐smoking activities, e.g. clean air awards, health promotion, Guide to Smoke‐Free Eating and Drinking, cessation support. Media and skills training for local health professionals, political lobbying, media publicity.

Outcomes

Quit rate, relapse rate, abstinence rate; smoking/not smoking at 18 m.
Definitions: current smoker ‒ answered yes to question 'Do you smoke cigs at all nowadays?' and reported number of cpd; ex‐smoker ‒ did not report current smoking, but answered yes to question: 'Have you ever smoked a cigarette, pipe or cigar?' and reported the number of cpd previously smoked; quitter ‒ baseline smokers reporting no current smoking at follow up; relapser ‒ ex‐smokers reporting current smoking at follow‐up.
Questionnaire: in person at home.
Biochemical confirmation of abstinence: none.
Analysis not ITT, since participants unaware of objectives and interventions.

Notes

Intermediate measures: attitudes at baseline, no follow‐up results.
Process measures: media weight.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Quasi‐experimental, 3 intervention regions: Granada, Tyne Tees and a region of Yorkshire (TV advertising), 1 control region: Central TV, not random by practical and ethical considerations.

Allocation concealment (selection bias)

High risk

Quasi‐experimental, 3 intervention regions: Granada, Tyne Tees and a region of Yorkshire (TV advertising), 1 control region: Central TV.

Blinding of participants and personnel (performance bias)
All outcomes

Low risk

Interviewers and respondents were unaware of the forthcoming TV campaign or of the intention to conduct follow‐up interviews; the follow‐up interviews were conducted by a different group of field workers who were unaware of the pre‐campaign responses or the relationship between the interviews and the interventions.

Blinding of outcome assessment (detection bias)
All outcomes

Low risk

Interviewers and respondents were unaware of the forthcoming TV campaign or of the intention to conduct follow‐up interviews; the follow‐up interviews were conducted by a different group of field workers who were unaware of the pre‐campaign responses or the relationship between the interviews and the interventions.

Incomplete outcome data (attrition bias)
All outcomes

High risk

Response rate not reported, cohort follow‐up ‒ just number of sampled at baseline and followed at 6 and 18 months provided; 39% to 52% of those sampled at baseline followed, lost to follow‐up not counted as eligible at follow‐up, reasons provided

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in methods section.

Other bias

Low risk

No other bias identified.

Mogielnicki 1986

Methods

Country: USA.
Objective: to improve smoking cessation achieved in clinic programme by mass‐media anti‐smoking campaign.
Study site: 2 outpatient clinics at Veterans Administration Hospitals ‒ Manchester (M) NH, and White River Junction (WRJ) Vt.
Design: quasi‐randomised study (sequential allocation 1 to 1) in 1st yr; subjects allocated to behaviour‐modification programme or mailed cessation materials. In 2nd yr patients were assigned on a 2:1 ratio to clinic and mailing group respectively. After 2nd yr clinics a mass media intervention was delivered to one of the hospital regions (WRJ) in quasi‐experimental design, but not to the control region (M). Only 2nd yr of study analysed here.
Analysis: Chi², F test, logistic regression analysis, with individual as unit of analysis. Smoking behaviour was assessed in a cohort of smokers at baseline and 6 m follow‐up.
No power estimates.

Participants

Total participants: 311 clinic enrolments, 66 mailed quit kit recipients.
Participants of interest: WRJ (clinic + media after 2nd yr) 71 veterans, M (clinic, no media after 2nd year) 33 veterans.
Target population: male veterans 18 to 65 yrs with self‐reported cigarette consumption of at least 10 a day.
Ethnicity: not reported.

Interventions

Theoretical basis: for clinic treatment, behavioural model of Best (Best 1978a; Best 1978b).

Study period: Nov 1980 to May 1981; 2nd phase July 1981 to December 1981.
Media campaign ran in WRJ region as a series of 3‐week 'flights' Nov 1981 to March 1982 (2 to 6 m after 2nd yr of clinics). Duration: 5 m.
Media campaign ‒ current marketing methodology used to develop a media campaign targeting cessation clinic participants.
Components and content:

TV and radio spots ‒ testimonial vignettes selected from interviews with quitters about benefits of quitting.

1 × 60 sec main ad (aired 60 times) and 2 × 30 sec variations (aired 106 times) made for TV and modified audio version for radio, all ending with the sentence 'Life is better without a cigarette; if you are smoke‐free, stay free'. Campaign aired on 2 most popular TV stations and 2 most popular radio stations in the WRJ region.

Outcomes

Abstinence rate.
No definitions given.
Questionnaire: in person at the clinic.
Biochemical confirmation of abstinence: exhaled CO; venous blood specimen for thiocyanate level.

Notes

Intermediate measures: at baseline: attitudes and beliefs on smoking, TV viewing habits.
Process measures: media weight, awareness/reach, intervention costs.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Quasi‐experimental: in one of the hospitals mass media intervention, no mass media in another hospital.

Allocation concealment (selection bias)

High risk

Quasi‐experimental: in one of the hospitals mass media intervention, no mass media in another hospital.

Blinding of participants and personnel (performance bias)
All outcomes

Unclear risk

Not done due to the nature of the intervention.

Blinding of outcome assessment (detection bias)
All outcomes

Low risk

No blinding; biochemical validation of smoking cessation, lack of blinding has no influence.

Incomplete outcome data (attrition bias)
All outcomes

High risk

Cohort follow‐up, 52% to 54% follow‐up, loss to follow‐up not reported for mailed control group.

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in Methods section.

Other bias

Low risk

No other bias identified.

North Coast QFL 1983

Methods

Country: Australia.
Objective: to lower prevalence of smoking.
Study sites: Lismore, Coffs Harbour and Tamworth, New South Wales.
Programme name: North Coast Quit for Life Programme as a part of North Coast Healthy Lifestyle Programme.
Design: quasi‐experimental, 2 intervention towns: Lismore (mass media and community programmes); Coffs Harbour (mass media programme alone); Tamworth (control town). Only Coffs Harbour and Tamworth included in our analysis.
Analysis: multiple logistic regression, Chi², with individual as the unit of analysis. Smoking behaviour was assessed in cross‐sectional surveys of random samples at baseline 3 yrs.
No power estimates.

Participants

Population of study sites: Lismore 22,083, Coffs Harbour 12,197, Tamworth 27,280.
Age: 18+
Sex: M & F.
Ethnicity: White.

Interventions

Theoretical basis: communication theory and social marketing.

Year started: 1978.

Duration: 2 yrs.
Formative research: focus groups, spot surveys.
Components:

(i) newspaper (Lismore: 1 paid local daily; Coffs Harbour: 1 paid local tri‐weekly newspaper and weekly free paper);

(ii) radio (Lismore: a local station; Coffs Harbour: a relay station)

(iii) TV (Lismore and Coffs Harbour a shared station)

(iv) stickers, posters, T‐shirts, balloons, and self‐help quit kits. First part of mass media intervention was part of 9‐week healthy lifestyle campaign and focused on general awareness. Second part was providing information, 3rd part was aimed to create 'a positive effect'. Parts 2 and 3 lasted 31 weeks. Ads in media were paid, with equal time also donated free by stations. Other media included editorial space, features, radio interviews, TV appearances, weekly programmes, retail ads and pictorial spreads. All ads were professionally created, entertaining and controversial; (see Notes).
The community intervention (in Lismore, not included in this analysis) included quit kits handed out by doctors, quit fact sheets, quitter tips packs, a quitline telephone message, a variety of smoking cessation groups, and public events such as fun runs.
We have not included the Lismore community intervention in this review, but it is covered fully in review by Secker‐Walker 2002

Outcomes

Smoking prevalence.
Definition: smoker ‐ no definition given.
Questionnaire: in‐person interview at a central screening centre or at home.
Biochemical confirmation of abstinence: plasma thiocyanate in 2nd and 3rd yr on a randomly selected 5% sub‐sample.

Notes

All printed ads were withdrawn 4 m after the start of campaign (Oct 1979) for 15 weeks because of complaints to the Media Council of Australia by 3 major tobacco companies. National publicity about the suspended ads reached the control town, and may have contaminated the comparison.
Intermediate measures: attitudes to smoking (6 questions), knowledge of the effects of smoking (6 questions), smoking behaviour (4 questions), influence in decision to quit and techniques of quitting by those quitting smoking were assessed. Knowledge and attitudes follow‐up data provided only in the graphical form without description and numbers.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Quasi‐experimental, no randomisation attempted, intervention and control sites chosen due to practical reasons.

Allocation concealment (selection bias)

High risk

Quasi‐experimental, no randomisation attempted.

Blinding of participants and personnel (performance bias)
All outcomes

High risk

No blinding due to the nature of the intervention.

Blinding of outcome assessment (detection bias)
All outcomes

High risk

Self‐reported smoking behaviour, interview carried out by trained interviewer at a central screening centre; only 5% subsample biochemical validation.

Incomplete outcome data (attrition bias)
All outcomes

High risk

Cross‐sectional surveys, no cohort follow‐up, response rates 71% to 74%.

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in Methods section.

Other bias

Low risk

No other bias identified.

Stanford 3 City 1977

Methods

Country: USA.
Objective: to increase knowledge of the risk factors for cardiovascular disease and change behaviour by decreasing smoking, improving diet, weight, physical activity and blood pressure.
Study sites: Watsonville, Gilroy and Tracy, California.
Programme name: Stanford 3‐City Project.
Design: quasi‐experimental. 2 intervention towns: Watsonville (mass media for the whole population + counselling for high‐risk individuals); Gilroy (mass media programme alone); Tracy as comparison town. High‐risk subjects were identified in each town and in Watsonville were randomly assigned to face‐to‐face counselling or no counselling. In Watsonille another sample of community members excluding the group receiving face‐to‐face counselling was created (= Watsonville‐reconstituted). Gilroy, Watsonville‐reconstituted and Tracy were included in the analysis. Within the high‐risk subjects analysis included Watsonville randomised control group, Gilroy and Tracy high‐risk participants.
Analysis: multiple logistic function of risk factors for 12‐yr event probability, cohort analyses, t‐tests, one‐sided P values, with individual as the unit of analysis. Smoking behaviour was assessed in cohort surveys at baseline and at yrs 1 and 2. In high‐risk group additional survey at yr 3.
No power estimates.

Participants

Population of study sites: Watsonville 14,569, Gilroy 12,665, Tracy 14,724.
Age: 35 to 59; target population sizes Watsonville 4115, Gilroy 3224, Tracy 4283.
High‐risk group defined as those in top quartile of risk at baseline.
Sex: M & F.
Ethnicity: White.

Interventions

Theoretical basis: social marketing, social learning theory and communication theory.

Year started: 1972.

Duration 3 yrs (3rd yr results for high‐risk group only).
Components:

(i) mass media campaigns in English and Spanish, with 3 hrs of TV programmes and 50 TV spots, several hrs of radio programmes and about 100 radio spots, weekly newspaper columns, newspaper and advertisement stories, billboards, printed materials sent via direct mail to participants, posters in buses, stores and worksites. Campaigns were conducted in both intervention towns for 9 m in 1973 and 9 m in 1974.
(ii) community intervention: high risk subjects identified in each city, with face‐to‐face counselling for a random subset in Watsonville.
We have not included the high‐risk subjects group from Watsonville community in this review, but it is covered fully in 'Community interventions for reducing smoking in adults' by Secker‐Walker 2002

Outcomes

Cigarette consumption, smoking prevalence within high‐risk group. Both reduction and cessation were study outcomes.
No definitions given, but daily consumption of cigarettes, pipe and cigar smoking recorded.
Questionnaire: in‐person interview at survey centres in each community.
Biochemical confirmation of abstinence: plasma thiocyanate.

Notes

Intermediate measures: 25‐item behavioural interview concerning participants' knowledge about risk factors (3 questions on smoking).
Process measures: media weight.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Quasi‐experimental, no randomisation, study sites chosen due to practical reasons.

Allocation concealment (selection bias)

High risk

Quasi‐experimental, no randomisation.

Blinding of participants and personnel (performance bias)
All outcomes

High risk

No blinding due to the nature of the intervention.

Blinding of outcome assessment (detection bias)
All outcomes

Unclear risk

In‐person interview at survey centres in each community ‒ surveys of behavioural knowledge and medical examination, not clear if interviewers aware of the intervention in the group; thiocyanate measurement mentioned, but not clear in how many people measured.

Incomplete outcome data (attrition bias)
All outcomes

Unclear risk

cross‐sectional surveys with response rate of 60% (Australia ‐wide) and cohort follow up with response rate of 73% to 76% at baseline and 63% of original sample at follow up

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in Methods section.

Other bias

Low risk

No other bias identified.

Sydney QFL 1986

Methods

Country: Australia.
Objective: To reduce the prevalence of smoking.
Study sites: Sydney, Melbourne, rest of Australia.
Programme name: Sydney 'Quit. For Life'.
Design: quasi‐experimental study, Year 1: Sydney intervention city, Melbourne control city, with rest of Australia as 2nd comparator; additional long‐term effectiveness assessed on ITS data 1981 to 1987 for Sydney and Melbourne. Melbourne received intervention from 2nd yr onwards to 1986.
Analysis: normal approximation; P values reported; test of difference in proportions; simplified form of linear regression model, full and parsimonious statistical models fitted to the age‐standardized data; individual as the unit of analysis; smoking behaviour assessed in cross‐sectional surveys in Sydney, Melbourne and the rest of Australia, and in Sydney and Melbourne in longitudinal cohort surveys.
No power estimates.

Participants

Population of study sites: Sydney 3.25 million; Melbourne, rest of Australia, not stated.
Target population: adult inhabitants of Sydney and Melbourne.
Age: 14+ (16+ in long‐term follow‐up).
Sex: M & F.
Ethnicity: not given.

Interventions

Theoretical basis: none reported.

Year started: 1983 (Sydney), 1984 (Melbourne).

Duration: 4 yrs.
Formative research on message effectiveness among target audience.
Components:

(i) media‐based campaign, with prime‐time ads on TV and radio, ads in newspapers and posters in public places. All ads ended with a 'Quitline' 24‐hr phone number (message encouraging to quit, information about 'Quit centre in Sydney Hospital' (choice of 6 standard anti‐smoking treatments for AUD 5) and a 'Quit Kit' self‐help booklet and audiocassette tape); billboards with simple message from TV spots, ads in newspapers included normal large ads and in Sydney a section covering smoking‐related events and issues; radio ads with anti‐smoking skits by major personalities. The campaign generated substantial news coverage in all mass media, and used strong visual images of the health consequences of smoking. Coverage alternated in 2‐week phases between heavy and nothing for 1st 3 m, + follow‐up ad campaign of half the intensity after 5 m.
(ii) After first year, community components added, e.g. physicians' offices and schools. TV spots during prime or fringe time for approx 4 weeks at the start of each campaign year.
Year started: Jun to Nov 1983 (Sydney) ‐ first assessment.
According to long‐term follow‐up study since 1984 the campaigns continued in both Sydney and Melbourne till 1986 with commercials shown each year on prime time TV for 6 to 8 weeks during winter months.
Duration: 6 months ‒ first evaluation; 6 to 8 weeks a year for 4 yrs ‒ long‐term evaluation.

Outcomes

Smoking prevalence, tobacco consumption.
Definitions: smoker ‒ anyone who responded positively to the question 'do you smoke factory‐made cigarettes, roll‐your‐own cigarettes, cigars or a pipe'; quitter ‒ a person smoking at baseline but not smoking at the time of 2nd (1984) survey; reducer ‒ a smoker who at 2nd survey smoked at least 5 cpd fewer than at baseline survey.
Questionnaire: in person, at home.
Biochemical confirmation of abstinence: saliva cotinine in 2 subsamples.
Measured at 1 yr (Sydney vs Melbourne), 2 years (Sydney vs rest of Australia).
Further follow‐up: every 6 m for 7 yrs (Sydney and Melbourne trends).

Notes

Intermediate measures: health beliefs and social influences assessed at baseline and at long‐term follow‐up; failed quit attempts assessed in a cohort of smokers in Sydney and Melbourne; information‐seeking behaviour of the population; number of calls to quitline, enrolments in 'Quit centre' stop‐smoking programmes, number of quit kits sold.
Process measures: media weight, awareness/reach, intervention costs.

Risk of bias

Bias

Authors' judgement

Support for judgement

Random sequence generation (selection bias)

High risk

Quasi‐experimental, study sites chosen due to the practical reasons.

Allocation concealment (selection bias)

High risk

Quasi‐experimental design did not allow for allocation concealment.

Blinding of participants and personnel (performance bias)
All outcomes

High risk

No blinding.

Blinding of outcome assessment (detection bias)
All outcomes

Unclear risk

Saliva cotinine in subsamples, self‐reported smoking behaviour, in‐person interview, not clear if interviewers were aware of the intervention.

Incomplete outcome data (attrition bias)
All outcomes

High risk

cohort study ‐ 61% to 63% followed up, cross‐sectional surveys response rates 60%.

Selective reporting (reporting bias)

Low risk

Outcomes presented as prespecified in Methods section.

Other bias

Low risk

no other bias identified

ad: advertisement
BRFSS: Behavior Risk Factor Surveillance System
CDC: Centers for Disease Control
CI: confidence interval
CME: continuous medical education
CO: carbon monoxide
cpd: cigarettes per day
CPS: Current Population Surveys
ETS: environmental tobacco smoke
F: female
ITS: interrupted time series
LTCN: local tobacco control network
m: month(s)
M: male
MTS: Massachusetts Tobacco Surveys
NHIS: National Health Interview Survey
OR: odds ratio
PSA: public service announcement
TTM: Trans‐Theoretical Model (stages of change)
TV: television
yr: year

Characteristics of excluded studies [ordered by study ID]

Study

Reason for exclusion

A Su Salud 1990

Community intervention, multicomponent, smoking and other risk factors; health education, mass media and an intensive programme of individual face‐to‐face and telephone counselling. Each mass media group received face‐to‐face contact.

Arizona 1998

Arizona statewide tobacco control program; description of the programme design; no baseline measurement, no control, no smoking‐related outcomes.

ASSIST 2003

Demonstration project in 17 states of policy interventions, media interventions and smoking cessation activities. No description of mass media component, effectiveness measured in print coverage, not possible to separate out the effects of the mass media component.

Barber 1990

Australia's media campaign against drug abuse; no non‐exposed control group, 1 measurement before and 1 immediately after the campaign.

Boyd 1998

RCT, randomising 14 media markets; targeting African Americans communication campaign utilizing radio and TV advertisements in combination with community outreach encouraging to call Cancer Information Service for smoking cessation information and materials; Outcome was volume of calls rather than changes to smoking behaviour.

Brownson 1996

The Bootheel Heart Health Project; quasi‐experimental; smoking as a part of the community programmes and coalition development; mass media (newspaper column) within community programmes, no separate results for mass media component alone.

Chicago I 1989

Mass media cessation series accompanying self‐help smoking manual and in some counselling for supported groups of adults at health maintenance organisations or worksites. Surveys immediately after the campaign, 3 months, 1 year, no concurrent nonexposed control group.

Chicago II 1992

Mass media cessation series accompanying self‐help smoking manual and group counselling in some participants; no control group without mass media exposure.

Chow 2009

Multicomponent health promotion campaign including posters, street theatre, mimicry, rally and community presentation; not possible to assess mass media (posters) component alone.

Coeur en santé 1999

Coeur en sante St‐Henri, Montreal, Canada, mass media component was included in a multicomponent communitywide intervention targeting women. No results for mass media component alone.

COMMIT 1995

Multicomponent community intervention, involved face‐to face contact, mass media included news and stories in newspapers, on radio and television, posters and billboards, mailings; not possible to assess effects of mass media separately.
Includes a cohort follow‐up (Hyland 2006) reporting association between level of exposure to campaign and RR of quitting.

Cummings 1987

Newspaper series and 1 survey after, no control group.

Cummings 1993

Media markets randomised; mass media used to encourage women with young children to call for information on quitting; mass media as a recruiting tool; the smoking intervention was the counselling they received when they called the NCI phone line.

Danaher 1984

Televised smoking cessation programme shown as a part of local news in Los Angeles area, no baseline measurement, registrants to the programme compared with cross‐sectional sample, all exposed to the programme

Davidson 1990

Unable to assess as reprint unobtainable.

Donovan 1984

'Give it away for a day' ‐ Australia smoke‐free day; mass media, events, competitions and community interventions used for awareness, call to action, encouraging commitment to quit, no control group.

Doxiadis 1985

Nationwide anti‐smoking campaign in Greece, mostly TV and radio, tobacco advertising ban; no control group; outcomes measured ‐ aggregated data on tobacco consumption, annual increase.

Dubren 1977

Televised stop smoking clinic; no control group, no baseline measurement.

Dyer 1983

Survey on impact of 'Smokers' Luck' TV programme on smokers' attitudes and behaviour, no control group.

Eiser 1978

National survey on impact of TV programmes 'Dying for a Fag' and 'Licence to Kill' on cigarette smokers' attitudes and behaviour ‐ one before and 2 after the programme, no control group.

Etter 2005

Poster campaign; controlled before‐after study, follow‐up too short

Etter 2007

Booklet sent; RCT, follow‐up too short

EX campaign 2010

Mass media campaign; no non‐exposed control group

Frith 1997

Nationwide No Smoking Day ‐ panel survey, no control group, 1 before and 2 after measurements.

GASO 2002

Great American Smokeout; mass media for recruitment; before and after surveys, 2 months follow up only, no control group.

Gredler 1981

National information campaign on smoking in Austria with 'stop now' programme, no control group, 1 survey after the campaign.

Heartbeat Wales 1998

Multicomponent intervention including mass media, self‐help materials, stop smoking groups and smoking cessation counselling. Smoking cessation not a reported outcome, only daily smoking and cigarettes per day.

HEBS 1997

Health Education Board for Scotland's anti‐smoking campaign involving mass media, phone line and booklet. No control group, 1 measurement of callers to quit line before and 3 after.

Hill 2003

National tobacco campaign, no control group, one baseline measurement.

Hunkeler 1990

Multicomponent community‐wide intervention targeted at minorities, including use of the mass media; description of the design of campaign and implementation, no smoking‐related results given.

IHHP 2003

Multicomponent community intervention, mass media public education (TV, newspapers, radio) supported several community programmes; not possible to assess effects of mass media separately.

Jason 1988

Televised smoking cessation programme combined with self‐help manual, supportive phone calls and group meetings led by community member and psychology graduate student; control group potentially exposed to TV programme.

Laugesen 2000

New Zealand's tobacco control programme involving mass media use, census data 1 before the programme and 2 after the beginning, no control group, country case study.

Le Net 1977

National against tobacco campaign involving mass media, no control group, 1 survey before and 1 after the campaign.

Ledwith 1984

RCT, mass media used as recruitment tool, the intervention was posted leaflet targeted at those recruited.

Leroux 1983

Quit line, smoking kit ‐ a letter explaining the programme, booklet, media clinic broadcast as a part of TV show and radio programme ‐ the format of talk show; comparison group exposed to the media programmes; follow up 3 months.

Lichtenstein 2008

RCT cross‐over trial of 1346 households, randomised to self‐help video information/no video and telephone counselling/no counselling. Interventions not counted as mass media, since only participating homes received videos, and outcomes were cessation rates and imposition of domestic smoking bans.

McAlister 2006

Comprehensive community and media programme in Beaumont/Port Arthur, measuring prevalence compared with other parts of Texas. Cannot separate effects of media from co‐interventions.

MHHP 1995

Minnesota Heart Health Programme ‐ comprehensive community intervention to reduce smoking, high cholesterol, high blood pressure and sedentary lifestyles; health education, policy intervention and mass media use. No separate results for the mass media component alone.

Millar 1987

Community‐based smoking cessation programme led by self‐help booklet as primary quitting aid and complemented by 3‐part TV series. Major design problems and confounders ‐ cigarette prices rose sharply in the control city in the 6 months post‐intervention, and thus confounded or nullified the effect of the comparison ‐ the quit rates were higher in control city than in experimental city.

Minnesota 2011

Statewide mass media campaign; only 1 measurement point before campaign

Mudde 1995

Multi‐component community‐wide smoking cessation intervention involving local mass media (newspaper, radio and TV), posters and leaflets, a local quit line, and self‐help materials, smoking cessation groups, and individual telephone counselling. Mass media primarily used to recruit to self‐help or group support rather than to disseminate tobacco control messages. Control community may have been 'contaminated' by national campaigns and national smoking ban.

Mudde 1999

Mass media‐led smoking cessation campaign, no control group, 1 measurement before and 2 after the campaign.

Multicity 1997

Both groups exposed to mass media campaign, designed to promote readiness to quit smoking; active vs passive intervention ‐ community+mass media vs mass media alone (to raise awareness).

New York

Statewide mass media campaign; only 1 measurement point pre‐campaign

North Karelia 1998

Comprehensive community‐based programme to reduce major cardiovascular risk factors; education and health services, community involvement, mass media, screening, appropriate practical skills training, social support for behaviour change and environmental modification. No separate results for the mass media component alone.

Oregon 1999

Oregon's Tobacco Prevention and Education Program, description of programme design, development and implementation; result concerning aggregate data on tobacco sales in Oregon compared to other states (tobacco boxes taxed before and after tax increase).

Osler 1993

Mix of community and mass media interventions (including smoking cessation programmes). Planned as community‐based cardiovascular disease prevention project, but the authors state that 'it almost ended up being pure mass media awareness'. Doubtful design and quality, under‐resourced and poorly executed.

Pallonen 1994

RCT, 6 months follow up, not smoking cessation mass media intervention; mass media used for recruitment and identification.

Perkins 1986

Intervention ‐ posters‐public places intervention (another review); study does not provide information about cessation or smoking or the change in smoking due to intervention; not possible to assess the effects of the intervention, as counting of butts and visible smokers may be a subjective and naive assessment of efficacy; although the study lasted for 26 weeks, smoking status of the patients was not recorded up to week 12 of the study.

Programa Latino 1994

Programa Latino para Dejar de Fumar; multi‐component, including mass media, community‐wide smoking cessation intervention for Spanish‐speaking Hispanics; 2 pre and 2 post intervention measurements, no control group.

Sansores 2002

No control group, monthly sales of all smoking cessation products before and after marketing a new nicotine patch.

Secker‐Walker 2000

Multicomponent community campaign involving mass media use. No separate results for mass media component alone.

Sogaard 1992

Mass media‐based health education campaign 'Heart for Life', no control group, post‐campaign survey only.

Stanford 5 City 2000

Multicomponent community‐wide cardiovascular disease risk factor reduction programme, including smoking prevention and cessation campaign. The intervention was implemented through the use of the media ‐ TV, radio, newspapers and direct face‐to‐face education in classes, contests and correspondence courses and school based programmes. Not possible to separate effect of mass media.

Stevens 2002

Economic evaluation of mass media‐based community smoking cessation intervention aimed at the Turkish community; no control group, before‐and‐after panel survey in Turkish population.

Sussman 1994

Self‐help media‐enhanced smoking cessation programme which had been aired in 7 cities in California. Subjects had been randomly assigned to be prompted or not prompted to view the mass media smoking cessation broadcast. 3 months follow up; control group potentially exposed to the programme, no smoking related outcomes.

Sutton 1987

Mass media smoking cessation intervention, the evaluation carried out in the workplace during BBC broadcasting of 'So You Want to Stop Smoking' programme; smokers at the workplaces were shown the 2 series of the programme and encouraged to watch the remaining four parts on TV. Control groups were shown film about political and economic aspects of smoking; no non‐exposed control group.

Terry‐McElrath 2013

Not specific campaign; no non‐exposed control group

TIPS

Randomised field trial comparing standard dose of mass media campaign (national campaign TIPS) and higher dose mass media campaign (additional). Follow up was too short ‐ tobacco related outcomes were assessed on average 55 days after the end of campaign.

TV Finland 1992

National TV smoking programmes in Finland based on North Karelia experience; National TV smoking cessation campaign in 1978, 'Keys to Health' in 1980,82, 84‐85, 'Quit Smoking 86'; North Karelia (community interventions) vs the rest of Finland or one city/county of Finland with no community activities; no non‐exposed control group.

Valois 1996

Community cable TV smoking cessation programme; time series design to assess effectiveness, 1 measurement before and 3 after, no control group.

Van Assema 1994

Multicomponent community project; community agencies and associations, local government, public events, newsprint, posters, pamphlets, mailings, stop smoking self‐help manuals and smoking cessation groups. No separate results for mass media component alone.

Webb 2009

Booklet vs booklet RCT; no non‐exposed control group

Wewers 1991

Mass media smoking cessation campaign; no control group, first survey after the campaign.

Wheeler 1988

Community‐wide smoking cessation campaign using self‐help manual and TV coverage. No control group, first survey after campaign.

Data and analyses

Open in table viewer
Comparison 1. Mass media versus no mass media

Outcome or subgroup title

No. of studies

No. of participants

Statistical method

Effect size

1 Response and retention rates Show forest plot

Other data

No numeric data

Analysis 1.1

Study

Design

Intervention

Control

California TCP 2003

Nested cross‐sectional

1990‐1 75.1%1 (CA only)
CTS: 71.3 ‐ 99.4% (CA only)
NHIS 79.5 ‐ 87.8% across all USA
BRFSS: 77% ‐ 84%
CPS: 87.9%

NHIS, BRFSS and CPS rates apply.

CORIS 1997

Nested cross‐sectional

Nested cohort

69.5% at baseline, 67.5% at resurvey
Cohort identified retrospectively at 2nd survey, i.e. 4087 (56.3%) of those originally surveyed.
In men 46.4% of cohort smoked vs 50% of drop‐outs; in women 15.4% of cohort smoked vs 21.4% of drop‐outs.
Non‐cohort (i.e 1 survey only) were younger, less educated and higher smoking, but differences applied equally across all groups.

64% at baseline, 63% at resurvey


Cohort control not reported separately

Jenkins 1997

Nested cross‐sectional

Pretest: 84%, post‐test: 94%

Pretest: 82%, post‐test: 88%

Massachusetts 2003

Nested cross‐sectional

Median (1995): 60.4%, median (1999): 42.7%6

Median (1995): 68.4%, median (1999): 55.2%

McAlister 2004

Nested cross‐sectional


Nested cohort

Baseline survey 9407, 7m follow‐up 8974; response rate approximately 60%.
Cohort identified retrospectively at 2nd survey, i.e. 622 (58%) of the 1069 baseline daily smokers. Conditions not reported separately. 835 valid phone numbers at follow‐up, giving retention rate of 74.5%.

Not reported separately

Not reported separately

McPhee 1995

Nested cross‐sectional

Pretest: 81%, post‐test: 82%

Pretest: 85%, post‐test: 88%

McVey 2000

Nested cohort

Smokers: 6m: 73%, 18m: 70%
Ex‐smokers: 6m: 76%, 18m: 75%

Smokers: 6m: 74%, 18m: 66%
Ex‐smokers: 6m: 80%, 18m: 76%

Mogielnicki 1986

Randomized cohort

Mail: Yr2 follow‐up: 17%
Clinic: Yr2 follow‐up: 54%

Mail: Yr2 follow‐up: 15%
Clinic: Yr2 follow‐up: 52%

North Coast QFL 1983

Nested cross‐sectional


Nested cohort

Baseline: 71%
Yr 2: 73%
Yr 3: 73%

Not reported here

Baseline: 72%
Yr 2: 74%
Yr 3: 74%

Not reported here

Stanford 3 City 1977

Nested cross‐sectional
(cohort + non‐cohort)

Nested cohort

Gilroy 116 at baseline.
Non‐cohort had higher baseline % rates of smoking (74.2/62.4) and more cpd (20/13.8) than cohort.

73.2% at 2yr follow‐up

Tracy 115 at baseline.
Non‐cohort had higher baseline % rates of smoking (78/52.8) and more cpd (17.4/14) than cohort.

72.2% at 2yr follow up

Sydney QFL 1986

Nested cross‐sectional

Nested cohort

Sydney: 66%
Melbourne: 67.9%
Sydney: 76%
Melbourne: 73%

Australia‐wide: 60%



Comparison 1 Mass media versus no mass media, Outcome 1 Response and retention rates.

2 Intermediate measures Show forest plot

Other data

No numeric data

Analysis 1.2

Study

Intermediate measure

Process measures

California TCP 2003

Between 1996 and 1999 slight increases in agreements that smoking causes cancer (82.2% and 83.3%) and that passive smoking harms children's health (93.2% and 94%).

Between 1992 and 2002 increases in agreement that passive smoking causes cancer in nonsmokers (1992: 62.4%, 1996: 66.8%, 1999: 68.9%, 2002: 72.1%) and that smoking harms children's health (1992: 85.5%, 1996: 87.7%, 1999: 90.1%, 2002: 90.9%).

In 2008, 67.2% of daily smokers, 80.2% of non‐daily smokers, and 85.8% of nonsmokers perceived secondhand smoke (SHS) as a cause of cancer and 88.6% of daily smokers, 94.1% of non‐daily smokers and 94.7% of non‐smokers believed that SHS can harm the health of children and babies.

Between 1990 and 1991 increase in % of smokers thinking about quitting (from 38.6 to 42%; NS); health‐enhancing attitude score decreased (from 68.4% to 66.3%; SS).

Further increase in % of smokers thinking about quitting between 1994 and 2005, in the next 30 days (1994: 30.9, 2000:37.8%, 2005: 43.9%) and in the next 6 months (1994: 65.6%, 2000: 74%, 2005: 75%).

Between 1992 and 2002 increases in: % of indoor workers reporting a smokefree work site (1990: 35%, 1992: 46.3%, 1993: 65%, 1996: 90.5%, 1999: 93.4%, 2002: 95.4%, 2005: 94.5%, 2008: 96.4%).

In 2008, 95.2% of smokers and 96.6% of nonsmokers report having a completely smoke‐free workplace.

Between 1992 and 2008 increase in % of homes with smoking ban (1992: 48.1, 1993: 50.9%, 1996: 64.5%, 1999: 72.8%, 2002: 76.9%, 2005: 78.4%, 2008: 80.8%).

Between 1992 and 2008 increase in % of smokers with a total home ban on smoking (1992: 19.4 ± 1.6%, 2008: 59.3 ± 2.6%; increase of 204.9%).

Between 1990 and 2008 decrease in % of nonsmokers exposed to ETS at work (1990: 29%, 1993: 22.4%, 1996: 11.8%, 1999: 15.6%, 2002: 12.0%, 2005: 13.9%, 2008: 13.5%).

Increases in: support for further increase in tax on tobacco (1992: 74%, 1993: 78%; 2008: 78%), support for ban on tobacco advertising (1990: 52%, 1996: 65% [read from the graph]), support for ban on tobacco sponsorships (1998: 56%, 2000: 60%), % of adults preferring non‐smoking bars (1996: 75%, 2000: 81%), support for banning smoking outside the entrance of buildings (2002: 62.8%, 2008: 72.1%) and in restaurant outdoor patios (2002: 62.4%, 2008: 75%), outdoor public places (2002: 52.3%, 2008: 60.4%), Indian casinos (2002: 59.9%, 2008: 66.5%), inside cars when children are in them (2005: 92.3%, 2008: 95.2%).
Support for smoking restrictions in public places in at last 4 out of 6 venues significantly higher in CA than the rest of USA (1992/3: 58.5% vs 46.5%; 1995/6: 70.2% vs 51.5%; 1998/9: 75.8% vs 57.3%).
1998 survey showed that multicomponent exposure was significantly associated with reductions in smoking prevalence, increases in home smoking bans and reductions in perceived violations of workplace no‐smoking rules (P < 0.05).

Systematic monitoring of the campaign and independent evaluations of the programme.

Media weight: 1990/1: 50+ TV spots, 50 radio spots (69 radio stations), 20 outdoor ads (775 outdoor venues), 40 newspaper ads (130 newspapers); 1992/3: 40 spots (20 TV, 12 radio, 8 in outdoor locations), 44% focused on reducing ETS, 34% on countering pro‐tobacco influences, 20% smoking cessation and 2% on reducing youth access. 1990/4: local health departments conducted 10,000+ multi‐session programmes focusing on prevention (61%), cessation (37%) and ETS (2%). July 1995 ‐ Dec 1996: 19 ads (11 TV, 4 radio, 4 outdoor); 1997 ‐ 1998: 40 general audience ads for distribution (20 TV, 12 radio, 8 outdoor); July 1998 ‐ Aug 1999: 50 general audience anti‐tobacco ads (22 TV, 13 radio, 15 print ‐ mostly billboards).

Awareness/reach: 1990/1: unaided awareness in adult smokers 38.4% (similar for most ethnic groups), unaided and aided awareness almost 78.7%. In 1992 > 50% of the adult respondents and > 80% of the adolescent respondents recalled having seen or heard anti‐tobacco messages. In 1992 recall of anti‐tobacco campaign highest among the youngest age group (18 ‐ 24: M 75%, W 70%, 25 ‐ 44: M 65%, W 60%, 45+: M 58%, W 50% [read from the graph]).

In 1996, 67% of adults reported seeing antismoking message on TV, 44% hearing on radio, 41% seeing antismoking billboard. In 1998 80% of adults were exposed to tobacco control programme through two or more components.
In 1999 vs 1996 more respondents reported being exposed to lots of tobacco messages on TV, radio, billboards. 91% of adults reported seeing at least one anti‐tobacco ad in 1996, 1998 and 2000. Between 1996 and 2002 the % of smokers reporting seeing a lot of anti‐smoking ads on TV in last month increased, but then decreased between 2002 and 2008 (18 ‐ 24 yrs: 1996: 16.1%, 1999: 29.9%, 2002: 37.9%, 2005: 21.2%, 2008: 20.8%; 25 ‐ 40 yrs: 1996: 13%, 1999: 20.1%, 2002: 23.2%, 2005: 15.7%, 2008: 10.8%; 41+ yrs: 1996: 10.3%, 1999: 14.9%, 2002: 13.6%, 2005: 8.9%, 2008: 5.6%).

In 2011 in CSC 28% to 61% recalled anti‐tobacco commercials.

Dose‐response: 1990 ‐ 1996 smokers reporting recall of media spots more likely to make a quit attempt in the last year than those who did not. The more channels recalled between 1990 and 1996 the higher increase in cessation was observed. 1996 ‐ 2000 in counties with the highest multicomponent exposure rates, there were greatest reductions in adult smoking prevalence, workplace no‐smoking policy violations and the largest increases in home smoking bans.

2011 recall of any of anti‐tobacco spots was not related to quitting 1 yr later, recall of one specific spot ("Stages") was associated with higher quit attempts rate and quitting for more than 1 month, after 1 yr.

Maintenance: The campaign was established as Proposition 99, as a constitutional amendment and mandated the conduct of a mass media campaign. The campaign has been running since 1990.

Intervention costs: CA TCP mass media campaign funding/ total expenditures targeted at tobacco use in millions of US dollars: 1989/90: 14.3/85.8; 1990/1: 14.3/132.0; 1991/2: 16.0/55.9; 1992/3: 15.4/84.0; 1993/4: 12.9/61.1; 1994/5: 12.2/56.3; 1995/6: 6.6/41.8; 1999‐2000: 19.6/60.8; 2000/1: 45.3/88.2; 2001/2: 45.3/108.1; 2002/3 21.1/61.7; 2003/4: 16.8/61.8; 2004/5: 15.7/56.8; 2005/6: 15.7/58.5; 2006/7: 20/65; 2007/8: 15.7/56.6; 2008/9: 15.7/56.5. Total expenditures for mass media campaign and for the programme between 1989 and 1996 were in millions of US dollars: 91.7 and 516.9. Average annual expenditure was USD 3.35 per capita per year, but from mid‐1993 to mid‐1996 when funding was decreased annual expenditure was USD 2.08 per capita per year.
The CTCP media funding was USD 1.33 per capita in 2001/2 and it decreased from 2002/3 (USD 0.6 per capita) to 2007/8 and 2008/9 (USD 0.43 per capita).

The analysis on the basis of per capita consumption of cigarettes and average per capita media expenditures gave estimates of a fall of 3.9 packs per capita per year for each per capita USD spent on the media campaign.

CORIS 1997

At baseline, knowledge scores higher in Swellenden than in Riversdale (both cross‐sectional and cohort surveys; no statistical comparisons given); at 4 yrs more increase in women in Swellenden (both cross‐sectional and cohort surveys; in cohort survey net change statistically significant); at 12 yrs increase in both communities and no difference.
Attitudes were assessed at baseline, but the results were not reported.

Participation and reach of activities recorded in mass media and community intervention town (not included in this analysis).

Media weight: Limited data. 1st yr: 6 different billboards, 6 posters, 8 mailings, frequent news items, health messages on electricity accounts, 1 special supplement in local newspaper. 2nd and 3rd yr frequency of billboards, posters and mailings about half of the initial rate, but news items frequent and annual special supplement in a local newspaper was added.

Awareness/reach: no evidence found.

Dose‐response: no evidence found.

Maintenance: after 4 yrs of active intervention a maintenance programme was run by community.

Intervention costs: per capita costs given (USD 5 over 4 yrs in intervention community).

Jenkins 1997

At baseline no differences in motivation to quit and self efficacy (SF/Houston: 29%/23%; 29%/25%). At follow‐up significant increase in motivation in both communities, but no difference (SF/Houston: 45%/ 44%), no significant change in self efficacy (SF/Houston: 33%/26%).

A 15‐month uncontrolled pilot anti‐tobacco campaign.

Media weight: newspaper articles ‐ 465,000 print media exposures; 15,000+ copies of brochure, 4600 copies of self‐help quit kit distributed; billboard and newspaper ads ‐ 2.8 million exposures, paid TV ads ‐ 100 mins of air time. Short anti‐tobacco presentations at 25 community events, 68 Vietnamese physicians took part in smoking cessation course and 400 Vietnamese students participated in anti‐tobacco activities.

Awareness/reach: Participants were asked if they had ever read, seen or attended any of five elements of media intervention. Recall of each was significantly greater in intervention than in control community (P < 0.05), except for newspaper articles. In both communities smokers were more likely than non‐smokers to recall the elements of the campaign.

Dose‐response: no evidence found.

Maintenance: no evidence found.

Intervention costs: no evidence found.

Massachusetts 2003

1993 ‐ 2000: the majority of non‐smokers believed that second‐hand smoke (SHS) can harm children (96%) and can cause lung cancer (89%); increase in % of smokers believing that environmental tobacco smoke (ETS) can harm their children (1993: 78%, 1999: 91%, 2000: 93%) and can cause cancer (1993: 58%, 1999: 72%, 2000: 74%);
1993 ‐ 1995: decrease in: % believing that ETS is harmful (1993: 90%, 1995: 84%), support for ban on support of sports and cultural events by tobacco companies (1993: 59%, 1995: 53%, P < 0.05); increase in: support for further tobacco tax increase with funds devoted to tobacco control (1993: 78%, 1995: 81%), support for ban on vending machines (1993: 54%, 1995: 64%, P < 0.05).
1993 ‐ 2000: Increase of % of workers reporting a smokefree work site (1993: 53%, 1995: 65%, P < 0.05; 2000: 75%, 2001: 82.2%), % of homes with smoking ban (1993: 41%, 1995: 51%, P < 0.05; 2001: 71.2%).

2007 ‐ 94.5% believe ETS is harmful.

Decrease in: ETS exposure at work (1993: 44%, 2000: 29%), at home (1993: 28%, 2000: 18%) and in restaurants (1995: 64%, 2000: 39%, 2002: 37%), mean hours of ETS exposure during prior week at work (1993: 4.2, 1995: 2.3, P < 0.05, 1997: 2.2, 2001: 1.4), and at home (1993: 4.7, 2000: 3.3). Increase in: % of population covered by smoking restrictions in restaurants (1995: 26%, 2000: 69%, 2001: 78%); % of population living in a town with some form of smoking restrictions in public places (1993: 22%, 2000: 78%, 2001: 85%); % of smokers reporting that fewer than half their friends and relatives smoke (1993: 40.8%, 2000: 46.9%; P for trend = 0.03); most non‐smokers reported so both in 1993 and 2000 (1993: 90.1, 2000: 89.9).
1995‐2000 significant increase in: support for complete ban on smoking in restaurants (1995: 42%, 2000: 51%), public buildings (1995: 46%, 2000: 58%), at indoor sporting events (1995: 52%, 2000: 67%), in shopping malls (1995: 53%, 2000: 67%), but not at outdoor sporting event (1995: 15%, 2000: 19%).
1993 ‐ 1999: decline in the proportion of people who allow visitors to smoke in their homes (1993: 57%, 1999: 37%, P < 0.01), increase in the proportion of people who asked a colleague not to smoke increased (1993: 29%, 1999: 36%).

2008 ‐ 80.7% voluntary tobacco‐free homes (vs 81.5% in 2007).

2009‐ 57% of schools with comprehensive smoking policies

2014 ‐ based BRFSS exposure for ETS decreased to 10% (vs 32% in 2002); only 16.4% of houses allow smoking. and exposure to ETS >1 hr/last wk decreased to 11% (vs 32% in 2002; 14.5% in 2008).

Increase in support smoking ban in all personal spaces ‐ of people living in smoke‐free houses 87% and of those living in non smoke‐free houses 52%.

Messages were developed through formative research including focus groups. Systematic evaluation of the programme.

Media weight: Oct 1993 ‐ Dec 1996: 66 spots aired; 35 TV ads (14,901 total gross rating points [GRP]), 27 radio spots (13,644 total GRP) on smoking cessation in adults; 38 TV (17,800 total GRP) and 22 radio (13,950 total GRP) ads targeted youth; 6 TV (17,727 total GRP) and 2 radio spots (6,308 total GRP) on ETS.

Awareness/reach: 88% respondents saw any TV ads, among quitters ‐ 97%. On average 4.48 ads recognised. In 1995, 65% of the adult population were aware of the statewide anti‐tobacco campaign, and 76% recognised the tag line of the media campaign "It's time we made smoking history". Around 89% of adult smokers reported having heard, read or seen information about quitting smoking within the past year.

Dose‐response: no evidence found.

Maintenance: The campaign is ongoing.

Intervention costs: On average USD13 million per year spent on anti‐tobacco advertising. About 48% expenditure (up to Dec 1996) spent on TV time (much prime time viewing hours). MTCP activities expenditures: 1st yr ‐ USD 43 million, 3rd yr ‐ USD 35 million. Mean per capita cost for media campaign was USD 2.42.
The analysis on the basis of per capita consumption of cigarettes and average per capita media expenditures gave estimates of a fall of 0.5 packs per capita per yr for each per capita dollar spent on the media campaign.

McAlister 2004

Knowledge, attitudes, beliefs, intentions, brand preferences were assessed at baseline. Process of change variables were measured by rating by responders how much they agree or disagree (1 = strongly disagree, 5 = strongly agree) with the statements regarding attitudes to smoking and occurrence of behaviours associated with smoking cessation. At follow‐up, mean level of agreement was significantly higher among those who reported daily smoking cessation compared to those who maintained daily smoking with regard to having support for quitting and behaviours associated with smoking cessation and dealing with tension. Participants still smoking at follow‐up were significantly more likely to agree with the statements about attitudes and ability to quit . There are no follow‐up results in comparison with control group.

In message development community forums, focus groups and pre‐testing were used. No information regarding ongoing monitoring of media campaign was provided.

Media weight: no evidence found.

Awareness/reach: At follow‐up correlations between media exposures and process variables were calculated for all groups. No separate result for mass media were given.

Dose‐response: Not given for mass media campaigns without community programmes separately.

Maintenance: no evidence found.

Intervention costs: per capita media spending reported, high level and low level media represented about USD 0.25 versus USD 1.00 per capita for media spending during the year of the campaign.

McPhee 1995

Motivation to quit and self efficacy significantly more common in Santa Clara (SC) at baseline (SC/Houston: 36%/23%; 34%/ 25%). At follow‐up motivation to quit increased significantly in both communities, but did not differ (SC/Houston: 44%/ 44%), self efficacy did not change significantly and did not differ at follow‐up (SC/Houston: 32%/ 26%). 'Any household member smokes' responses similar at baseline (SC/Houston: 31%/29%) fell significantly in both communities (SC/Houston: 26%/24%). 'No friend smokes' responses not different at baseline (SC/Houston: 4%/3%) increased significantly in both communities (SC/Houston: 15%/13%). 'All or most friends smoke' responses similar at baseline (SC/Houston: 43%/46%) increased in Houston (SC/H: 44%/51%). Never‐smokers or former smokers advising family or friends to quit smoking increased from baseline in both communities (SC vs Houston: from 51% to 62% (P < 0.05) vs from 34% to 66% (P < 0.05)). Current or former smokers being advised to quit by family or friends increased from baseline in both communities (SC vs Houston: from 53% to 63% (P < 0.05) vs from 51% to 62% (P< 0.05)).

Media weight: newspaper articles ‐ 562,000 print media exposures, videotape copies distributed to 60+ Vietnamese doctors' offices and 20 community agencies in SC; nearly 42,000 brochures at 500+ location in SC, nearly 6000 self‐help quit kit copies at 250+ locations; 1140 signs with adhesive backings saying "Please do not smoke" in Vietnamese distributed; 50 copies of 3 billboards posted each month and printed in newspapers and magazines (8,000,000 print media exposures). Paid TV ads ‐13,000 secs of air time; short anti‐tobacco presentations at 30 community events. CME courses on smoking cessation ‐ 68 Vietnamese physicians.

Awareness/reach: Participants were asked if they had ever read article or seen advertisement in Vietnamese language newspaper or seen television programme or billboard in Vietnamese or heard a speech at a Vietnamese community meeting. Significantly more respondents from SC reported exposure to anti‐smoking activities in Vietnamese language (except for newspaper articles and public speaking). Smokers and non‐smokers in SC recalled significantly more intervention elements (SC vs Houston: out of a possible 5: 3.0 vs 1.6, P < 0.01 and 2.8 vs 1.4, P < 0.01). In SC significantly more physicians reported using antismoking brochures in Vietnamese, providing self‐help quit kits and referring patients to smoking cessation programmes (SC vs Houston: 86.4% vs 32.5%, P = 0.001; 66.7% vs 43.2%, P = 0.03; 35.9% vs 13.5%, P = 0.02). Quality of health education materials assessed: at follow‐up 96.4% of physicians reported they were helpful.

Dose‐response: no evidence found.

Maintenance: no evidence found.

Intervention costs: no evidence found.

McVey 2000

Smoking and health‐related attitudes assessed at baseline, no follow‐up data given.

Qualitative pilot research studies using focus groups and in‐depth interviews with smokers and ex‐smokers were conducted before and during the campaign.

Media weight: It was estimated that over the course of the campaign in the double‐weight regions each viewer could see at least 20 screenings, and in single‐weight regions about 15.

Awareness/reach: no evidence found.

Dose‐response: nearly 20,000 calls to the quit line during the campaign. No evidence of an effect of intensity of ads measured at 6 month follow‐up found.

Maintenance: After study completion the TV advertisements were shown nationally in all TV regions in England.

Intervention costs: evidence not found.

Mogielnicki 1986

Attitudes and beliefs regarding cigarette smoking were assessed on enrolment on a 5‐point rating scale (1 = disagree strongly, 5 = agree strongly). No separate results for 2nd yr follow‐up (mass media) given.

Surveys, interviews, copy tests among patients fulfilling study inclusion criteria but not included in the final study group.

Media weight: One 60‐sec main commercial and 2 x 30‐sec variants used. A series of 3‐week 'flights'; main spot on TV was broadcast 40 times, and variants 106 times, radio spot ‐ 90 times. Each flight lasted 2 ‐ 3 weeks.

Awareness/reach: Participants were asked about the recall of advertising campaign, clinic participants in media group (White River Jct): 61% did and 39% did not recall the advertising campaign at 6 month follow‐up.

Dose‐response: Among those who recalled, 43.5% were abstinent at 6 months, while among those who did not 26.7% abstinent.

Maintenance: no evidence found.

Intervention costs: total costs of commercials development and production: USD 7480, broadcast time cost: USD15,150.

North Coast QFL 1983

Attitudes to smoking and knowledge of the effects of smoking assessed in a questionnaire (6 questions each). No significant differences were found. Influences in decision to quit smoking reported ‐ the most common: health concerns (Coffs Harbour/Tamworth: 53%/ 59%), the least common: social pressure and media advertisement (Coffs Harbour/Tamworth: 1%/1%, 2%/1%), other were friends/family (Coffs Harbour/Tamworth: 15%/ 13%) and doctor's advice (Coffs Harbour/Tamworth: 7%/5%). Technique of quitting: most common ‐ 'cold turkey' (87% in both towns), quitting with help was rare (Coffs Harbour: self‐help kits 2%, group programme 1%, hypnosis 2%).

Focus groups and spot surveys were used to assess the effectiveness of the intervention techniques used.

Media weight: Information collected only on community programme exposure.

Awareness/reach: no evidence found.

Dose‐response: no evidence found.

Maintenance: no evidence found.

Intervention costs: no evidence found.

Stanford 3 City 1977

25‐item behavioural interview on participants' knowledge about risk factors (diet, weight, exercise and smoking ‐ 3 items). Significant increases in knowledge scores in intervention cities compared to control city after 2 yrs.

Mass media campaign was monitored and revised; instructional content, development, pretesting, application and reformulation co‐ordinated by study staff.

Media weight: limited data; TV programming ‐ 3 hours, 50 TV spots, 100 radio spots, several hours of radio programming, weekly newspaper columns, no details about newspaper and ads stories, billboards, printed materials sent via direct mail to participants, posters in buses, stores and work sites.

Awareness/reach: no evidence found.

Dose‐response: increases in risk factor knowledge related to increases in intervention intensity, even in Watsonville‐reconstituted group (mass media only, but a group in community received intensive face‐to‐face counselling) the gains were bigger than in Gilroy (mass media only group) and in both intervention communities the gains were bigger than in Tracy.

Maintenance: After intensive 2 yrs, 1 yr reduced level campaign.

Intervention costs: no evidence found.

Sydney QFL 1986

Additional random samples of Sydney and Melbourne smoker populations surveyed in 1983 and 1986 (Sydney/Melbourne: 1983: 271/217; 1986: 557/ 550). Health beliefs and social influences increased between 1983 and 1986 (Sydney/Melbourne: 40+ years old: males 10% to 29%/21% to 29%; females 12% to 27%/22% to 32%; < 40: males 34% to 53%/30% to 43%, females 30% to 44%/31% to 41%).
Information‐seeking behaviour was measured as the number of calls to quitline and the number of enrolments to quit centre ‐ see dose‐response.

Formative research on messages' effectiveness among target audience.

Media weight: 1983: 389 primetime ads spots (3 commercials used) ‐ the intensity alternated in 2‐wk phases: between heavy and nothing for 1st 3 months, follow‐up ad campaign of half the intensity after 5 months. After 1st year spots during prime or fringe time for approx 4 wks at the start of the campaign yr ‐ each yr nearly 40 spots/wk.

Awareness/reach: During 1st yr each month 750 people in Sydney and 200 in Melbourne interviewed on recall of campaign messages and response to the question on likelihood of giving up smoking in the next 12 months. The recall rates for the commercial most frequently shown were 87% in smokers, 82% in non‐smokers and 85% in ex‐smokers, the second in the frequency: 73%, 69% and 67% respectively. The third commercial recall rates were: smokers: 39%, non‐smokers: 31%, ex‐smokers: 33%. During campaign more smokers in Sydney indicated they were likely to quit smoking, especially after 4 months (difference: 18.6%); the difference disappeared by the end of the campaign. Long‐term assessment: TV ads recall measured each yr in separate random samples of the population of at least 1000 persons higher in Sydney (Sydney/Melbourne: 1984: males: 72%/70%, females: 80%/67%; 1986: males: 92%/68, females: 94%/67%).

Dose‐response: number of calls to quitline was measured as a direct relationship to the number of TV spots aired (presented on the graph); peak response ‐ 11,000 calls in wk 8; 50,000+ calls to the quitline in the 1st 3 months. 'Quit centre' stop smoking programmes enrolments: almost 3000 in 1983 (previous yr 500); in wk 8 with peak quitline calls number ‐ 352. 19,196 quit kits sold.

Maintenance: Since 1984 the campaigns continued in both Sydney and Melbourne till 1986, ads shown on primetime TV for 6 ‐ 8 weeks during winter months.

Intervention costs: The budget for the campaign given (AUD 620,000), most of which was used for purchase of newspaper space and radio and TV time (AUD 500,000).



Comparison 1 Mass media versus no mass media, Outcome 2 Intermediate measures.

3 Primary measures of smoking behaviour Show forest plot

Other data

No numeric data

Analysis 1.3

Study

Study population

Smoking prevalence

Tobacco consumption

Other outcomes

California TCP 2003

Selection: population‐based surveys conducted nationally and in California (CA):

National Health Interview Surveys (NHIS) 1978 ‐ 1994: household survey of a stratified, multistage, probability sample of the US population administered to all adults 18+ yrs;

Behavioral Risk Factor Surveillance System (BRFSS) 1984 ‐ 1994: state surveillance of behavioural risk factors among adults, data collected through random digit dial telephone interviews (with multistage cluster sampling method); with California Adult Tobacco Survey from 1994, data up to 2008.

Current Population Surveys (CPS) 1985, 1989, 1992/3 ‐ 1996: national survey of the US household population 15+ yrs, interviews in person; data up to 2008

California Tobacco Survey 1990‐2008

N of participants:
NHIS 1978 ‐ 1994,

response rate 79.5% to 87.8%; CA, rest of US: from 1112 to 5747, from 9205 to 39,203;

BRFSS 1984 ‐ 1994, response rate from 77% to 84%: from 1081 to 3959; with CATS from 1994 over 10,000 in CA and 80,000 to 400,000 in the rest of US.

CPS 1985 ‐ 1996: response rate from 86% to 89%: CA, rest of US: from 4076 to 8272, from 70,164 to 97,856; CPS 1992/3 to 2006/7, CA, rest of US: from 16,000 to 24,000, from 210,000 to 290,000; CTS, CA only between 44,000 and 93,000 respondents per survey.
Comparability of demographic data at baseline: not analysed.

Final data (From 1990 programme start to 2008):

All surveys (CTS, CPS and BRFSS pooled analysis years 1990 ‐ 2008, decline of smoking prevalence:

CA 0.32% points per year, rest of US 0.24% points per year.

2008 smoking prevalence: CA 13.1%,

rest of US: 19%

Interim reported data:

According to the analysis of the results of NHIS,

adult smoking prevalence decreased more rapidly from 1985 to 1990 than from 1978 to 1985 in CA and in the rest of USA . Increase in rate of decline between 2 periods in CA was 0.62% points per year (95% CI ‐1.27 to ‐0.03), and in rest of USA 0.43 (‐0.70 to ‐0.16).

Adult smoking prevalence decreased less rapidly from 1990 to 1994 than from 1985 to 1990 in CA (0.39% points per year) and in the rest of USA (0.05%, non‐sig different from zero).

Between 1978 and 1994 adult smoking prevalence was 2 ‐ 5 percentage points lower in CA than in the rest of USA, but the estimated rate of decline in smoking prevalence was no‐sig different from that of the rest of USA during any of the 3 time periods.
Combined estimated rate of decline before CA TCP began was similar in CA to the rest of USA. Rates of decline were not statistically different but estimated prevalence in CA was lower than in the rest of USA. The rate of decline increased significantly in CA at the early period of the programme but not in the rest of USA. In the late phase of the programme the rate of decline in CA and in the rest of USA was non‐sig different from zero and in both cases it was less than in the previous period.
BRFSS trends similar, but consistently around 2% lower.
CPS, CTS and CATS trends similar to BFRSS (all telephone surveys, while NHIS is face‐to‐face).

NHIS only: rate of change in smoking prevalence 1978 ‐ 1985, CA vs rest of US:
Adults aged 18+ yrs:

‐0.6 (95% CI ‐0.79 to ‐0.40) vs ‐0.5 (‐0.67 to ‐0.33);
1985 ‐ 1990: ‐1.22* (‐1.51 to ‐0.93) vs ‐0.93* (‐1.13 to ‐0.73);
1990 ‐ 1994: ‐0.39* (‐0.76 to ‐0.03) vs ‐0.05* (‐0.34 to +0.24);
Adults aged 25+ yrs:
1978 ‐ 1985: ‐0.61(‐0.83 to ‐0.39) vs ‐0.43 (‐0.63 to ‐0.24); 1985 ‐ 1990: ‐1.11* (‐1.37 to ‐0.84) vs ‐0.88* (‐1.10 to ‐0.66);
1990 ‐ 94: ‐0.61 (‐0.99 to ‐0.23) vs ‐0.20* (‐0.52 to +0.12); *significant difference (P < 0.05) between the estimated rate of change for that period and that for the previous period.

All surveys combined analysis: CA, rest of US rate of decline % (SE) ‐ smoking prevalence %:
pre‐1989 (preprogramme): ‐0.74 (0.12) ‐ 23.3, ‐0.77 (0.09) ‐ 26.2;
1989 ‐ 1993 (early programme): ‐1.06 (0.17) ‐ 18.0, ‐0.57 (0.14) ‐ 23.3;
1994 ‐ 1996 (late programme): 0.01 (0.21) ‐ 18.0, ‐0.28 (0.26) ‐ 22.4; P < 0.05 CA vs rest of US, P < 0.001, change from previous period.

Final data (1970 ‐ 2008), per capita taxable sales:

1970 per capita sales similar in CA and rest of US.

In 1989, CA sales 26.1% lower than sales for the rest of the US (108.8 versus 147.2 packs per year).

Faster rate of decline in CA vs rest of the US.

2002 CA vs rest of the US lower sales (48 versus 101 packs per capita per year).

From 2002 slower rate of decline in CA vs rest of the US (from 44.6 to 40.4 packs per year, a decline of 9.4%), but still lower sales than in the rest of the US. 2008: 40 versus 77 packs per capita per year, or 3.37 versus 6.42 packs per capita per month).

1989/90 to 2006/7, per capita consumption

greater decline in CA vs rest of the US: 60.8% vs 41.0%

(40 vs 92 packs per person per year).

Per capita consumption based on aggregated sales data declined faster in CA in the early period of the campaign than before it started, and the decline was significantly greater than in the rest of USA. During late programme the decline slowed but remained greater than in the rest of USA.
Rate of decline in per capita consumption based on aggregated sales data CA/rest of US pack (SE), packs/month:
Baseline 1989: ‐0.42* (0.03), 9.7/ ‐0.36 (0.02), 12.5;
Early programme 1989 ‐ 1993: ‐0.64#^ (0.03), 6.5/ ‐0.42 (0.03), 10.4;
Late programme 1994 ‐ 1996: ‐0.17 *# (0.07), 6.0/ 0.04 # (0.06), 10.5.

In another analysis presenting the results up to 1999 the rate of decline of tobacco consumption (tobacco consumption pack/month), CA/rest of US: Baseline 1/1983 ‐ 12/1988: ‐0.46 (9.5)/ ‐0.35 (12.4);
Early programme 1/1989 ‐ 12/1993: ‐0.58 (6.6)/ ‐0.4 (10.3);
Mid‐programme 1/1994‐10/1998: ‐0.16 (5.8)/ ‐0.07 (10);
Recent programme 10/1998‐12/1999: ‐1.56 (4.1)/ ‐0.78 (9.1).

In analysis presenting the results for tobacco consumption up to 2002, CA,/rest of US packs/month
1988: 9.8/12.5;
2002: 3.9/7.5.

* CA vs rest of US P < 0.01.
^CA vs rest of US P < 0.001.
#change from previous period P < 0.001.

Quit ratio, defined as % of ever‐smokers (current and former) who were former smokers in a given year, accelerated non‐sig in CA and in the rest of US between 1985 and 1990. 1990 to 1994, rate of increase in quit ratio decreased, with no significant changes in CA and in the rest of US compared to the period of 1985 to 1990.

Quit ratio: NHIS only, 1978 ‐ 1994, CA, rest of US, adults 18+years: 1978 ‐ 1985: 0.73 (0.22 to 1.24), 0.73 (0.40 to 1.05);
1985 ‐ 1990: 1.36 (0.74 to 1.97), 1.04 (0.62 to 1.46);
1990 ‐ 94: 0.18 (‐0.8 to 1.15), 0.15 (‐0.47 to 0.77);

Quit attempts: mean number of quit attempts in last yr decreased between 1990 and 1992 from 48.9% to 38.1%, then increased in 1996 and 1999 to 56.0% and 61.5%.
% of smokers attempting quitting and abstinent for 3m at time of survey increased (1990:15.3%, 1992:18.6%, 1993: 20.2%).
% of smokers with quit attempt in last yr lasting 7 days+ increased between 1990 and 2002 (1990: 29.2%, 1992: 25.1%, 1996: 36.1, 1999: 41.4%, 2002: 40.5%).

% of smokers who are currently quit for 3+ months did not change significantly (1990: 8.5 ± 1.0%, 1999: 5.4 ± 0.5%, 2008: 8.0 ± 2.5%)

% of recent smokers currently in a quit attempt of 6+ months did not change significantly (1990: 5.6 ± 0.7%, 1999: 4.2 ± 0.5%, 2008: 6.4 ± 2.5%)

CORIS 1997

Selection: Total population included in baseline and 4‐yr follow‐up surveys (15 ‐ 64 yrs at baseline and 15 ‐ 68 at 4‐yr follow‐up).
At 12‐yr follow‐up a random sample of white participants aged 15 ‐ 64 was selected ‐ about 50 people per sex‐ and age‐specific decile were selected, excluding people living < 2 yrs in the community.
High‐risk cohort was identified within cohort sample ‐ people who had at least 1 of: high cholesterol (> 20th percentile for age), high blood pressure (systolic > 160 or diastolic > 95 mm Hg or antihypertensive drug use), regular smokers.

N of participants:
Total population Swellendam [Intervention]/Riversdale [Control] [M: men; F: women]. Baseline 1979 (% 1980 census): M: 1224 (65)/ 1082 (60); F: 1396 (74)/ 1208 (68); Follow‐up: 1983 (% of 1980 census): M: 1171 (65)/ 1109 (62); F: 1323 (70)/ 1150 (64); 12‐yr follow‐up 1991: random sample Swellendam/Riversdale: M: 273/269; F: 267/274; response rate not reported.
Cohort Swellendam/Riversdale (% of total population): M: 710 (58)/595 (55); F: 821 (59)/710 (59);
High‐risk cohort, Swellendam/ Riversdale (% of cohort): M: 388 (55)/320 (53); F: 234 (29)/224 (32). Comparability of demographic data at baseline: Authors state that age and sex distributions were very similar.

In total population % of smokers was lower in men and women in intervention compared to control city (non‐sig difference).
Baseline prevalence (1979) and (% change) at 4 yrs (1983): Swellendam/ Riversdale: M: 49.5 (‐9.1)/45.7 (‐7.6); F: 17.6 (‐3.6)/16.1 (‐0.5).
At 4 yr follow‐up in the intervention cohort compared to control cohort the % of smokers decreased (non‐sig) in women but not in men.
Cohort: baseline Swellendam/ Riversdale, and (net change): M: 46.5/44.4 (+0.9); F: 14.5 /14.4 (‐3.0);
At 4 yr follow‐up in the high‐risk cohort in the intervention group compared to control group the % of smokers decreased (non‐sig) in women but not in men.
High‐risk cohort baseline Swellendam/ Riversdale (net change): M: 85.3/82.5 (+1.3), F: 50.9/45.5 (‐7.4);
At 12 yr (1991) follow‐up smoking prevalence was lower in intervention compared to control city.
1991 random sample smoking prevalence Swellendam/ Riversdale: M: 25.3*/34.2; F: 12.4/12.8; *significantly lower than Riversdale.

In total population tobacco consumption was lower in men and women in intervention compared to control city (non‐sig difference).
Baseline per capita consumption of tobacco grams/day (1979) and % change at 4 years (1983): Swellendam/Riversdale: M: 11.2/‐2.6, 8.8/‐1.8; F: 2.6/‐0.4, 2.3/0.1;
At 4 yr follow‐up in the intervention cohort compared to control cohort tobacco consumption decreased in men and women (non‐sig difference).
Cohort baseline Swellendam/ Riversdale and (net change): M: 11.3/9.2 (‐0.4); F: 2.1/2.3 (‐0.2).
At 4 yr follow‐up in the high‐risk cohort in the intervention group compared to control group tobacco consumption decreased in men and women (non‐sig difference). High‐risk cohort baseline Swellendam/Riversdale (net change): M: 20.5/17.3 (‐0.8), F: 7.5/7.3 (‐0.4).
At 12 yr (1991) follow‐up tobacco consumption was lower in intervention compared to control city. 1991 random sample per capita consumption Swllendam/Riversdale: M: 4.8/5.8, W: 1.9/1.8.

In women quit rates were significantly lower in intervention compared to control city.
Quit rates: Swellendam/Riversdale: after 4 yrs: % of all smokers: M: 16.9/20.1; F: 28.3*/15.5; Light smokers: M: 26.5/32.6**; F: 44.4**/28.6**; Heavy smokers: M: 15.8/17.7; F: 23.7/10.7.

*significant difference compared to control; **significant compared with heavy smokers.

Jenkins 1997

Selection: Phone surveys of randomly selected Vietnamese men, with numbers chosen randomly from 23 most common Vietnamese surnames listed in area phone books. After enumerating all men aged 18+ living in the household and speaking Vietnamese, subjects for interview were selected according to random age rankings in the household.

N of participants:
San Francisco (SF)/Houston: Baseline (% response rate):
1990: 1133 (84)/1581 (82); Follow‐up (response rate %): 1992: 1202 (94)/1209 (88). Comparability of demographic data at baseline: significant differences in educational level, English language proficiency, income, unemployment and mean immigration year.

Smoking prevalence did not differ significantly at baseline. Post‐intervention, smoking prevalence fell significantly (P = 0.004) in SF and increased in Houston. Current smokers % among Vietnamese men: SF/Houston:
Baseline (1990): 36.1/39.6;
Follow‐up (1992): 33.9/40.9. Significant intervention effect (P ⋝ 0.01). The odds of being a smoker at follow‐up were significantly lower in intervention than in control community; OR: 0.82, 95% CI: 0.68 to 0.99). The odds of being a smoker after the intervention were lower for following characteristics: age groups 18 ‐ 24, 45+; at least a college education; good or fluent English; being a student. The odds of being a smoker were higher for unemployed and more recent immigrants.

Significant difference in cpd at baseline (Houston higher), significant decrease in Houston, but not in SF. At follow‐up mean cpd was significantly higher in Houston.
Mean cpd among smokers: SF/Houston, Baseline 1990: 11.1/13.2;
Follow‐up 1992: 10.3/11.9.

Significant increase in quit rate in SF compared to Houston (P = 0.017).
% Quit rates in past 2 yrs: SF/Houston:
Baseline 1990: 7.2/5.8;
Follow‐up 1992: 10.2/7.4; significant increase in SF, but not in Houston.
The odds of being a quitter in 1992 were significantly higher in intervention compared to control community (OR: 1.65, 95% CI: 1.27 to 2.15). Predictors for quitting included: being a student (OR: 2.19, 95% CI 1.45 to 3.33); more recent year of immigration (OR: 1.03, 95% CI 1.0 to 1.05); each additional year of age (OR: 1.03, 95% CI 1.02 to 1.04); at least high school education (OR: 1.33, 95% CI 1.04 to 1.7).
Mean number of quit attempts was similar in both communities at baseline and increased significantly in Houston but not in SF. Mean number of quit attempts: SF/Houston:
Baseline 1990: 1.4/1.1
Follow‐up 1992: 2.1/2.5.
At baseline % reporting any quit attempt in the past was significantly higher in SF. At follow‐up it increased significantly in both communities, but did not differ. Percentage reporting any quit attempt in the past: SF/Houston:
Baseline 1990: 61%/49%
Follow‐up 1992: 73%/77%.

Massachusetts 2003

Selection: population‐based survey in Massachusetts (MA) and in the rest of USA (excluding CA) ‐ Behavioral Risk Factor Surveillance System (BRFSS): annual, state‐based, standardised, random digit dialled telephone survey of non‐institutionalised US adults aged 18+; 42 states including MA and CA participated consistently in BRFSS between 1989 and 1998; MA tobacco surveys conducted since 1993‐4 (baseline) in adults, and every month since March 1995, estimates of adult smoking prevalence are derived from household screening interviews with an adult informant who reported on smoking status for all adult members of the household.

N of participants:
1990/9: 22,309 responses from MA, 946,241 from the rest of USA;
BRFSS 1989/98 sample sizes in MA from 1221 to 4944; in rest of USA (excluding CA) from 63,255 to 113,214.
MA surveys 1994/98 respondents: from 5736 to 21,909. Response rates for BRFSS were 77% to 84% between 1984 and 1994; Between 1995 and 1999 response rates for USA fell from 68.4% to 55.2% and in MA from 60.4% to 42.7%.
Comparability of demographic data at baseline: MA respondents were more likely to be white non‐Hispanic and more likely to be college graduates than the respondents from the rest of USA.

According to the analysis based on BRFSS 1989 to 1998 and MA Tobacco Surveys 1993 to 1999 the slope of smoking prevalence after 1992 was significantly different from zero and from the slope for the rest of USA. Based only on BRFSS 1990 ‐ 1999 controlling for age, sex, race, and education there was a greater decline in current smoking between 1990 and 1999 among MA men than among MA women, and the decline was greater in MA than in the rest of the USA for men and for both sexes combined.

Based on BRFSS 1989 to 1998 and MA Tobacco Surveys 1993 to 1999: for MA the slope of smoking prevalence after 1992 was ‐0.43*% a year ( 95% CI ‐0.66% to ‐0.21; P = 0.001); for the rest of USA the slope after 1992 was +0.03% a year (95% CI ‐0.06% to 0.12%; P = 0.46), * significantly different from zero and from the slope for the rest of USA (P < 0.001).
BRFSS only 1990 ‐ 1999: Baseline % prevalence (95% CI) total pop 1990, MA/rest of USA: 23.5 (21.0 to 26.1)/24.2 (23.7 to 24.7);
Follow‐up 1999: 19.4 (18.0 to 20.8)/23.3 (22.9 to 23.7); significantly lower than in the rest of USA (P < 0.001); crude prevalence OR of current smoking in MA in 1999 compared to 1990: 0.78 (0.66 to 0.92, P trend = 0.01); adjusted for sex, age, race, education OR: 0.83, (0.70 to 0.99, P trend = 0.08); in rest of USA prevalence of current smoking in 1999 compared to 1990: OR 0.95 (0.92 to 0.99, P trend = 0.99); adjusted for sex, age, race, education OR: 1.01 (0.97 to 1.05, P trend < 0.001). Between 1990 and 1999 average change in the log odds was ‐1.3% in MA and in rest of USA +0.6% (sig difference between slopes, P = 0.01). Men/women baseline prevalence % (95% CI) 1990, MA/rest of USA: M: 25.9 (22.0 to 29.8)/26.0 (25.2 to 26.7); W: 21.5 (18.2 to 24.8)/22.5 (21.9 to 23.2);
Follow‐up 1999: M: 19.5* (17.3 to 21.6)/25.6 (24.9 to 26.2); *significantly different from the rest of USA, P < 0.001; W: 19.3* (17.5 to 21.1). 21.2 (20.7 to 21.7), * significantly different from the rest of USA P = 0.04;
Crude prevalence OR (95% CI) of current smoking in MA/rest of USA in 1999 compared to 1990:
M: OR: 0.69 ( 0.54 to 0.89; P trend = 0.03)/0.98 (0.93 to 1.03; P trend = 0.07); Adjusted for age, race/ethnicity. education OR: 0.73 (0.56 to 0.94; P trend = 0.09)/1.03 (0.97 to 1.08, P trend < 0.001);
W: crude OR: 0.87 (0.70 to 1.09; P trend = 0.09)/0.93 (0.88 to 0.97, P trend = 0.03);
Adjusted for age, race/ethnicity, education OR: 0.95 (0.75 to 1.2; P trend = 0.43)/0.99 (0.95 to 1.04, P trend = 0.03);
Average decline per year in the log odds of current smoking between 1990 and 1999 in MA was M: 1.8% (SE: 0.011) and W: 0.7% (SE: 0.010), and in rest of USA there was an average annual increase of : M: 0.8% (SE: 0.002), W: 0.4% (SE: 0.002); Men: significant difference between MA and USA, P = 0.016; W: non‐sig difference between MA and USA P = 0.243.

Annual per capita tobacco consumption based on aggregated sales data:
1988 ‐ 1992 ‐ before tax increase declines in consumption for MA adults and for rest of USA were similar: 15%, 14% ‐ this = annual decline 3 ‐ 4%.
In 1993 consumption declined by 12% in MA and by 4% in rest of USA.
After 1993 there was consistent annual decline of more than 4% in MA and less then 1% in rest of USA.

Quit ratio: not reported.
Quit attempts: % of smokers planning to quit within the next 30 days increased between 1993 and 1997 (from 22% to 42%). Percentage of past year smokers making quit attempt increased non‐sig between 1993 and 1996 (from 47.5% to 54.2%). Percentage of quitters making successful quit attempt significantly increased between 1993 and 2000 (from 18% to 25%), % of smoking pregnant women decreased (from 25% in 1990 to 11% in 1999, while in the rest of US these %s were 18% and 12% respectively).

McAlister 2004

Selection: telephone surveys of randomly selected (random digit dialing) adults from Texas (including individuals not resident in treatment areas); respondent in each household with the most recent birthday aged 18+ selected. Cohort of smokers identified in baseline cross‐sectional sample was followed.

N of participants:
1069 daily cig smokers identified in baseline survey; 622 available for follow‐up.
Baseline: High media 133, low media 274, control 232: Follow‐up (% of original sample): 87 (65), 158 (58), 137 (59). Comparability of demographic data at baseline: not analysed.

Not reported ‐ only levels of daily smoking given (15.7 at baseline, 17.5 at follow up)..

Not reported.

Complete cessation achieved by approx 2% of panel of daily smokers.
% quitting daily smoking, i.e. reduction, not complete cessation (data estimated from bar graph): non‐significantly higher in high and low media groups compared to group without any intervention, both in the whole sample and in followed‐up subsample.
% of whole sample quitting daily smoking: high media 4.5, low media 4.7, control 3.0 (taken from graph); Followed group 7.0%, 8.0%, 5%.
Quit attempts: Among the participants still smoking at follow‐up 27.3% had made quit attempt.

McPhee 1995

Selection: Phone surveys of randomly selected Vietnamese men, chosen randomly from 23 most common Vietnamese surnames in area telephone books. After enumerating all men aged 18+ living in the household and speaking Vietnamese, subjects for interview were selected according to modified Kish procedure.

N of participants:
Santa Clara(SC)/Houston: Baseline (% response rate):
1990: 1322 (81)/1581 (82); Follow‐up (% response rate)
1992 (response rate): 1264 (85)/1209 (88).
Comparability of demographic data at baseline: sig differences in educational level, English language proficiency, mean year of immigration and unemployment.

At baseline smoking prevalence in SC differed significantly from Houston (control community). It slightly increased in control area and remained unchanged in intervention area ‐ there was no intervention effect. Current smokers among Vietnamese men, SC/ Houston: Baseline 1990: 36%/40%;
Followup 1992: 36%/41%.

At baseline SC cig consumption differed significantly from Houston. It fell significantly in the control area , but there was no significant intervention effect. Mean cpd among smokers: SC/Houston: Baseline 1990: 9.9/13.2;
Follow‐up 1992: 9.6/12.0.

Quit rates in past 2 yrs differed significantly at baseline, but there was no significant intervention effect.
SC/Houston: Baseline 1990: 8.0%/6.0%,
Follow‐up 1992: 10%/7%.

Mean number of quit attempts was significantly higher in SC at baseline. At follow‐up it increased significantly in both communities, but did not differ. Mean number of quit attempts: SC/Houston:
Baseline 1990: 1.3/1.1.
Follow‐up 1992: 2.3/2.5.
At baseline percentage reporting any quit attempt in the past was significantly higher in SC. At follow‐up it increased significantly in both communities, but did not differ. Percentage reporting any quit attempt in the past: SC/Houston:
Baseline 1990: 64%/49%.
Follow‐up 1992: 79%/77%.
Adjusted OR for intervention in regression model was 1.1 (95% CI 0.9 to 1.4), i.e. no sig effect of programme.

McVey 2000

Selection: In each TV region a random sample of enumeration districts were selected (1 in 40) and within each district a random sample of households (1 in 20) using computer‐generated lists. Selected households were visited by interviewers and a resident member was selected for the sample with the use of pseudo‐random Kish‐grid method. Only adults at least 16 yrs were sampled. Only smokers and ex‐smokers were included in the study. Attempts were made to re‐interview the same respondents 6m and 18m later with similar structured questionnaire.

N of participants:
TV‐media (smokers/ex‐smokers), Baseline sample: 1744/1256;
Followed at 6m: 1064/854; Followed at 18m: 673/598; Control (smokers/ex‐smokers),
Baseline sample: 719/775;
Followed at 6m: 475/571;
Followed at 18m: 288/402.
Comparability of demographic data at baseline: no statistical analyses.

Not reported other than varying in different target regions.

Not reported.

By 18m, more successfully followed smokers in TV media group gave up smoking than in control group (non‐sig). Quit rates in smokers (TV media/control): 9.7%/8.7%; OR: 1.27 (95% CI 0.77 to 2.08), adjusted for predictors of giving up cigarettes: sex, age, manual/non‐manual job, daily cig consumption, "want to reduce smoking" and sex by manual/non‐manual work interaction. More ex‐smokers not relapsing were observed in TV media than in control group (significant): 96.3% vs 94.5%, adjusted OR: 2.21 (95% CI 1.11 to 4.40, P = 0.025); adjusted for predictors of remaining off cigs: number of cpd before stopping, length of time since stopping, whether persuaded to stop smoking by somebody else. TV media compared to no intervention was estimated to have increased the odds of not smoking.
Pooled common effect on not smoking: adjusted OR: 1.53 (1.02 to 2.29, P = 0.04).

Mogielnicki 1986

Selection: reception clerks asked all patients registering for clinic visits about smoking and interest in stopping.

N of participants:
In 2nd yr clinic response rates not reported (media group/control group) Baseline: 71/33; Follow‐up (% retention rate): 38 (54)/17 (52);
Mailing group: Baseline: no information given;
Follow‐up: 12/5, response rates not reported.
Comparability of demographic data at baseline for clinic participants: non‐sig differences in age, number of cigs smoked, thiocyanate level, % smoking high‐tar cigarettes. No details for mailing group participants given.

Not reported.

Not reported.

In clinic participants, number abstinent was higher in group receiving mass media compared to group not receiving mass media (calculated for all participants included in the study at baseline). In the mailing group in both media and no‐media groups no participant was abstinent and no number of participants included in the study at baseline was given.
Abstinence rates % (media group vs control group), clinic participants, total sample: 19.7 vs 3;
Followed sample: 36.8 vs 5.9.
Mailing group participants, followed sample: 0% vs 0%.

North Coast QFL 1983

Selection: systematic random sample of men and women 18+ from each town, up to 2 adults per household.

N of participants:
Coffs Harbour (CH)/Tamworth (control):
Baseline 1978 (% response rate): 612 (71)/589 (72);
1980: 1272 (73)/1239 (74); 1981: 1195 (73)/1195 (74). Comparability of demographic data at baseline: age and sex differences across towns and yrs.

Significant effect of the intervention was found.
Prevalence reported for 1978 (baseline), 1980, 1981 by town, age, sex and yr. In both towns younger people tended to have bigger declines and 65+ smallest. % points change in prevalence between 1978 and 1981: in CH Men (M) from 7.1 (65+ yrs) to 11.2 (18 ‐ 25 yrs) decline, Women (F): from 6.0 (65+ yrs) to 11.1 (18 ‐ 25 yrs) decline; in Tamworth M 4.1 (65+ yrs) to 5.1 (36 ‐ 45 and 46 ‐ 55 yrs), F 2.1 ( 65+ yrs) to 5.1 (18 ‐ 25 yrs). Effect of intervention compared to control was significant (P < 0.05).

Not reported.

Quit rates not reported.

Stanford 3 City 1977

Selection: in each city random multistage probability sample of men and women 35 ‐ 59 yrs old.

N of participants:
Watsonville/Gilroy/Tracy; Baseline (% of original sample): 605 (73)/542 (82)/532 (81);
Completing yr 2 follow‐up (% of baseline sample): 423 (70)/397 (73)/384 (72).
High‐risk subjects ‐ Watsonville media only/Gilroy/Tracy, Baseline: 56, 139, 136; Completing baseline and 2 yr follow‐up: 40, 94, 95. Comparability of demographic data at baseline: no statistical comparisons made.

Reported only for high‐risk group (baseline/1st /2nd /3rd year follow‐ups):
Watsonville‐randomised control (media only): 56.8/no results given as difference in the direction contrary to prediction;
Gilroy: 62.4/‐15.1/‐15.1/‐11.3;
Tracy: 52.8/‐6.4/‐10.6/‐14.9,
all non‐sig.

Per capita cig consumption for adults 35 ‐ 59 yrs (baseline/% change 1973/1974):
Watsonville reconstituted: 6.8/‐6.9,‐13.7*; Gilroy: 6.8/‐2.3/‐7.3;
Tracy: 6.9/‐1.1/‐2.5;
* statistically sig difference (P < 0.05) for % change values compared to Gilroy and Tracy (control).
High‐risk subjects (baseline 1972/% change 1973/1974): Watsonville‐randomised control: 14.2/‐5.8/‐15.1;
Gilroy: 14.6/‐9.8/‐13.8;
Tracy: 13.7/‐8.5/‐17.2.
In the publication presenting results for high‐risk group only the results are given for 3 yrs follow‐ up and they differ from the main Stanford Three City publication (Maccoby 1977) as they present the results for subjects who completed baseline and all 3 annual follow‐up surveys (baseline 1972/% change; 1973/1974/1975), Watsonville‐randomised control: 15.4/‐5.8/‐15.1/‐16.0;
Gilroy: 13.8/‐7.0/‐12.3/‐11.8;
Tracy: 14.0/‐7.4/‐15.9/‐21.0; all non‐sig.

Quit rates not reported.

Sydney QFL 1986

Selection: random weekly selection from list of all Australian electoral subdivisions. Selected 10 separate households starting from randomly selected address from each electoral subdivision (clockwise direction around the residential block). Within household youngest person 14+ or 16+ interviewed.

N of participants:
Cross‐sectional surveys (Sydney/rest of Australia), Baseline 1983: 3978/ 5154; Follow‐up 1984: 4051/4318. Melbourne cross sectional survey ‐ 1518.
Cohort (Sydney/Melbourne), Baseline 1983 : 900/600;
Follow‐up 1984 (% retention rate after excluding people moved): 570 (76)/364 (73). Australia‐wide response rate of approximately 60%.
Long‐term follow‐up weekly surveys, Sydney and Melbourne 1981 ‐ 1987: 68,136 males, 70,634 females.
Comparability of demographic data at baseline: no statistical comparisons made.

At 1st cross‐sectional surveys prevalence decreased in Sydney compared to the rest of Australia (non‐sig).
Baseline prevalence 1983/% change at follow‐up 1984; cross‐sectional Sydney: Men (M): 35.9/‐4.2; Women (F): 30.4/‐1.6; Total: 33.1/‐2.8; Rest of Australia: M: 39.2/‐3.3; F: 29.3/‐0.2; Total: 34.1/‐1.6.
Difference between Sydney and rest of Australia: 1.2% (SE 1.49; non‐sig).
In cohort study significant decrease in smoking prevalence in Sydney compared to Melbourne.
Cohort % Sydney/Melbourne: 30.9 (‐3.4)/36.8 (+0.)%. Standardised (to baseline prevalence) difference Sydney vs Melbourne: 5.4% (SE: 1.89, P < 0.01).
Overall campaign effect assessed as a weighted mean of cross‐sectional and cohort assessments showed significant effect of campaign on smoking prevalence. Overall campaign effect weighted mean: 2.8% (95% CI: 0.5 to 5.1).
In model assessing long‐term effects of the campaign (1981 ‐ 1987) there was an immediate decrease in smoking prevalence after the beginning of the intervention (6m) in Sydney and Melbourne in males and females. Continuation of the campaign in subsequent years was associated with further drop in percentage points in Sydney and Melbourne men, but not women. Long‐term effectiveness model: Sydney (baseline estimated prevalence 1981 ‐ June 1983: immediate campaign effect 1983 ‐ 6m % points change/continuation of the campaign 1983 ‐ 1987 % points change per year): M: 38.7/‐2.52/‐1.12; F: 31.6/‐2.61/no sig decline. Melbourne (baseline estimated prevalence 1981 ‐ June 1984: immediate campaign effect 1984 ‐ 6m % points change/continuation of the campaign 1984 ‐ 1987 % points change per year): M: 40.1/‐2.87/‐1.9; F: 30.9/‐2.5/no decline.

Cross‐sectional studies after 1st issue of the campaign found decrease in cpd in Sydney compared to the rest of Australia (non‐sig).
Changes in mean cpd in Sydney and rest of Australia,
Baseline 1983/% change in 1984:
M: 20.1 (‐0.6)/20.3 (+0.2); F: 18.4 (‐1.3)/18.2 (‐0.8).

Significantly larger proportion of Sydney smokers compared to Melbourne smokers quit smoking or cut down on cigs. Relapse rate and initiation rate were similar in both cities.
Cohort Sydney/Melbourne 1983 ‐ 1984: initial smokers % quit: 23/9; initial smokers cut down: 12/9; total quit + cut down: 35*/18; initial ex‐smokers relapse: 10/11; initial non‐smokers started: 4/4.

Quit attempts: Failed attempts to quit or cut down were assessed in cohort of smokers in Sydney and Melbourne ‐ proportions were not significantly different: S vs M: quit: 11% vs 19%, cut down: 20% vs 23%.

* P < 0.05.



Comparison 1 Mass media versus no mass media, Outcome 3 Primary measures of smoking behaviour.

4 Study summary by type of outcome Show forest plot

Other data

No numeric data

Analysis 1.4

Study

Type of outcome

Orientation

Cost

Effect

California TCP 2003

Prevalence







Packs per day

Social diffusion theory, with social marketing and social policy change

April 1990 ‐ June 1993 USD 26m for media campaign.
1990 ‐ 5 total programme spend USD 694m

Pre‐programme prevalence 23.3 in CA vs 26.2 in rest of USA. By 1993 down to 18.0 in CA, 23.3 rest of USA; By 1996, prevalence still 18.0 in CA vs 22.4 in rest of USA.

Final data (2008):

CA 13.1%,

rest of US: 19%

Rate of decline % (SE): CA: Pre‐TCP: ‐0.74 (0.12) > early period : ‐1.06 (0.17) [P < 0.05 CA vs USA, P < 0.001 change from pre‐TCP] > late period 0.01 (0.21) P < 0.05 CA vs USA.
USA: ‐0.77 (0.09) > 0.57 (0.14) > 0.28 (0.26).

Final data (up to 2008):

CA 0.32% points per year, rest of US 0.24% points per year.

Media campaign alone (without other Prop 99 components) led to decline in cig consumption of 12.2% between April 1990 and March 1991.
Decline of 7.7 packs per capita (pc) attributable to media campaign alone, with 10% increased expenditure on media yielding 0.5% reduction in cig sales.
1989 ‐ 96, CA spent USD 0.50 pc per year on media, leading to a fall of 3.9 packs pc per yr for each USD spent on media.

Final data (1970 ‐ 2008), per capita taxable sales:

CA sales 26.1% lower than sales for the rest of the US (108.8 versus 147.2 packs per year).

Faster decline in CA vs rest of the US.

2002 48 versus 101 pc per yr

2008: 40 versus 77 pc per yr,

CORIS 1997

Prevalence




Cigarettes per day

Not reported

pc cost of USD 5 over 4 yrs in media‐only intervention town.

After 4 yrs, Intervention (Control) prevalence in men dropped from 49.5 (45.7) to 40.4 (38.1) , and in women from 17.6 (16.1) to 14.0 (15.6) . Data contains paired and unpaired observations, in total population.

After 4 yrs, Intervention (Control) tobacco per day (pd) in men moved from 11.2 (8.8) to 8.6 (7.0) grams, and in women from 2.6 (2.3) to 2.2 (2.4).

Jenkins 1997

Prevalence



Cigarettes per day


Odds of quitting

Not reported

Not reported

At 2 yrs, San Francisco (int) prevalence down from 36.1 to 33.9 (P ≤ 0.01), and Houston (control) up from 39.6 to 40.9. Net change was ‐3.5 % points (P = 0.004).

San Francisco (Houston) cpd pretest: 11.1 (13.2); post‐test: 10.3 (11.9)

OR 1.65 (1.27 to 2.15) in favour of San Francisco

Massachusetts 2003

Prevalence




Packs per day

Social diffusion theory, with social marketing and social policy change

USD 39m pa for whole programme.




MA pc USD 6.50 (in 2000); cf. CA USD 3.31, UK $0.89, Australia $0.48 (1997), South Africa $0.04, France $0.32, spent on tobacco control programmes.

1990 ‐ 9: MA prevalence declined from 23.5 (CI 21.0 to 26.1) to 19.4 (CI 18.0 to 20.8).
41 US states: 24.2 (CI 23.7 to 24.7) to 23.3 (CI 22.9 to 23.7)

1993 ‐ 6: reduction of 0.5 per annum (pa) for each pc USD spent on media.
From 1993 pc MA consumption declined > 4% pa, compared with < 1% pa in comparison states.

McAlister 2004

Prevalence

Daily smoking cessation

Cessation

Social learning theory, transtheoretical model. using modelling, social reinforcement for behaviour change, and emotional arousal.

USD 9m pa

Prevalence of daily smoking increased from 15.7 to 17.5.

% of baseline smokers ceasing daily smoking: Low‐level media: 4.7, High‐level media 4.5, No media 3.0

Media‐only areas achieved 8% cessation vs control 5%.

McPhee 1995

Prevalence



Cigarettes per day

Not reported

Not reported

No change in prevalence in Intervention community from baseline to 2 yrs: 36.0, while controls increased (non‐sig) from 40 to 41.

Intervention cpd down from 9.9 to 9.6, controls down from 13.2 to 12.0.

McVey 2000

Quit and relapse rates.

No formal attributing, but 'morbid humour' used.

Not reported

At 18m, 9.8% of intervention smokers vs 8.7% of control had quit, and 4.3% of intervention ex‐smokers vs 3.7% controls had relapsed. Pooled OR of not smoking was 1.53 (CI 1.02 to 2.29, P = 0.04).

Mogielnicki 1986

Cessation rates

Mass marketing techniques (situation analysis, objective definition, copy development, media plan, campaign launch) combined with traditional clinical trial approach.

Copy development and production: USD 7480, Air time: USD 15,150 (1980 USD)

Per protocol clinic cessation at 2nd year:
Media only 36.8%, No media 5.9%.
Mailing recipients: no quitters in either group.

North Coast QFL 1983

Prevalence

Social marketing and communication theory

Not reported

Mean declines in prevalence in Intervention community of 9.5% from baseline to Yr 3, and in controls of 4.4%.

Stanford 3 City 1977

Prevalence



Cigarettes per day

Social marketing, social learning and communication theory

Not reported

All participants: Baseline: Intervention 65.5, Control 55.6.
Cohort: Baseline Intervention (Control) 62.4 (52.8), declined by 11.3% (14.9%) after 3 yrs (P < 0.05).

Mean Intervention cpd down 7.3% over 2 yrs from 6.8 at baseline; controls down 2.5% from 6.9 at baseline.

N.B. Within treatment groups, cohort baseline cpd of Int 13.8, control 14.0.



Comparison 1 Mass media versus no mass media, Outcome 4 Study summary by type of outcome.

5 Baseline differences and possible confounding Show forest plot

Other data

No numeric data

Analysis 1.5

Study

Differences

Possible confounders

Adjusted effects

Jenkins 1997

San Francisco (Int) respondents significantly less proficient in English, of lower education, lower income, less likely to be employed and more recent year of immigration than Houston (control) respondents.

All these factors were included in multivariate analyses.

Unadjusted ORs not reported. Strongest predictor of quitting was being a student (adjusted OR 2.19, 95% CI 1.45 to 3.33); at least high school education (OR 1.33: CI 1.04 to 1.70); more recent year of immigration (OR 1.03: CI 1.00 to 1.05); each + yr of age (OR 1.03: CI 1.02 to 1.04).

Massachusetts 2003

MA respondents were more likely to be white non‐Hispanic and more likely to be college graduates than the respondents from the rest of USA.

Age, sex, race and education were treated as confounders.

Unadjusted OR for current smoking in MA in 1999 vs 1990 was 0.78 (CI 0.66 to 0.92, P trend 0.01).
Adjusting for sex, age, race and education, OR was 0.83 (CI 0.70 to 0.99).
Compare with USA: 1990 unadjusted OR 0.95 (CI 0.92 to 0.99). Adjusted 1.01 (CI 0.97 to 1.05).

Adjusted prevalence OR for current smoking in MA showed annual decline in log odds of 1.3% pa, whereas USA showed average increase of 0.6% (P = 0.01).

McAlister 2004

Responders and non‐responders to follow‐up survey differed significantly by age, race and gender.
Also significant differences in gender and education between baseline and follow‐up samples.

Daily smoking rate "adjusted for age, gender and educational level". No OR reported.

McPhee 1995

Small but significant differences in mean age, educational level, English language proficiency, mean year of immigration, between SC and Houston and/or between pre‐ and post‐test samples. Larger differences in employment (8% unemployed in SC vs 6% in Houston pre‐test) and income (32% below poverty level in SC vs 26% in Houston post‐test).

Analyses controlled for site, time, (pre‐, post‐test), intervention term (site x time), age, education, English language proficiency, year of immigration, employment status, income.

Differences in prevalence between SC and Houston did not persist after controlling for sociodemographic characteristics. Strongest predictor of current non‐smoking was age 65+. Age 18 ‐ 24, immigration before 1977, college education and English fluency all predicted non‐smoking, but employment and income did not. Adjusted OR for intervention term was 1.1 (95% CI 0.9 to 1.4). Adjusted OR for recent quitting 1.1 (95% CI 0.7 to 1.7).

North Coast QFL 1983

Age and sex differences across towns and years.

To counter known confounding, an AGE*SEX*TOWN term was constructed for the regression model, and a TOWN*YEAR factor.

Sydney QFL 1986

Location of interview (Sydney vs Melbourne) was significant predictor of quitting (P < 0.05).

Sex, age, education, marital status, SES did not predict quitting or act as confounders.

No ORs reported.



Comparison 1 Mass media versus no mass media, Outcome 5 Baseline differences and possible confounding.

Prisma flow diagram of search results
Figuras y tablas -
Figure 1

Prisma flow diagram of search results

Risk of bias graph: review authors' judgements about each risk of bias item presented as percentages across all included studies.
Figuras y tablas -
Figure 2

Risk of bias graph: review authors' judgements about each risk of bias item presented as percentages across all included studies.

Study

Design

Intervention

Control

California TCP 2003

Nested cross‐sectional

1990‐1 75.1%1 (CA only)
CTS: 71.3 ‐ 99.4% (CA only)
NHIS 79.5 ‐ 87.8% across all USA
BRFSS: 77% ‐ 84%
CPS: 87.9%

NHIS, BRFSS and CPS rates apply.

CORIS 1997

Nested cross‐sectional

Nested cohort

69.5% at baseline, 67.5% at resurvey
Cohort identified retrospectively at 2nd survey, i.e. 4087 (56.3%) of those originally surveyed.
In men 46.4% of cohort smoked vs 50% of drop‐outs; in women 15.4% of cohort smoked vs 21.4% of drop‐outs.
Non‐cohort (i.e 1 survey only) were younger, less educated and higher smoking, but differences applied equally across all groups.

64% at baseline, 63% at resurvey


Cohort control not reported separately

Jenkins 1997

Nested cross‐sectional

Pretest: 84%, post‐test: 94%

Pretest: 82%, post‐test: 88%

Massachusetts 2003

Nested cross‐sectional

Median (1995): 60.4%, median (1999): 42.7%6

Median (1995): 68.4%, median (1999): 55.2%

McAlister 2004

Nested cross‐sectional


Nested cohort

Baseline survey 9407, 7m follow‐up 8974; response rate approximately 60%.
Cohort identified retrospectively at 2nd survey, i.e. 622 (58%) of the 1069 baseline daily smokers. Conditions not reported separately. 835 valid phone numbers at follow‐up, giving retention rate of 74.5%.

Not reported separately

Not reported separately

McPhee 1995

Nested cross‐sectional

Pretest: 81%, post‐test: 82%

Pretest: 85%, post‐test: 88%

McVey 2000

Nested cohort

Smokers: 6m: 73%, 18m: 70%
Ex‐smokers: 6m: 76%, 18m: 75%

Smokers: 6m: 74%, 18m: 66%
Ex‐smokers: 6m: 80%, 18m: 76%

Mogielnicki 1986

Randomized cohort

Mail: Yr2 follow‐up: 17%
Clinic: Yr2 follow‐up: 54%

Mail: Yr2 follow‐up: 15%
Clinic: Yr2 follow‐up: 52%

North Coast QFL 1983

Nested cross‐sectional


Nested cohort

Baseline: 71%
Yr 2: 73%
Yr 3: 73%

Not reported here

Baseline: 72%
Yr 2: 74%
Yr 3: 74%

Not reported here

Stanford 3 City 1977

Nested cross‐sectional
(cohort + non‐cohort)

Nested cohort

Gilroy 116 at baseline.
Non‐cohort had higher baseline % rates of smoking (74.2/62.4) and more cpd (20/13.8) than cohort.

73.2% at 2yr follow‐up

Tracy 115 at baseline.
Non‐cohort had higher baseline % rates of smoking (78/52.8) and more cpd (17.4/14) than cohort.

72.2% at 2yr follow up

Sydney QFL 1986

Nested cross‐sectional

Nested cohort

Sydney: 66%
Melbourne: 67.9%
Sydney: 76%
Melbourne: 73%

Australia‐wide: 60%

Figuras y tablas -
Analysis 1.1

Comparison 1 Mass media versus no mass media, Outcome 1 Response and retention rates.

Study

Intermediate measure

Process measures

California TCP 2003

Between 1996 and 1999 slight increases in agreements that smoking causes cancer (82.2% and 83.3%) and that passive smoking harms children's health (93.2% and 94%).

Between 1992 and 2002 increases in agreement that passive smoking causes cancer in nonsmokers (1992: 62.4%, 1996: 66.8%, 1999: 68.9%, 2002: 72.1%) and that smoking harms children's health (1992: 85.5%, 1996: 87.7%, 1999: 90.1%, 2002: 90.9%).

In 2008, 67.2% of daily smokers, 80.2% of non‐daily smokers, and 85.8% of nonsmokers perceived secondhand smoke (SHS) as a cause of cancer and 88.6% of daily smokers, 94.1% of non‐daily smokers and 94.7% of non‐smokers believed that SHS can harm the health of children and babies.

Between 1990 and 1991 increase in % of smokers thinking about quitting (from 38.6 to 42%; NS); health‐enhancing attitude score decreased (from 68.4% to 66.3%; SS).

Further increase in % of smokers thinking about quitting between 1994 and 2005, in the next 30 days (1994: 30.9, 2000:37.8%, 2005: 43.9%) and in the next 6 months (1994: 65.6%, 2000: 74%, 2005: 75%).

Between 1992 and 2002 increases in: % of indoor workers reporting a smokefree work site (1990: 35%, 1992: 46.3%, 1993: 65%, 1996: 90.5%, 1999: 93.4%, 2002: 95.4%, 2005: 94.5%, 2008: 96.4%).

In 2008, 95.2% of smokers and 96.6% of nonsmokers report having a completely smoke‐free workplace.

Between 1992 and 2008 increase in % of homes with smoking ban (1992: 48.1, 1993: 50.9%, 1996: 64.5%, 1999: 72.8%, 2002: 76.9%, 2005: 78.4%, 2008: 80.8%).

Between 1992 and 2008 increase in % of smokers with a total home ban on smoking (1992: 19.4 ± 1.6%, 2008: 59.3 ± 2.6%; increase of 204.9%).

Between 1990 and 2008 decrease in % of nonsmokers exposed to ETS at work (1990: 29%, 1993: 22.4%, 1996: 11.8%, 1999: 15.6%, 2002: 12.0%, 2005: 13.9%, 2008: 13.5%).

Increases in: support for further increase in tax on tobacco (1992: 74%, 1993: 78%; 2008: 78%), support for ban on tobacco advertising (1990: 52%, 1996: 65% [read from the graph]), support for ban on tobacco sponsorships (1998: 56%, 2000: 60%), % of adults preferring non‐smoking bars (1996: 75%, 2000: 81%), support for banning smoking outside the entrance of buildings (2002: 62.8%, 2008: 72.1%) and in restaurant outdoor patios (2002: 62.4%, 2008: 75%), outdoor public places (2002: 52.3%, 2008: 60.4%), Indian casinos (2002: 59.9%, 2008: 66.5%), inside cars when children are in them (2005: 92.3%, 2008: 95.2%).
Support for smoking restrictions in public places in at last 4 out of 6 venues significantly higher in CA than the rest of USA (1992/3: 58.5% vs 46.5%; 1995/6: 70.2% vs 51.5%; 1998/9: 75.8% vs 57.3%).
1998 survey showed that multicomponent exposure was significantly associated with reductions in smoking prevalence, increases in home smoking bans and reductions in perceived violations of workplace no‐smoking rules (P < 0.05).

Systematic monitoring of the campaign and independent evaluations of the programme.

Media weight: 1990/1: 50+ TV spots, 50 radio spots (69 radio stations), 20 outdoor ads (775 outdoor venues), 40 newspaper ads (130 newspapers); 1992/3: 40 spots (20 TV, 12 radio, 8 in outdoor locations), 44% focused on reducing ETS, 34% on countering pro‐tobacco influences, 20% smoking cessation and 2% on reducing youth access. 1990/4: local health departments conducted 10,000+ multi‐session programmes focusing on prevention (61%), cessation (37%) and ETS (2%). July 1995 ‐ Dec 1996: 19 ads (11 TV, 4 radio, 4 outdoor); 1997 ‐ 1998: 40 general audience ads for distribution (20 TV, 12 radio, 8 outdoor); July 1998 ‐ Aug 1999: 50 general audience anti‐tobacco ads (22 TV, 13 radio, 15 print ‐ mostly billboards).

Awareness/reach: 1990/1: unaided awareness in adult smokers 38.4% (similar for most ethnic groups), unaided and aided awareness almost 78.7%. In 1992 > 50% of the adult respondents and > 80% of the adolescent respondents recalled having seen or heard anti‐tobacco messages. In 1992 recall of anti‐tobacco campaign highest among the youngest age group (18 ‐ 24: M 75%, W 70%, 25 ‐ 44: M 65%, W 60%, 45+: M 58%, W 50% [read from the graph]).

In 1996, 67% of adults reported seeing antismoking message on TV, 44% hearing on radio, 41% seeing antismoking billboard. In 1998 80% of adults were exposed to tobacco control programme through two or more components.
In 1999 vs 1996 more respondents reported being exposed to lots of tobacco messages on TV, radio, billboards. 91% of adults reported seeing at least one anti‐tobacco ad in 1996, 1998 and 2000. Between 1996 and 2002 the % of smokers reporting seeing a lot of anti‐smoking ads on TV in last month increased, but then decreased between 2002 and 2008 (18 ‐ 24 yrs: 1996: 16.1%, 1999: 29.9%, 2002: 37.9%, 2005: 21.2%, 2008: 20.8%; 25 ‐ 40 yrs: 1996: 13%, 1999: 20.1%, 2002: 23.2%, 2005: 15.7%, 2008: 10.8%; 41+ yrs: 1996: 10.3%, 1999: 14.9%, 2002: 13.6%, 2005: 8.9%, 2008: 5.6%).

In 2011 in CSC 28% to 61% recalled anti‐tobacco commercials.

Dose‐response: 1990 ‐ 1996 smokers reporting recall of media spots more likely to make a quit attempt in the last year than those who did not. The more channels recalled between 1990 and 1996 the higher increase in cessation was observed. 1996 ‐ 2000 in counties with the highest multicomponent exposure rates, there were greatest reductions in adult smoking prevalence, workplace no‐smoking policy violations and the largest increases in home smoking bans.

2011 recall of any of anti‐tobacco spots was not related to quitting 1 yr later, recall of one specific spot ("Stages") was associated with higher quit attempts rate and quitting for more than 1 month, after 1 yr.

Maintenance: The campaign was established as Proposition 99, as a constitutional amendment and mandated the conduct of a mass media campaign. The campaign has been running since 1990.

Intervention costs: CA TCP mass media campaign funding/ total expenditures targeted at tobacco use in millions of US dollars: 1989/90: 14.3/85.8; 1990/1: 14.3/132.0; 1991/2: 16.0/55.9; 1992/3: 15.4/84.0; 1993/4: 12.9/61.1; 1994/5: 12.2/56.3; 1995/6: 6.6/41.8; 1999‐2000: 19.6/60.8; 2000/1: 45.3/88.2; 2001/2: 45.3/108.1; 2002/3 21.1/61.7; 2003/4: 16.8/61.8; 2004/5: 15.7/56.8; 2005/6: 15.7/58.5; 2006/7: 20/65; 2007/8: 15.7/56.6; 2008/9: 15.7/56.5. Total expenditures for mass media campaign and for the programme between 1989 and 1996 were in millions of US dollars: 91.7 and 516.9. Average annual expenditure was USD 3.35 per capita per year, but from mid‐1993 to mid‐1996 when funding was decreased annual expenditure was USD 2.08 per capita per year.
The CTCP media funding was USD 1.33 per capita in 2001/2 and it decreased from 2002/3 (USD 0.6 per capita) to 2007/8 and 2008/9 (USD 0.43 per capita).

The analysis on the basis of per capita consumption of cigarettes and average per capita media expenditures gave estimates of a fall of 3.9 packs per capita per year for each per capita USD spent on the media campaign.

CORIS 1997

At baseline, knowledge scores higher in Swellenden than in Riversdale (both cross‐sectional and cohort surveys; no statistical comparisons given); at 4 yrs more increase in women in Swellenden (both cross‐sectional and cohort surveys; in cohort survey net change statistically significant); at 12 yrs increase in both communities and no difference.
Attitudes were assessed at baseline, but the results were not reported.

Participation and reach of activities recorded in mass media and community intervention town (not included in this analysis).

Media weight: Limited data. 1st yr: 6 different billboards, 6 posters, 8 mailings, frequent news items, health messages on electricity accounts, 1 special supplement in local newspaper. 2nd and 3rd yr frequency of billboards, posters and mailings about half of the initial rate, but news items frequent and annual special supplement in a local newspaper was added.

Awareness/reach: no evidence found.

Dose‐response: no evidence found.

Maintenance: after 4 yrs of active intervention a maintenance programme was run by community.

Intervention costs: per capita costs given (USD 5 over 4 yrs in intervention community).

Jenkins 1997

At baseline no differences in motivation to quit and self efficacy (SF/Houston: 29%/23%; 29%/25%). At follow‐up significant increase in motivation in both communities, but no difference (SF/Houston: 45%/ 44%), no significant change in self efficacy (SF/Houston: 33%/26%).

A 15‐month uncontrolled pilot anti‐tobacco campaign.

Media weight: newspaper articles ‐ 465,000 print media exposures; 15,000+ copies of brochure, 4600 copies of self‐help quit kit distributed; billboard and newspaper ads ‐ 2.8 million exposures, paid TV ads ‐ 100 mins of air time. Short anti‐tobacco presentations at 25 community events, 68 Vietnamese physicians took part in smoking cessation course and 400 Vietnamese students participated in anti‐tobacco activities.

Awareness/reach: Participants were asked if they had ever read, seen or attended any of five elements of media intervention. Recall of each was significantly greater in intervention than in control community (P < 0.05), except for newspaper articles. In both communities smokers were more likely than non‐smokers to recall the elements of the campaign.

Dose‐response: no evidence found.

Maintenance: no evidence found.

Intervention costs: no evidence found.

Massachusetts 2003

1993 ‐ 2000: the majority of non‐smokers believed that second‐hand smoke (SHS) can harm children (96%) and can cause lung cancer (89%); increase in % of smokers believing that environmental tobacco smoke (ETS) can harm their children (1993: 78%, 1999: 91%, 2000: 93%) and can cause cancer (1993: 58%, 1999: 72%, 2000: 74%);
1993 ‐ 1995: decrease in: % believing that ETS is harmful (1993: 90%, 1995: 84%), support for ban on support of sports and cultural events by tobacco companies (1993: 59%, 1995: 53%, P < 0.05); increase in: support for further tobacco tax increase with funds devoted to tobacco control (1993: 78%, 1995: 81%), support for ban on vending machines (1993: 54%, 1995: 64%, P < 0.05).
1993 ‐ 2000: Increase of % of workers reporting a smokefree work site (1993: 53%, 1995: 65%, P < 0.05; 2000: 75%, 2001: 82.2%), % of homes with smoking ban (1993: 41%, 1995: 51%, P < 0.05; 2001: 71.2%).

2007 ‐ 94.5% believe ETS is harmful.

Decrease in: ETS exposure at work (1993: 44%, 2000: 29%), at home (1993: 28%, 2000: 18%) and in restaurants (1995: 64%, 2000: 39%, 2002: 37%), mean hours of ETS exposure during prior week at work (1993: 4.2, 1995: 2.3, P < 0.05, 1997: 2.2, 2001: 1.4), and at home (1993: 4.7, 2000: 3.3). Increase in: % of population covered by smoking restrictions in restaurants (1995: 26%, 2000: 69%, 2001: 78%); % of population living in a town with some form of smoking restrictions in public places (1993: 22%, 2000: 78%, 2001: 85%); % of smokers reporting that fewer than half their friends and relatives smoke (1993: 40.8%, 2000: 46.9%; P for trend = 0.03); most non‐smokers reported so both in 1993 and 2000 (1993: 90.1, 2000: 89.9).
1995‐2000 significant increase in: support for complete ban on smoking in restaurants (1995: 42%, 2000: 51%), public buildings (1995: 46%, 2000: 58%), at indoor sporting events (1995: 52%, 2000: 67%), in shopping malls (1995: 53%, 2000: 67%), but not at outdoor sporting event (1995: 15%, 2000: 19%).
1993 ‐ 1999: decline in the proportion of people who allow visitors to smoke in their homes (1993: 57%, 1999: 37%, P < 0.01), increase in the proportion of people who asked a colleague not to smoke increased (1993: 29%, 1999: 36%).

2008 ‐ 80.7% voluntary tobacco‐free homes (vs 81.5% in 2007).

2009‐ 57% of schools with comprehensive smoking policies

2014 ‐ based BRFSS exposure for ETS decreased to 10% (vs 32% in 2002); only 16.4% of houses allow smoking. and exposure to ETS >1 hr/last wk decreased to 11% (vs 32% in 2002; 14.5% in 2008).

Increase in support smoking ban in all personal spaces ‐ of people living in smoke‐free houses 87% and of those living in non smoke‐free houses 52%.

Messages were developed through formative research including focus groups. Systematic evaluation of the programme.

Media weight: Oct 1993 ‐ Dec 1996: 66 spots aired; 35 TV ads (14,901 total gross rating points [GRP]), 27 radio spots (13,644 total GRP) on smoking cessation in adults; 38 TV (17,800 total GRP) and 22 radio (13,950 total GRP) ads targeted youth; 6 TV (17,727 total GRP) and 2 radio spots (6,308 total GRP) on ETS.

Awareness/reach: 88% respondents saw any TV ads, among quitters ‐ 97%. On average 4.48 ads recognised. In 1995, 65% of the adult population were aware of the statewide anti‐tobacco campaign, and 76% recognised the tag line of the media campaign "It's time we made smoking history". Around 89% of adult smokers reported having heard, read or seen information about quitting smoking within the past year.

Dose‐response: no evidence found.

Maintenance: The campaign is ongoing.

Intervention costs: On average USD13 million per year spent on anti‐tobacco advertising. About 48% expenditure (up to Dec 1996) spent on TV time (much prime time viewing hours). MTCP activities expenditures: 1st yr ‐ USD 43 million, 3rd yr ‐ USD 35 million. Mean per capita cost for media campaign was USD 2.42.
The analysis on the basis of per capita consumption of cigarettes and average per capita media expenditures gave estimates of a fall of 0.5 packs per capita per yr for each per capita dollar spent on the media campaign.

McAlister 2004

Knowledge, attitudes, beliefs, intentions, brand preferences were assessed at baseline. Process of change variables were measured by rating by responders how much they agree or disagree (1 = strongly disagree, 5 = strongly agree) with the statements regarding attitudes to smoking and occurrence of behaviours associated with smoking cessation. At follow‐up, mean level of agreement was significantly higher among those who reported daily smoking cessation compared to those who maintained daily smoking with regard to having support for quitting and behaviours associated with smoking cessation and dealing with tension. Participants still smoking at follow‐up were significantly more likely to agree with the statements about attitudes and ability to quit . There are no follow‐up results in comparison with control group.

In message development community forums, focus groups and pre‐testing were used. No information regarding ongoing monitoring of media campaign was provided.

Media weight: no evidence found.

Awareness/reach: At follow‐up correlations between media exposures and process variables were calculated for all groups. No separate result for mass media were given.

Dose‐response: Not given for mass media campaigns without community programmes separately.

Maintenance: no evidence found.

Intervention costs: per capita media spending reported, high level and low level media represented about USD 0.25 versus USD 1.00 per capita for media spending during the year of the campaign.

McPhee 1995

Motivation to quit and self efficacy significantly more common in Santa Clara (SC) at baseline (SC/Houston: 36%/23%; 34%/ 25%). At follow‐up motivation to quit increased significantly in both communities, but did not differ (SC/Houston: 44%/ 44%), self efficacy did not change significantly and did not differ at follow‐up (SC/Houston: 32%/ 26%). 'Any household member smokes' responses similar at baseline (SC/Houston: 31%/29%) fell significantly in both communities (SC/Houston: 26%/24%). 'No friend smokes' responses not different at baseline (SC/Houston: 4%/3%) increased significantly in both communities (SC/Houston: 15%/13%). 'All or most friends smoke' responses similar at baseline (SC/Houston: 43%/46%) increased in Houston (SC/H: 44%/51%). Never‐smokers or former smokers advising family or friends to quit smoking increased from baseline in both communities (SC vs Houston: from 51% to 62% (P < 0.05) vs from 34% to 66% (P < 0.05)). Current or former smokers being advised to quit by family or friends increased from baseline in both communities (SC vs Houston: from 53% to 63% (P < 0.05) vs from 51% to 62% (P< 0.05)).

Media weight: newspaper articles ‐ 562,000 print media exposures, videotape copies distributed to 60+ Vietnamese doctors' offices and 20 community agencies in SC; nearly 42,000 brochures at 500+ location in SC, nearly 6000 self‐help quit kit copies at 250+ locations; 1140 signs with adhesive backings saying "Please do not smoke" in Vietnamese distributed; 50 copies of 3 billboards posted each month and printed in newspapers and magazines (8,000,000 print media exposures). Paid TV ads ‐13,000 secs of air time; short anti‐tobacco presentations at 30 community events. CME courses on smoking cessation ‐ 68 Vietnamese physicians.

Awareness/reach: Participants were asked if they had ever read article or seen advertisement in Vietnamese language newspaper or seen television programme or billboard in Vietnamese or heard a speech at a Vietnamese community meeting. Significantly more respondents from SC reported exposure to anti‐smoking activities in Vietnamese language (except for newspaper articles and public speaking). Smokers and non‐smokers in SC recalled significantly more intervention elements (SC vs Houston: out of a possible 5: 3.0 vs 1.6, P < 0.01 and 2.8 vs 1.4, P < 0.01). In SC significantly more physicians reported using antismoking brochures in Vietnamese, providing self‐help quit kits and referring patients to smoking cessation programmes (SC vs Houston: 86.4% vs 32.5%, P = 0.001; 66.7% vs 43.2%, P = 0.03; 35.9% vs 13.5%, P = 0.02). Quality of health education materials assessed: at follow‐up 96.4% of physicians reported they were helpful.

Dose‐response: no evidence found.

Maintenance: no evidence found.

Intervention costs: no evidence found.

McVey 2000

Smoking and health‐related attitudes assessed at baseline, no follow‐up data given.

Qualitative pilot research studies using focus groups and in‐depth interviews with smokers and ex‐smokers were conducted before and during the campaign.

Media weight: It was estimated that over the course of the campaign in the double‐weight regions each viewer could see at least 20 screenings, and in single‐weight regions about 15.

Awareness/reach: no evidence found.

Dose‐response: nearly 20,000 calls to the quit line during the campaign. No evidence of an effect of intensity of ads measured at 6 month follow‐up found.

Maintenance: After study completion the TV advertisements were shown nationally in all TV regions in England.

Intervention costs: evidence not found.

Mogielnicki 1986

Attitudes and beliefs regarding cigarette smoking were assessed on enrolment on a 5‐point rating scale (1 = disagree strongly, 5 = agree strongly). No separate results for 2nd yr follow‐up (mass media) given.

Surveys, interviews, copy tests among patients fulfilling study inclusion criteria but not included in the final study group.

Media weight: One 60‐sec main commercial and 2 x 30‐sec variants used. A series of 3‐week 'flights'; main spot on TV was broadcast 40 times, and variants 106 times, radio spot ‐ 90 times. Each flight lasted 2 ‐ 3 weeks.

Awareness/reach: Participants were asked about the recall of advertising campaign, clinic participants in media group (White River Jct): 61% did and 39% did not recall the advertising campaign at 6 month follow‐up.

Dose‐response: Among those who recalled, 43.5% were abstinent at 6 months, while among those who did not 26.7% abstinent.

Maintenance: no evidence found.

Intervention costs: total costs of commercials development and production: USD 7480, broadcast time cost: USD15,150.

North Coast QFL 1983

Attitudes to smoking and knowledge of the effects of smoking assessed in a questionnaire (6 questions each). No significant differences were found. Influences in decision to quit smoking reported ‐ the most common: health concerns (Coffs Harbour/Tamworth: 53%/ 59%), the least common: social pressure and media advertisement (Coffs Harbour/Tamworth: 1%/1%, 2%/1%), other were friends/family (Coffs Harbour/Tamworth: 15%/ 13%) and doctor's advice (Coffs Harbour/Tamworth: 7%/5%). Technique of quitting: most common ‐ 'cold turkey' (87% in both towns), quitting with help was rare (Coffs Harbour: self‐help kits 2%, group programme 1%, hypnosis 2%).

Focus groups and spot surveys were used to assess the effectiveness of the intervention techniques used.

Media weight: Information collected only on community programme exposure.

Awareness/reach: no evidence found.

Dose‐response: no evidence found.

Maintenance: no evidence found.

Intervention costs: no evidence found.

Stanford 3 City 1977

25‐item behavioural interview on participants' knowledge about risk factors (diet, weight, exercise and smoking ‐ 3 items). Significant increases in knowledge scores in intervention cities compared to control city after 2 yrs.

Mass media campaign was monitored and revised; instructional content, development, pretesting, application and reformulation co‐ordinated by study staff.

Media weight: limited data; TV programming ‐ 3 hours, 50 TV spots, 100 radio spots, several hours of radio programming, weekly newspaper columns, no details about newspaper and ads stories, billboards, printed materials sent via direct mail to participants, posters in buses, stores and work sites.

Awareness/reach: no evidence found.

Dose‐response: increases in risk factor knowledge related to increases in intervention intensity, even in Watsonville‐reconstituted group (mass media only, but a group in community received intensive face‐to‐face counselling) the gains were bigger than in Gilroy (mass media only group) and in both intervention communities the gains were bigger than in Tracy.

Maintenance: After intensive 2 yrs, 1 yr reduced level campaign.

Intervention costs: no evidence found.

Sydney QFL 1986

Additional random samples of Sydney and Melbourne smoker populations surveyed in 1983 and 1986 (Sydney/Melbourne: 1983: 271/217; 1986: 557/ 550). Health beliefs and social influences increased between 1983 and 1986 (Sydney/Melbourne: 40+ years old: males 10% to 29%/21% to 29%; females 12% to 27%/22% to 32%; < 40: males 34% to 53%/30% to 43%, females 30% to 44%/31% to 41%).
Information‐seeking behaviour was measured as the number of calls to quitline and the number of enrolments to quit centre ‐ see dose‐response.

Formative research on messages' effectiveness among target audience.

Media weight: 1983: 389 primetime ads spots (3 commercials used) ‐ the intensity alternated in 2‐wk phases: between heavy and nothing for 1st 3 months, follow‐up ad campaign of half the intensity after 5 months. After 1st year spots during prime or fringe time for approx 4 wks at the start of the campaign yr ‐ each yr nearly 40 spots/wk.

Awareness/reach: During 1st yr each month 750 people in Sydney and 200 in Melbourne interviewed on recall of campaign messages and response to the question on likelihood of giving up smoking in the next 12 months. The recall rates for the commercial most frequently shown were 87% in smokers, 82% in non‐smokers and 85% in ex‐smokers, the second in the frequency: 73%, 69% and 67% respectively. The third commercial recall rates were: smokers: 39%, non‐smokers: 31%, ex‐smokers: 33%. During campaign more smokers in Sydney indicated they were likely to quit smoking, especially after 4 months (difference: 18.6%); the difference disappeared by the end of the campaign. Long‐term assessment: TV ads recall measured each yr in separate random samples of the population of at least 1000 persons higher in Sydney (Sydney/Melbourne: 1984: males: 72%/70%, females: 80%/67%; 1986: males: 92%/68, females: 94%/67%).

Dose‐response: number of calls to quitline was measured as a direct relationship to the number of TV spots aired (presented on the graph); peak response ‐ 11,000 calls in wk 8; 50,000+ calls to the quitline in the 1st 3 months. 'Quit centre' stop smoking programmes enrolments: almost 3000 in 1983 (previous yr 500); in wk 8 with peak quitline calls number ‐ 352. 19,196 quit kits sold.

Maintenance: Since 1984 the campaigns continued in both Sydney and Melbourne till 1986, ads shown on primetime TV for 6 ‐ 8 weeks during winter months.

Intervention costs: The budget for the campaign given (AUD 620,000), most of which was used for purchase of newspaper space and radio and TV time (AUD 500,000).

Figuras y tablas -
Analysis 1.2

Comparison 1 Mass media versus no mass media, Outcome 2 Intermediate measures.

Study

Study population

Smoking prevalence

Tobacco consumption

Other outcomes

California TCP 2003

Selection: population‐based surveys conducted nationally and in California (CA):

National Health Interview Surveys (NHIS) 1978 ‐ 1994: household survey of a stratified, multistage, probability sample of the US population administered to all adults 18+ yrs;

Behavioral Risk Factor Surveillance System (BRFSS) 1984 ‐ 1994: state surveillance of behavioural risk factors among adults, data collected through random digit dial telephone interviews (with multistage cluster sampling method); with California Adult Tobacco Survey from 1994, data up to 2008.

Current Population Surveys (CPS) 1985, 1989, 1992/3 ‐ 1996: national survey of the US household population 15+ yrs, interviews in person; data up to 2008

California Tobacco Survey 1990‐2008

N of participants:
NHIS 1978 ‐ 1994,

response rate 79.5% to 87.8%; CA, rest of US: from 1112 to 5747, from 9205 to 39,203;

BRFSS 1984 ‐ 1994, response rate from 77% to 84%: from 1081 to 3959; with CATS from 1994 over 10,000 in CA and 80,000 to 400,000 in the rest of US.

CPS 1985 ‐ 1996: response rate from 86% to 89%: CA, rest of US: from 4076 to 8272, from 70,164 to 97,856; CPS 1992/3 to 2006/7, CA, rest of US: from 16,000 to 24,000, from 210,000 to 290,000; CTS, CA only between 44,000 and 93,000 respondents per survey.
Comparability of demographic data at baseline: not analysed.

Final data (From 1990 programme start to 2008):

All surveys (CTS, CPS and BRFSS pooled analysis years 1990 ‐ 2008, decline of smoking prevalence:

CA 0.32% points per year, rest of US 0.24% points per year.

2008 smoking prevalence: CA 13.1%,

rest of US: 19%

Interim reported data:

According to the analysis of the results of NHIS,

adult smoking prevalence decreased more rapidly from 1985 to 1990 than from 1978 to 1985 in CA and in the rest of USA . Increase in rate of decline between 2 periods in CA was 0.62% points per year (95% CI ‐1.27 to ‐0.03), and in rest of USA 0.43 (‐0.70 to ‐0.16).

Adult smoking prevalence decreased less rapidly from 1990 to 1994 than from 1985 to 1990 in CA (0.39% points per year) and in the rest of USA (0.05%, non‐sig different from zero).

Between 1978 and 1994 adult smoking prevalence was 2 ‐ 5 percentage points lower in CA than in the rest of USA, but the estimated rate of decline in smoking prevalence was no‐sig different from that of the rest of USA during any of the 3 time periods.
Combined estimated rate of decline before CA TCP began was similar in CA to the rest of USA. Rates of decline were not statistically different but estimated prevalence in CA was lower than in the rest of USA. The rate of decline increased significantly in CA at the early period of the programme but not in the rest of USA. In the late phase of the programme the rate of decline in CA and in the rest of USA was non‐sig different from zero and in both cases it was less than in the previous period.
BRFSS trends similar, but consistently around 2% lower.
CPS, CTS and CATS trends similar to BFRSS (all telephone surveys, while NHIS is face‐to‐face).

NHIS only: rate of change in smoking prevalence 1978 ‐ 1985, CA vs rest of US:
Adults aged 18+ yrs:

‐0.6 (95% CI ‐0.79 to ‐0.40) vs ‐0.5 (‐0.67 to ‐0.33);
1985 ‐ 1990: ‐1.22* (‐1.51 to ‐0.93) vs ‐0.93* (‐1.13 to ‐0.73);
1990 ‐ 1994: ‐0.39* (‐0.76 to ‐0.03) vs ‐0.05* (‐0.34 to +0.24);
Adults aged 25+ yrs:
1978 ‐ 1985: ‐0.61(‐0.83 to ‐0.39) vs ‐0.43 (‐0.63 to ‐0.24); 1985 ‐ 1990: ‐1.11* (‐1.37 to ‐0.84) vs ‐0.88* (‐1.10 to ‐0.66);
1990 ‐ 94: ‐0.61 (‐0.99 to ‐0.23) vs ‐0.20* (‐0.52 to +0.12); *significant difference (P < 0.05) between the estimated rate of change for that period and that for the previous period.

All surveys combined analysis: CA, rest of US rate of decline % (SE) ‐ smoking prevalence %:
pre‐1989 (preprogramme): ‐0.74 (0.12) ‐ 23.3, ‐0.77 (0.09) ‐ 26.2;
1989 ‐ 1993 (early programme): ‐1.06 (0.17) ‐ 18.0, ‐0.57 (0.14) ‐ 23.3;
1994 ‐ 1996 (late programme): 0.01 (0.21) ‐ 18.0, ‐0.28 (0.26) ‐ 22.4; P < 0.05 CA vs rest of US, P < 0.001, change from previous period.

Final data (1970 ‐ 2008), per capita taxable sales:

1970 per capita sales similar in CA and rest of US.

In 1989, CA sales 26.1% lower than sales for the rest of the US (108.8 versus 147.2 packs per year).

Faster rate of decline in CA vs rest of the US.

2002 CA vs rest of the US lower sales (48 versus 101 packs per capita per year).

From 2002 slower rate of decline in CA vs rest of the US (from 44.6 to 40.4 packs per year, a decline of 9.4%), but still lower sales than in the rest of the US. 2008: 40 versus 77 packs per capita per year, or 3.37 versus 6.42 packs per capita per month).

1989/90 to 2006/7, per capita consumption

greater decline in CA vs rest of the US: 60.8% vs 41.0%

(40 vs 92 packs per person per year).

Per capita consumption based on aggregated sales data declined faster in CA in the early period of the campaign than before it started, and the decline was significantly greater than in the rest of USA. During late programme the decline slowed but remained greater than in the rest of USA.
Rate of decline in per capita consumption based on aggregated sales data CA/rest of US pack (SE), packs/month:
Baseline 1989: ‐0.42* (0.03), 9.7/ ‐0.36 (0.02), 12.5;
Early programme 1989 ‐ 1993: ‐0.64#^ (0.03), 6.5/ ‐0.42 (0.03), 10.4;
Late programme 1994 ‐ 1996: ‐0.17 *# (0.07), 6.0/ 0.04 # (0.06), 10.5.

In another analysis presenting the results up to 1999 the rate of decline of tobacco consumption (tobacco consumption pack/month), CA/rest of US: Baseline 1/1983 ‐ 12/1988: ‐0.46 (9.5)/ ‐0.35 (12.4);
Early programme 1/1989 ‐ 12/1993: ‐0.58 (6.6)/ ‐0.4 (10.3);
Mid‐programme 1/1994‐10/1998: ‐0.16 (5.8)/ ‐0.07 (10);
Recent programme 10/1998‐12/1999: ‐1.56 (4.1)/ ‐0.78 (9.1).

In analysis presenting the results for tobacco consumption up to 2002, CA,/rest of US packs/month
1988: 9.8/12.5;
2002: 3.9/7.5.

* CA vs rest of US P < 0.01.
^CA vs rest of US P < 0.001.
#change from previous period P < 0.001.

Quit ratio, defined as % of ever‐smokers (current and former) who were former smokers in a given year, accelerated non‐sig in CA and in the rest of US between 1985 and 1990. 1990 to 1994, rate of increase in quit ratio decreased, with no significant changes in CA and in the rest of US compared to the period of 1985 to 1990.

Quit ratio: NHIS only, 1978 ‐ 1994, CA, rest of US, adults 18+years: 1978 ‐ 1985: 0.73 (0.22 to 1.24), 0.73 (0.40 to 1.05);
1985 ‐ 1990: 1.36 (0.74 to 1.97), 1.04 (0.62 to 1.46);
1990 ‐ 94: 0.18 (‐0.8 to 1.15), 0.15 (‐0.47 to 0.77);

Quit attempts: mean number of quit attempts in last yr decreased between 1990 and 1992 from 48.9% to 38.1%, then increased in 1996 and 1999 to 56.0% and 61.5%.
% of smokers attempting quitting and abstinent for 3m at time of survey increased (1990:15.3%, 1992:18.6%, 1993: 20.2%).
% of smokers with quit attempt in last yr lasting 7 days+ increased between 1990 and 2002 (1990: 29.2%, 1992: 25.1%, 1996: 36.1, 1999: 41.4%, 2002: 40.5%).

% of smokers who are currently quit for 3+ months did not change significantly (1990: 8.5 ± 1.0%, 1999: 5.4 ± 0.5%, 2008: 8.0 ± 2.5%)

% of recent smokers currently in a quit attempt of 6+ months did not change significantly (1990: 5.6 ± 0.7%, 1999: 4.2 ± 0.5%, 2008: 6.4 ± 2.5%)

CORIS 1997

Selection: Total population included in baseline and 4‐yr follow‐up surveys (15 ‐ 64 yrs at baseline and 15 ‐ 68 at 4‐yr follow‐up).
At 12‐yr follow‐up a random sample of white participants aged 15 ‐ 64 was selected ‐ about 50 people per sex‐ and age‐specific decile were selected, excluding people living < 2 yrs in the community.
High‐risk cohort was identified within cohort sample ‐ people who had at least 1 of: high cholesterol (> 20th percentile for age), high blood pressure (systolic > 160 or diastolic > 95 mm Hg or antihypertensive drug use), regular smokers.

N of participants:
Total population Swellendam [Intervention]/Riversdale [Control] [M: men; F: women]. Baseline 1979 (% 1980 census): M: 1224 (65)/ 1082 (60); F: 1396 (74)/ 1208 (68); Follow‐up: 1983 (% of 1980 census): M: 1171 (65)/ 1109 (62); F: 1323 (70)/ 1150 (64); 12‐yr follow‐up 1991: random sample Swellendam/Riversdale: M: 273/269; F: 267/274; response rate not reported.
Cohort Swellendam/Riversdale (% of total population): M: 710 (58)/595 (55); F: 821 (59)/710 (59);
High‐risk cohort, Swellendam/ Riversdale (% of cohort): M: 388 (55)/320 (53); F: 234 (29)/224 (32). Comparability of demographic data at baseline: Authors state that age and sex distributions were very similar.

In total population % of smokers was lower in men and women in intervention compared to control city (non‐sig difference).
Baseline prevalence (1979) and (% change) at 4 yrs (1983): Swellendam/ Riversdale: M: 49.5 (‐9.1)/45.7 (‐7.6); F: 17.6 (‐3.6)/16.1 (‐0.5).
At 4 yr follow‐up in the intervention cohort compared to control cohort the % of smokers decreased (non‐sig) in women but not in men.
Cohort: baseline Swellendam/ Riversdale, and (net change): M: 46.5/44.4 (+0.9); F: 14.5 /14.4 (‐3.0);
At 4 yr follow‐up in the high‐risk cohort in the intervention group compared to control group the % of smokers decreased (non‐sig) in women but not in men.
High‐risk cohort baseline Swellendam/ Riversdale (net change): M: 85.3/82.5 (+1.3), F: 50.9/45.5 (‐7.4);
At 12 yr (1991) follow‐up smoking prevalence was lower in intervention compared to control city.
1991 random sample smoking prevalence Swellendam/ Riversdale: M: 25.3*/34.2; F: 12.4/12.8; *significantly lower than Riversdale.

In total population tobacco consumption was lower in men and women in intervention compared to control city (non‐sig difference).
Baseline per capita consumption of tobacco grams/day (1979) and % change at 4 years (1983): Swellendam/Riversdale: M: 11.2/‐2.6, 8.8/‐1.8; F: 2.6/‐0.4, 2.3/0.1;
At 4 yr follow‐up in the intervention cohort compared to control cohort tobacco consumption decreased in men and women (non‐sig difference).
Cohort baseline Swellendam/ Riversdale and (net change): M: 11.3/9.2 (‐0.4); F: 2.1/2.3 (‐0.2).
At 4 yr follow‐up in the high‐risk cohort in the intervention group compared to control group tobacco consumption decreased in men and women (non‐sig difference). High‐risk cohort baseline Swellendam/Riversdale (net change): M: 20.5/17.3 (‐0.8), F: 7.5/7.3 (‐0.4).
At 12 yr (1991) follow‐up tobacco consumption was lower in intervention compared to control city. 1991 random sample per capita consumption Swllendam/Riversdale: M: 4.8/5.8, W: 1.9/1.8.

In women quit rates were significantly lower in intervention compared to control city.
Quit rates: Swellendam/Riversdale: after 4 yrs: % of all smokers: M: 16.9/20.1; F: 28.3*/15.5; Light smokers: M: 26.5/32.6**; F: 44.4**/28.6**; Heavy smokers: M: 15.8/17.7; F: 23.7/10.7.

*significant difference compared to control; **significant compared with heavy smokers.

Jenkins 1997

Selection: Phone surveys of randomly selected Vietnamese men, with numbers chosen randomly from 23 most common Vietnamese surnames listed in area phone books. After enumerating all men aged 18+ living in the household and speaking Vietnamese, subjects for interview were selected according to random age rankings in the household.

N of participants:
San Francisco (SF)/Houston: Baseline (% response rate):
1990: 1133 (84)/1581 (82); Follow‐up (response rate %): 1992: 1202 (94)/1209 (88). Comparability of demographic data at baseline: significant differences in educational level, English language proficiency, income, unemployment and mean immigration year.

Smoking prevalence did not differ significantly at baseline. Post‐intervention, smoking prevalence fell significantly (P = 0.004) in SF and increased in Houston. Current smokers % among Vietnamese men: SF/Houston:
Baseline (1990): 36.1/39.6;
Follow‐up (1992): 33.9/40.9. Significant intervention effect (P ⋝ 0.01). The odds of being a smoker at follow‐up were significantly lower in intervention than in control community; OR: 0.82, 95% CI: 0.68 to 0.99). The odds of being a smoker after the intervention were lower for following characteristics: age groups 18 ‐ 24, 45+; at least a college education; good or fluent English; being a student. The odds of being a smoker were higher for unemployed and more recent immigrants.

Significant difference in cpd at baseline (Houston higher), significant decrease in Houston, but not in SF. At follow‐up mean cpd was significantly higher in Houston.
Mean cpd among smokers: SF/Houston, Baseline 1990: 11.1/13.2;
Follow‐up 1992: 10.3/11.9.

Significant increase in quit rate in SF compared to Houston (P = 0.017).
% Quit rates in past 2 yrs: SF/Houston:
Baseline 1990: 7.2/5.8;
Follow‐up 1992: 10.2/7.4; significant increase in SF, but not in Houston.
The odds of being a quitter in 1992 were significantly higher in intervention compared to control community (OR: 1.65, 95% CI: 1.27 to 2.15). Predictors for quitting included: being a student (OR: 2.19, 95% CI 1.45 to 3.33); more recent year of immigration (OR: 1.03, 95% CI 1.0 to 1.05); each additional year of age (OR: 1.03, 95% CI 1.02 to 1.04); at least high school education (OR: 1.33, 95% CI 1.04 to 1.7).
Mean number of quit attempts was similar in both communities at baseline and increased significantly in Houston but not in SF. Mean number of quit attempts: SF/Houston:
Baseline 1990: 1.4/1.1
Follow‐up 1992: 2.1/2.5.
At baseline % reporting any quit attempt in the past was significantly higher in SF. At follow‐up it increased significantly in both communities, but did not differ. Percentage reporting any quit attempt in the past: SF/Houston:
Baseline 1990: 61%/49%
Follow‐up 1992: 73%/77%.

Massachusetts 2003

Selection: population‐based survey in Massachusetts (MA) and in the rest of USA (excluding CA) ‐ Behavioral Risk Factor Surveillance System (BRFSS): annual, state‐based, standardised, random digit dialled telephone survey of non‐institutionalised US adults aged 18+; 42 states including MA and CA participated consistently in BRFSS between 1989 and 1998; MA tobacco surveys conducted since 1993‐4 (baseline) in adults, and every month since March 1995, estimates of adult smoking prevalence are derived from household screening interviews with an adult informant who reported on smoking status for all adult members of the household.

N of participants:
1990/9: 22,309 responses from MA, 946,241 from the rest of USA;
BRFSS 1989/98 sample sizes in MA from 1221 to 4944; in rest of USA (excluding CA) from 63,255 to 113,214.
MA surveys 1994/98 respondents: from 5736 to 21,909. Response rates for BRFSS were 77% to 84% between 1984 and 1994; Between 1995 and 1999 response rates for USA fell from 68.4% to 55.2% and in MA from 60.4% to 42.7%.
Comparability of demographic data at baseline: MA respondents were more likely to be white non‐Hispanic and more likely to be college graduates than the respondents from the rest of USA.

According to the analysis based on BRFSS 1989 to 1998 and MA Tobacco Surveys 1993 to 1999 the slope of smoking prevalence after 1992 was significantly different from zero and from the slope for the rest of USA. Based only on BRFSS 1990 ‐ 1999 controlling for age, sex, race, and education there was a greater decline in current smoking between 1990 and 1999 among MA men than among MA women, and the decline was greater in MA than in the rest of the USA for men and for both sexes combined.

Based on BRFSS 1989 to 1998 and MA Tobacco Surveys 1993 to 1999: for MA the slope of smoking prevalence after 1992 was ‐0.43*% a year ( 95% CI ‐0.66% to ‐0.21; P = 0.001); for the rest of USA the slope after 1992 was +0.03% a year (95% CI ‐0.06% to 0.12%; P = 0.46), * significantly different from zero and from the slope for the rest of USA (P < 0.001).
BRFSS only 1990 ‐ 1999: Baseline % prevalence (95% CI) total pop 1990, MA/rest of USA: 23.5 (21.0 to 26.1)/24.2 (23.7 to 24.7);
Follow‐up 1999: 19.4 (18.0 to 20.8)/23.3 (22.9 to 23.7); significantly lower than in the rest of USA (P < 0.001); crude prevalence OR of current smoking in MA in 1999 compared to 1990: 0.78 (0.66 to 0.92, P trend = 0.01); adjusted for sex, age, race, education OR: 0.83, (0.70 to 0.99, P trend = 0.08); in rest of USA prevalence of current smoking in 1999 compared to 1990: OR 0.95 (0.92 to 0.99, P trend = 0.99); adjusted for sex, age, race, education OR: 1.01 (0.97 to 1.05, P trend < 0.001). Between 1990 and 1999 average change in the log odds was ‐1.3% in MA and in rest of USA +0.6% (sig difference between slopes, P = 0.01). Men/women baseline prevalence % (95% CI) 1990, MA/rest of USA: M: 25.9 (22.0 to 29.8)/26.0 (25.2 to 26.7); W: 21.5 (18.2 to 24.8)/22.5 (21.9 to 23.2);
Follow‐up 1999: M: 19.5* (17.3 to 21.6)/25.6 (24.9 to 26.2); *significantly different from the rest of USA, P < 0.001; W: 19.3* (17.5 to 21.1). 21.2 (20.7 to 21.7), * significantly different from the rest of USA P = 0.04;
Crude prevalence OR (95% CI) of current smoking in MA/rest of USA in 1999 compared to 1990:
M: OR: 0.69 ( 0.54 to 0.89; P trend = 0.03)/0.98 (0.93 to 1.03; P trend = 0.07); Adjusted for age, race/ethnicity. education OR: 0.73 (0.56 to 0.94; P trend = 0.09)/1.03 (0.97 to 1.08, P trend < 0.001);
W: crude OR: 0.87 (0.70 to 1.09; P trend = 0.09)/0.93 (0.88 to 0.97, P trend = 0.03);
Adjusted for age, race/ethnicity, education OR: 0.95 (0.75 to 1.2; P trend = 0.43)/0.99 (0.95 to 1.04, P trend = 0.03);
Average decline per year in the log odds of current smoking between 1990 and 1999 in MA was M: 1.8% (SE: 0.011) and W: 0.7% (SE: 0.010), and in rest of USA there was an average annual increase of : M: 0.8% (SE: 0.002), W: 0.4% (SE: 0.002); Men: significant difference between MA and USA, P = 0.016; W: non‐sig difference between MA and USA P = 0.243.

Annual per capita tobacco consumption based on aggregated sales data:
1988 ‐ 1992 ‐ before tax increase declines in consumption for MA adults and for rest of USA were similar: 15%, 14% ‐ this = annual decline 3 ‐ 4%.
In 1993 consumption declined by 12% in MA and by 4% in rest of USA.
After 1993 there was consistent annual decline of more than 4% in MA and less then 1% in rest of USA.

Quit ratio: not reported.
Quit attempts: % of smokers planning to quit within the next 30 days increased between 1993 and 1997 (from 22% to 42%). Percentage of past year smokers making quit attempt increased non‐sig between 1993 and 1996 (from 47.5% to 54.2%). Percentage of quitters making successful quit attempt significantly increased between 1993 and 2000 (from 18% to 25%), % of smoking pregnant women decreased (from 25% in 1990 to 11% in 1999, while in the rest of US these %s were 18% and 12% respectively).

McAlister 2004

Selection: telephone surveys of randomly selected (random digit dialing) adults from Texas (including individuals not resident in treatment areas); respondent in each household with the most recent birthday aged 18+ selected. Cohort of smokers identified in baseline cross‐sectional sample was followed.

N of participants:
1069 daily cig smokers identified in baseline survey; 622 available for follow‐up.
Baseline: High media 133, low media 274, control 232: Follow‐up (% of original sample): 87 (65), 158 (58), 137 (59). Comparability of demographic data at baseline: not analysed.

Not reported ‐ only levels of daily smoking given (15.7 at baseline, 17.5 at follow up)..

Not reported.

Complete cessation achieved by approx 2% of panel of daily smokers.
% quitting daily smoking, i.e. reduction, not complete cessation (data estimated from bar graph): non‐significantly higher in high and low media groups compared to group without any intervention, both in the whole sample and in followed‐up subsample.
% of whole sample quitting daily smoking: high media 4.5, low media 4.7, control 3.0 (taken from graph); Followed group 7.0%, 8.0%, 5%.
Quit attempts: Among the participants still smoking at follow‐up 27.3% had made quit attempt.

McPhee 1995

Selection: Phone surveys of randomly selected Vietnamese men, chosen randomly from 23 most common Vietnamese surnames in area telephone books. After enumerating all men aged 18+ living in the household and speaking Vietnamese, subjects for interview were selected according to modified Kish procedure.

N of participants:
Santa Clara(SC)/Houston: Baseline (% response rate):
1990: 1322 (81)/1581 (82); Follow‐up (% response rate)
1992 (response rate): 1264 (85)/1209 (88).
Comparability of demographic data at baseline: sig differences in educational level, English language proficiency, mean year of immigration and unemployment.

At baseline smoking prevalence in SC differed significantly from Houston (control community). It slightly increased in control area and remained unchanged in intervention area ‐ there was no intervention effect. Current smokers among Vietnamese men, SC/ Houston: Baseline 1990: 36%/40%;
Followup 1992: 36%/41%.

At baseline SC cig consumption differed significantly from Houston. It fell significantly in the control area , but there was no significant intervention effect. Mean cpd among smokers: SC/Houston: Baseline 1990: 9.9/13.2;
Follow‐up 1992: 9.6/12.0.

Quit rates in past 2 yrs differed significantly at baseline, but there was no significant intervention effect.
SC/Houston: Baseline 1990: 8.0%/6.0%,
Follow‐up 1992: 10%/7%.

Mean number of quit attempts was significantly higher in SC at baseline. At follow‐up it increased significantly in both communities, but did not differ. Mean number of quit attempts: SC/Houston:
Baseline 1990: 1.3/1.1.
Follow‐up 1992: 2.3/2.5.
At baseline percentage reporting any quit attempt in the past was significantly higher in SC. At follow‐up it increased significantly in both communities, but did not differ. Percentage reporting any quit attempt in the past: SC/Houston:
Baseline 1990: 64%/49%.
Follow‐up 1992: 79%/77%.
Adjusted OR for intervention in regression model was 1.1 (95% CI 0.9 to 1.4), i.e. no sig effect of programme.

McVey 2000

Selection: In each TV region a random sample of enumeration districts were selected (1 in 40) and within each district a random sample of households (1 in 20) using computer‐generated lists. Selected households were visited by interviewers and a resident member was selected for the sample with the use of pseudo‐random Kish‐grid method. Only adults at least 16 yrs were sampled. Only smokers and ex‐smokers were included in the study. Attempts were made to re‐interview the same respondents 6m and 18m later with similar structured questionnaire.

N of participants:
TV‐media (smokers/ex‐smokers), Baseline sample: 1744/1256;
Followed at 6m: 1064/854; Followed at 18m: 673/598; Control (smokers/ex‐smokers),
Baseline sample: 719/775;
Followed at 6m: 475/571;
Followed at 18m: 288/402.
Comparability of demographic data at baseline: no statistical analyses.

Not reported other than varying in different target regions.

Not reported.

By 18m, more successfully followed smokers in TV media group gave up smoking than in control group (non‐sig). Quit rates in smokers (TV media/control): 9.7%/8.7%; OR: 1.27 (95% CI 0.77 to 2.08), adjusted for predictors of giving up cigarettes: sex, age, manual/non‐manual job, daily cig consumption, "want to reduce smoking" and sex by manual/non‐manual work interaction. More ex‐smokers not relapsing were observed in TV media than in control group (significant): 96.3% vs 94.5%, adjusted OR: 2.21 (95% CI 1.11 to 4.40, P = 0.025); adjusted for predictors of remaining off cigs: number of cpd before stopping, length of time since stopping, whether persuaded to stop smoking by somebody else. TV media compared to no intervention was estimated to have increased the odds of not smoking.
Pooled common effect on not smoking: adjusted OR: 1.53 (1.02 to 2.29, P = 0.04).

Mogielnicki 1986

Selection: reception clerks asked all patients registering for clinic visits about smoking and interest in stopping.

N of participants:
In 2nd yr clinic response rates not reported (media group/control group) Baseline: 71/33; Follow‐up (% retention rate): 38 (54)/17 (52);
Mailing group: Baseline: no information given;
Follow‐up: 12/5, response rates not reported.
Comparability of demographic data at baseline for clinic participants: non‐sig differences in age, number of cigs smoked, thiocyanate level, % smoking high‐tar cigarettes. No details for mailing group participants given.

Not reported.

Not reported.

In clinic participants, number abstinent was higher in group receiving mass media compared to group not receiving mass media (calculated for all participants included in the study at baseline). In the mailing group in both media and no‐media groups no participant was abstinent and no number of participants included in the study at baseline was given.
Abstinence rates % (media group vs control group), clinic participants, total sample: 19.7 vs 3;
Followed sample: 36.8 vs 5.9.
Mailing group participants, followed sample: 0% vs 0%.

North Coast QFL 1983

Selection: systematic random sample of men and women 18+ from each town, up to 2 adults per household.

N of participants:
Coffs Harbour (CH)/Tamworth (control):
Baseline 1978 (% response rate): 612 (71)/589 (72);
1980: 1272 (73)/1239 (74); 1981: 1195 (73)/1195 (74). Comparability of demographic data at baseline: age and sex differences across towns and yrs.

Significant effect of the intervention was found.
Prevalence reported for 1978 (baseline), 1980, 1981 by town, age, sex and yr. In both towns younger people tended to have bigger declines and 65+ smallest. % points change in prevalence between 1978 and 1981: in CH Men (M) from 7.1 (65+ yrs) to 11.2 (18 ‐ 25 yrs) decline, Women (F): from 6.0 (65+ yrs) to 11.1 (18 ‐ 25 yrs) decline; in Tamworth M 4.1 (65+ yrs) to 5.1 (36 ‐ 45 and 46 ‐ 55 yrs), F 2.1 ( 65+ yrs) to 5.1 (18 ‐ 25 yrs). Effect of intervention compared to control was significant (P < 0.05).

Not reported.

Quit rates not reported.

Stanford 3 City 1977

Selection: in each city random multistage probability sample of men and women 35 ‐ 59 yrs old.

N of participants:
Watsonville/Gilroy/Tracy; Baseline (% of original sample): 605 (73)/542 (82)/532 (81);
Completing yr 2 follow‐up (% of baseline sample): 423 (70)/397 (73)/384 (72).
High‐risk subjects ‐ Watsonville media only/Gilroy/Tracy, Baseline: 56, 139, 136; Completing baseline and 2 yr follow‐up: 40, 94, 95. Comparability of demographic data at baseline: no statistical comparisons made.

Reported only for high‐risk group (baseline/1st /2nd /3rd year follow‐ups):
Watsonville‐randomised control (media only): 56.8/no results given as difference in the direction contrary to prediction;
Gilroy: 62.4/‐15.1/‐15.1/‐11.3;
Tracy: 52.8/‐6.4/‐10.6/‐14.9,
all non‐sig.

Per capita cig consumption for adults 35 ‐ 59 yrs (baseline/% change 1973/1974):
Watsonville reconstituted: 6.8/‐6.9,‐13.7*; Gilroy: 6.8/‐2.3/‐7.3;
Tracy: 6.9/‐1.1/‐2.5;
* statistically sig difference (P < 0.05) for % change values compared to Gilroy and Tracy (control).
High‐risk subjects (baseline 1972/% change 1973/1974): Watsonville‐randomised control: 14.2/‐5.8/‐15.1;
Gilroy: 14.6/‐9.8/‐13.8;
Tracy: 13.7/‐8.5/‐17.2.
In the publication presenting results for high‐risk group only the results are given for 3 yrs follow‐ up and they differ from the main Stanford Three City publication (Maccoby 1977) as they present the results for subjects who completed baseline and all 3 annual follow‐up surveys (baseline 1972/% change; 1973/1974/1975), Watsonville‐randomised control: 15.4/‐5.8/‐15.1/‐16.0;
Gilroy: 13.8/‐7.0/‐12.3/‐11.8;
Tracy: 14.0/‐7.4/‐15.9/‐21.0; all non‐sig.

Quit rates not reported.

Sydney QFL 1986

Selection: random weekly selection from list of all Australian electoral subdivisions. Selected 10 separate households starting from randomly selected address from each electoral subdivision (clockwise direction around the residential block). Within household youngest person 14+ or 16+ interviewed.

N of participants:
Cross‐sectional surveys (Sydney/rest of Australia), Baseline 1983: 3978/ 5154; Follow‐up 1984: 4051/4318. Melbourne cross sectional survey ‐ 1518.
Cohort (Sydney/Melbourne), Baseline 1983 : 900/600;
Follow‐up 1984 (% retention rate after excluding people moved): 570 (76)/364 (73). Australia‐wide response rate of approximately 60%.
Long‐term follow‐up weekly surveys, Sydney and Melbourne 1981 ‐ 1987: 68,136 males, 70,634 females.
Comparability of demographic data at baseline: no statistical comparisons made.

At 1st cross‐sectional surveys prevalence decreased in Sydney compared to the rest of Australia (non‐sig).
Baseline prevalence 1983/% change at follow‐up 1984; cross‐sectional Sydney: Men (M): 35.9/‐4.2; Women (F): 30.4/‐1.6; Total: 33.1/‐2.8; Rest of Australia: M: 39.2/‐3.3; F: 29.3/‐0.2; Total: 34.1/‐1.6.
Difference between Sydney and rest of Australia: 1.2% (SE 1.49; non‐sig).
In cohort study significant decrease in smoking prevalence in Sydney compared to Melbourne.
Cohort % Sydney/Melbourne: 30.9 (‐3.4)/36.8 (+0.)%. Standardised (to baseline prevalence) difference Sydney vs Melbourne: 5.4% (SE: 1.89, P < 0.01).
Overall campaign effect assessed as a weighted mean of cross‐sectional and cohort assessments showed significant effect of campaign on smoking prevalence. Overall campaign effect weighted mean: 2.8% (95% CI: 0.5 to 5.1).
In model assessing long‐term effects of the campaign (1981 ‐ 1987) there was an immediate decrease in smoking prevalence after the beginning of the intervention (6m) in Sydney and Melbourne in males and females. Continuation of the campaign in subsequent years was associated with further drop in percentage points in Sydney and Melbourne men, but not women. Long‐term effectiveness model: Sydney (baseline estimated prevalence 1981 ‐ June 1983: immediate campaign effect 1983 ‐ 6m % points change/continuation of the campaign 1983 ‐ 1987 % points change per year): M: 38.7/‐2.52/‐1.12; F: 31.6/‐2.61/no sig decline. Melbourne (baseline estimated prevalence 1981 ‐ June 1984: immediate campaign effect 1984 ‐ 6m % points change/continuation of the campaign 1984 ‐ 1987 % points change per year): M: 40.1/‐2.87/‐1.9; F: 30.9/‐2.5/no decline.

Cross‐sectional studies after 1st issue of the campaign found decrease in cpd in Sydney compared to the rest of Australia (non‐sig).
Changes in mean cpd in Sydney and rest of Australia,
Baseline 1983/% change in 1984:
M: 20.1 (‐0.6)/20.3 (+0.2); F: 18.4 (‐1.3)/18.2 (‐0.8).

Significantly larger proportion of Sydney smokers compared to Melbourne smokers quit smoking or cut down on cigs. Relapse rate and initiation rate were similar in both cities.
Cohort Sydney/Melbourne 1983 ‐ 1984: initial smokers % quit: 23/9; initial smokers cut down: 12/9; total quit + cut down: 35*/18; initial ex‐smokers relapse: 10/11; initial non‐smokers started: 4/4.

Quit attempts: Failed attempts to quit or cut down were assessed in cohort of smokers in Sydney and Melbourne ‐ proportions were not significantly different: S vs M: quit: 11% vs 19%, cut down: 20% vs 23%.

* P < 0.05.

Figuras y tablas -
Analysis 1.3

Comparison 1 Mass media versus no mass media, Outcome 3 Primary measures of smoking behaviour.

Study

Type of outcome

Orientation

Cost

Effect

California TCP 2003

Prevalence







Packs per day

Social diffusion theory, with social marketing and social policy change

April 1990 ‐ June 1993 USD 26m for media campaign.
1990 ‐ 5 total programme spend USD 694m

Pre‐programme prevalence 23.3 in CA vs 26.2 in rest of USA. By 1993 down to 18.0 in CA, 23.3 rest of USA; By 1996, prevalence still 18.0 in CA vs 22.4 in rest of USA.

Final data (2008):

CA 13.1%,

rest of US: 19%

Rate of decline % (SE): CA: Pre‐TCP: ‐0.74 (0.12) > early period : ‐1.06 (0.17) [P < 0.05 CA vs USA, P < 0.001 change from pre‐TCP] > late period 0.01 (0.21) P < 0.05 CA vs USA.
USA: ‐0.77 (0.09) > 0.57 (0.14) > 0.28 (0.26).

Final data (up to 2008):

CA 0.32% points per year, rest of US 0.24% points per year.

Media campaign alone (without other Prop 99 components) led to decline in cig consumption of 12.2% between April 1990 and March 1991.
Decline of 7.7 packs per capita (pc) attributable to media campaign alone, with 10% increased expenditure on media yielding 0.5% reduction in cig sales.
1989 ‐ 96, CA spent USD 0.50 pc per year on media, leading to a fall of 3.9 packs pc per yr for each USD spent on media.

Final data (1970 ‐ 2008), per capita taxable sales:

CA sales 26.1% lower than sales for the rest of the US (108.8 versus 147.2 packs per year).

Faster decline in CA vs rest of the US.

2002 48 versus 101 pc per yr

2008: 40 versus 77 pc per yr,

CORIS 1997

Prevalence




Cigarettes per day

Not reported

pc cost of USD 5 over 4 yrs in media‐only intervention town.

After 4 yrs, Intervention (Control) prevalence in men dropped from 49.5 (45.7) to 40.4 (38.1) , and in women from 17.6 (16.1) to 14.0 (15.6) . Data contains paired and unpaired observations, in total population.

After 4 yrs, Intervention (Control) tobacco per day (pd) in men moved from 11.2 (8.8) to 8.6 (7.0) grams, and in women from 2.6 (2.3) to 2.2 (2.4).

Jenkins 1997

Prevalence



Cigarettes per day


Odds of quitting

Not reported

Not reported

At 2 yrs, San Francisco (int) prevalence down from 36.1 to 33.9 (P ≤ 0.01), and Houston (control) up from 39.6 to 40.9. Net change was ‐3.5 % points (P = 0.004).

San Francisco (Houston) cpd pretest: 11.1 (13.2); post‐test: 10.3 (11.9)

OR 1.65 (1.27 to 2.15) in favour of San Francisco

Massachusetts 2003

Prevalence




Packs per day

Social diffusion theory, with social marketing and social policy change

USD 39m pa for whole programme.




MA pc USD 6.50 (in 2000); cf. CA USD 3.31, UK $0.89, Australia $0.48 (1997), South Africa $0.04, France $0.32, spent on tobacco control programmes.

1990 ‐ 9: MA prevalence declined from 23.5 (CI 21.0 to 26.1) to 19.4 (CI 18.0 to 20.8).
41 US states: 24.2 (CI 23.7 to 24.7) to 23.3 (CI 22.9 to 23.7)

1993 ‐ 6: reduction of 0.5 per annum (pa) for each pc USD spent on media.
From 1993 pc MA consumption declined > 4% pa, compared with < 1% pa in comparison states.

McAlister 2004

Prevalence

Daily smoking cessation

Cessation

Social learning theory, transtheoretical model. using modelling, social reinforcement for behaviour change, and emotional arousal.

USD 9m pa

Prevalence of daily smoking increased from 15.7 to 17.5.

% of baseline smokers ceasing daily smoking: Low‐level media: 4.7, High‐level media 4.5, No media 3.0

Media‐only areas achieved 8% cessation vs control 5%.

McPhee 1995

Prevalence



Cigarettes per day

Not reported

Not reported

No change in prevalence in Intervention community from baseline to 2 yrs: 36.0, while controls increased (non‐sig) from 40 to 41.

Intervention cpd down from 9.9 to 9.6, controls down from 13.2 to 12.0.

McVey 2000

Quit and relapse rates.

No formal attributing, but 'morbid humour' used.

Not reported

At 18m, 9.8% of intervention smokers vs 8.7% of control had quit, and 4.3% of intervention ex‐smokers vs 3.7% controls had relapsed. Pooled OR of not smoking was 1.53 (CI 1.02 to 2.29, P = 0.04).

Mogielnicki 1986

Cessation rates

Mass marketing techniques (situation analysis, objective definition, copy development, media plan, campaign launch) combined with traditional clinical trial approach.

Copy development and production: USD 7480, Air time: USD 15,150 (1980 USD)

Per protocol clinic cessation at 2nd year:
Media only 36.8%, No media 5.9%.
Mailing recipients: no quitters in either group.

North Coast QFL 1983

Prevalence

Social marketing and communication theory

Not reported

Mean declines in prevalence in Intervention community of 9.5% from baseline to Yr 3, and in controls of 4.4%.

Stanford 3 City 1977

Prevalence



Cigarettes per day

Social marketing, social learning and communication theory

Not reported

All participants: Baseline: Intervention 65.5, Control 55.6.
Cohort: Baseline Intervention (Control) 62.4 (52.8), declined by 11.3% (14.9%) after 3 yrs (P < 0.05).

Mean Intervention cpd down 7.3% over 2 yrs from 6.8 at baseline; controls down 2.5% from 6.9 at baseline.

N.B. Within treatment groups, cohort baseline cpd of Int 13.8, control 14.0.

Figuras y tablas -
Analysis 1.4

Comparison 1 Mass media versus no mass media, Outcome 4 Study summary by type of outcome.

Study

Differences

Possible confounders

Adjusted effects

Jenkins 1997

San Francisco (Int) respondents significantly less proficient in English, of lower education, lower income, less likely to be employed and more recent year of immigration than Houston (control) respondents.

All these factors were included in multivariate analyses.

Unadjusted ORs not reported. Strongest predictor of quitting was being a student (adjusted OR 2.19, 95% CI 1.45 to 3.33); at least high school education (OR 1.33: CI 1.04 to 1.70); more recent year of immigration (OR 1.03: CI 1.00 to 1.05); each + yr of age (OR 1.03: CI 1.02 to 1.04).

Massachusetts 2003

MA respondents were more likely to be white non‐Hispanic and more likely to be college graduates than the respondents from the rest of USA.

Age, sex, race and education were treated as confounders.

Unadjusted OR for current smoking in MA in 1999 vs 1990 was 0.78 (CI 0.66 to 0.92, P trend 0.01).
Adjusting for sex, age, race and education, OR was 0.83 (CI 0.70 to 0.99).
Compare with USA: 1990 unadjusted OR 0.95 (CI 0.92 to 0.99). Adjusted 1.01 (CI 0.97 to 1.05).

Adjusted prevalence OR for current smoking in MA showed annual decline in log odds of 1.3% pa, whereas USA showed average increase of 0.6% (P = 0.01).

McAlister 2004

Responders and non‐responders to follow‐up survey differed significantly by age, race and gender.
Also significant differences in gender and education between baseline and follow‐up samples.

Daily smoking rate "adjusted for age, gender and educational level". No OR reported.

McPhee 1995

Small but significant differences in mean age, educational level, English language proficiency, mean year of immigration, between SC and Houston and/or between pre‐ and post‐test samples. Larger differences in employment (8% unemployed in SC vs 6% in Houston pre‐test) and income (32% below poverty level in SC vs 26% in Houston post‐test).

Analyses controlled for site, time, (pre‐, post‐test), intervention term (site x time), age, education, English language proficiency, year of immigration, employment status, income.

Differences in prevalence between SC and Houston did not persist after controlling for sociodemographic characteristics. Strongest predictor of current non‐smoking was age 65+. Age 18 ‐ 24, immigration before 1977, college education and English fluency all predicted non‐smoking, but employment and income did not. Adjusted OR for intervention term was 1.1 (95% CI 0.9 to 1.4). Adjusted OR for recent quitting 1.1 (95% CI 0.7 to 1.7).

North Coast QFL 1983

Age and sex differences across towns and years.

To counter known confounding, an AGE*SEX*TOWN term was constructed for the regression model, and a TOWN*YEAR factor.

Sydney QFL 1986

Location of interview (Sydney vs Melbourne) was significant predictor of quitting (P < 0.05).

Sex, age, education, marital status, SES did not predict quitting or act as confounders.

No ORs reported.

Figuras y tablas -
Analysis 1.5

Comparison 1 Mass media versus no mass media, Outcome 5 Baseline differences and possible confounding.

Mass media smoking cessation intervention compared with no intervention for smoking cessation

Patient or population: Adults who smoke

Settings: Community

Intervention: Mass media

Comparison: No mass media

Outcomes

Impact

No of
Studies

Quality of the evidence
(GRADE)

Comments

Smoking prevalence

(follow up from 6 months to 18 years)

The 2 state‐wide programmes, in which mass media campaigns were part of comprehensive tobacco control programmes, observed greater declines in smoking prevalence than in the rest of the USA. Of the 5 community‐based studies, 2 found significant decreases in smoking prevalence, 1 did not detect a significant effect, and of 2 studies conducted among Vietnamese‐American men, one detected a statistically significant decrease in smoking prevalence at 2 years, while the other did not.

7 (n = 1,965,478)1

⊕⊝⊝⊝
very low 2,3

2 studies were interrupted time series; and 5 were quasi experimental

Tobacco consumption

(follow up from 2 to 18 years)

In the 2 state‐wide campaigns cigarette consumption was measured on the basis of aggregated sales data. In 1, a statistically significant decline was observed compared with the rest of the USA. In the other, declines in consumption were reported, but without statistical comparisons. Of the five community‐based studies, 1 study detected a significant reduction in cigarette or tobacco consumption for a high‐risk group, who also received counselling, but not in the media‐only intervention community compared with controls. The remaining 4 studies did not detect significant differences.

7 (n = 1,964,292)1

⊕⊝⊝⊝
very low 2,3

2 studies were interrupted time series and 5 were quasi experimental

Abstinence or quit rates (follow up from 6 months to 18 years)

Among the 8 studies presenting abstinence or quit rates, 4 showed some positive effect. 1 state‐wide campaign presented the quit ratio (i.e. the percentage of ever‐smokers (current and former) who were ex‐smokers in a given year), finding a statistically significant difference in favour of the intervention. The other 7 studies were community based; of these, 4 detected a statistically significant effect in favour of the intervention across the study population, 1 found statistically significant differences only in women, 1 study reported a non‐statistically significant difference in point prevalence of daily smoking, and 1 detected no significant differences.

8 (n = 987,800)1

⊕⊝⊝⊝
very low 2,3

1 study was quasi RCT and 7 were quasi experimental

Quit attempts

(follow‐up from 7 months to 18 years)

Of the 5 studies that assessed quit attempts, 3 were community‐based studies, and 2 were state‐wide campaigns. 2 community‐based studies among Vietnamese‐American men found no significant differences between the intervention and control communities. A third study only assessed quit attempts among participants still smoking at follow‐up, of whom 27.3% had made quit attempt. 2 state‐wide campaigns assessed quit attempts only in the intervention community. In 1 they increased in line with campaign duration but not to a statistically significant extent, in the other the percentage of smokers with a quit attempt in the last year increased over the duration of the study, but without statistical comparisons.

5 (n=1,947,674)1

⊕⊝⊝⊝
very low 2,3

2 studies were interrupted time series and 3 studies were quasi experimental

GRADE Working Group grades of evidence
High quality: Further research is very unlikely to change our confidence in the estimate of effect.
Moderate quality: Further research is likely to have an important impact on our confidence in the estimate of effect and may change the estimate.
Low quality: Further research is very likely to have an important impact on our confidence in the estimate of effect and is likely to change the estimate.
Very low quality: We are very uncertain about the estimate.

1 Estimated participant numbers based on the number of respondents to surveys and the number of participants at follow up in the quasi‐experimental studies.

2 GRADE rating started at 'low': none of the studies were RCTs. One was a quasi‐RCT, two were interrupted time series and eight were quasi‐experimental.

3 Downgraded one level for inconsistency: included studies varied in design, intervention, comparator and population, and results were not homogenous.

Figuras y tablas -
Comparison 1. Mass media versus no mass media

Outcome or subgroup title

No. of studies

No. of participants

Statistical method

Effect size

1 Response and retention rates Show forest plot

Other data

No numeric data

2 Intermediate measures Show forest plot

Other data

No numeric data

3 Primary measures of smoking behaviour Show forest plot

Other data

No numeric data

4 Study summary by type of outcome Show forest plot

Other data

No numeric data

5 Baseline differences and possible confounding Show forest plot

Other data

No numeric data

Figuras y tablas -
Comparison 1. Mass media versus no mass media